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Functional significance of the platelet immune receptors GPVI and CLEC-2

The Journal of clinical investigation, 2019-01, Vol.129 (1), p.12-23

2019

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Platelets in Sepsis: An Update on Experimental Models and Clinical Data

Frontiers in immunology, 2019-07, Vol.10, p.1687-1687

2019

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Podoplanin: An emerging cancer biomarker and therapeutic target

Cancer science, 2018-05, Vol.109 (5), p.1292-1299

2018

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Endothelial cells: source, barrier, and target of defensive mediators

Immunological reviews, 2016-11, Vol.274 (1), p.307-329

2016

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The podoplanin-CLEC-2 axis inhibits inflammation in sepsis

Nature communications, 2017-12, Vol.8 (1), p.2239-14, Article 2239

2017

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The dual role of platelet‐innate immune cell interactions in thrombo‐inflammation

Research and practice in thrombosis and haemostasis, 2020-01, Vol.4 (1), p.23-35

2020

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Basic Mechanisms of Hemolysis-Associated Thrombo-Inflammation and Immune Dysregulation

Arteriosclerosis, thrombosis, and vascular biology, 2023-08, Vol.43 (8), p.1349-1361

2023

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Heme induces human and mouse platelet activation through C-type-lectin-like receptor-2

Haematologica (Roma), 2021-02, Vol.106 (2), p.626-629

2021

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Platelets: bridging thrombosis and inflammation

Platelets (Edinburgh), 2021-04, Vol.32 (3), p.293-294

2021

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It takes guts to boost platelet reactivity and inflammation

Blood, 2022-03, Vol.139 (12), p.1781-1783

Rayes, Julie

2022

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Post-translational polymodification of β1-tubulin regulates motor protein localisation in platelet production and function

Haematologica (Roma), 2022-01, Vol.107 (1), p.243-259

2022

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The contribution of platelet glycoprotein receptors to inflammatory bleeding prevention is stimulus and organ dependent

Haematologica (Roma), 2018-06, Vol.103 (6), p.e256-e258

2018

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Platelet glycoprotein VI and C-type lectin-like receptor 2 deficiency accelerates wound healing by impairing vascular integrity in mice

Haematologica (Roma), 2019-08, Vol.104 (8), p.1648-1660

2019

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Hot under the clot: venous thrombogenesis is an inflammatory process

Blood, 2024-08, Vol.144 (5), p.477-489

2024

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Severity of SARS-CoV-2 infection is associated with high numbers of alveolar mast cells and their degranulation

Frontiers in immunology, 2022-09, Vol.13, p.968981-968981

2022

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Megakaryocyte NLRP3 hyperactivation induces mild anemia and potentiates inflammatory response in mice

Frontiers in immunology, 2023-08, Vol.14, p.1226196

2023

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CLEC-2 Prevents Accumulation and Retention of Inflammatory Macrophages During Murine Peritonitis

Frontiers in immunology, 2021-06, Vol.12, p.693974-693974

2021

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The interaction between factor H and VWF increases factor H cofactor activity and regulates VWF prothrombotic status

Blood, 2014-01, Vol.123 (1), p.121-125

2014

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Lymphatic blood filling in CLEC-2-deficient mouse models

Platelets (Edinburgh), 2021-04, Vol.32 (3), p.352-367

2021

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Complement C3 is a novel modulator of the anti-factor VIII immune response

Haematologica (Roma), 2018-02, Vol.103 (2), p.351-360

2018

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Prevention of the anti-factor VIII memory B-cell response by inhibition of Bruton tyrosine kinase in experimental hemophilia A

Haematologica (Roma), 2019-05, Vol.104 (5), p.1046-1054

2019

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SARS-CoV-2 Spike- and Nucleoprotein-Specific Antibodies Induced After Vaccination or Infection Promote Classical Complement Activation

Frontiers in immunology, 2022-07, Vol.13, p.838780

2022

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Adverse Outcome in COVID-19 Is Associated With an Aggravating Hypo-Responsive Platelet Phenotype

Frontiers in cardiovascular medicine, 2021-12, Vol.8, p.795624-795624

2021

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Mutation and ADAMTS13-dependent modulation of disease severity in a mouse model for von Willebrand disease type 2B

Blood, 2010-06, Vol.115 (23), p.4870-4877

2010

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Modelling the pathology and treatment of cardiac fibrosis in vascularised atrial and ventricular cardiac microtissues

Frontiers in cardiovascular medicine, 2023-09, Vol.10, p.1156759-1156759

2023

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Functional significance of the platelet immune receptors GPVI and CLEC-2

Platelets in Sepsis: An Update on Experimental Models and Clinical Data

Podoplanin: An emerging cancer biomarker and therapeutic target

Endothelial cells: source, barrier, and target of defensive mediators

The podoplanin-CLEC-2 axis inhibits inflammation in sepsis

The dual role of platelet‐innate immune cell interactions in thrombo‐inflammation

Basic Mechanisms of Hemolysis-Associated Thrombo-Inflammation and Immune Dysregulation

Heme induces human and mouse platelet activation through C-type-lectin-like receptor-2

Platelets: bridging thrombosis and inflammation

It takes guts to boost platelet reactivity and inflammation

Post-translational polymodification of β1-tubulin regulates motor protein localisation in platelet production and function

The contribution of platelet glycoprotein receptors to inflammatory bleeding prevention is stimulus and organ dependent

Platelet glycoprotein VI and C-type lectin-like receptor 2 deficiency accelerates wound healing by impairing vascular integrity in mice

Hot under the clot: venous thrombogenesis is an inflammatory process

Severity of SARS-CoV-2 infection is associated with high numbers of alveolar mast cells and their degranulation

Megakaryocyte NLRP3 hyperactivation induces mild anemia and potentiates inflammatory response in mice

CLEC-2 Prevents Accumulation and Retention of Inflammatory Macrophages During Murine Peritonitis

The interaction between factor H and VWF increases factor H cofactor activity and regulates VWF prothrombotic status

Lymphatic blood filling in CLEC-2-deficient mouse models

Complement C3 is a novel modulator of the anti-factor VIII immune response

Prevention of the anti-factor VIII memory B-cell response by inhibition of Bruton tyrosine kinase in experimental hemophilia A

SARS-CoV-2 Spike- and Nucleoprotein-Specific Antibodies Induced After Vaccination or Infection Promote Classical Complement Activation

Adverse Outcome in COVID-19 Is Associated With an Aggravating Hypo-Responsive Platelet Phenotype

Mutation and ADAMTS13-dependent modulation of disease severity in a mouse model for von Willebrand disease type 2B

Modelling the pathology and treatment of cardiac fibrosis in vascularised atrial and ventricular cardiac microtissues

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