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The first‐in‐man randomized trial of a beta3 adrenoceptor agonist in chronic heart failure: the BEAT‐HF trial

European journal of heart failure, 2017-04, Vol.19 (4), p.566-575

2017

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Glutathionylation-Dependence of Na+-K+-Pump Currents Can Mimic Reduced Subsarcolemmal Na+ Diffusion

Biophysical journal, 2016-03, Vol.110 (5), p.1099-1109

2016

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Displacement of Native FXYD Protein From Na + /K + -ATPase With Novel FXYD Peptide Derivatives: Effects on Doxorubicin Cytotoxicity

Frontiers in oncology, 2022-03, Vol.12, p.859216-859216

2022

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β3 Adrenergic Stimulation of the Cardiac Na+-K+ Pump by Reversal of an Inhibitory Oxidative Modification

Circulation (New York, N.Y.), 2010-12, Vol.122 (25), p.2699-2708

2010

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Identification of Electric-Field-Dependent Steps in the Na+,K+-Pump Cycle

Biophysical journal, 2014-09, Vol.107 (6), p.1352-1363

2014

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Extracellular Allosteric Na+ Binding to the Na+,K+-ATPase in Cardiac Myocytes

Biophysical journal, 2013-12, Vol.105 (12), p.2695-2705

2013

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Kinetic Comparisons of Heart and Kidney Na+,K+-ATPases

Biophysical journal, 2012-08, Vol.103 (4), p.677-688

2012

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S-glutathionylation of the Na+-K+ Pump: A Novel Redox Mechanism in Preeclampsia

The journal of clinical endocrinology and metabolism, 2021-03, Vol.106 (4), p.1091-1100

2021

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Exaggeration of nonculprit stenosis severity during acute myocardial infarction: implications for immediate multivessel revascularization

Journal of the American College of Cardiology, 2002-09, Vol.40 (5), p.911-916

2002

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The nitric oxide donor sodium nitroprusside stimulates the Na+âK+ pump in isolated rabbit cardiac myocytes

The Journal of physiology, 2005-06, Vol.565 (3), p.815-825

2005

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Reversible Oxidative Modification: A Key Mechanism of Na+-K+ Pump Regulation

Circulation research, 2009-07, Vol.105 (2), p.185-193

2009

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Oxidative regulation of the Na+–K+ pump in the cardiovascular system

Free radical biology & medicine, 2012-12, Vol.53 (12), p.2263-2268

2012

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Hemodynamic Effects of Cyclic Guanosine Monophosphate-Dependent Signaling Through β3 Adrenoceptor Stimulation in Patients With Advanced Heart Failure: A Randomized Invasive Clinical Trial

Circulation. Heart failure, 2022-07, Vol.15 (7), p.e009120-e009120

2022

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β3-Adrenoceptor activation relieves oxidative inhibition of the cardiac Na+-K+ pump in hyperglycemia induced by insulin receptor blockade

American Journal of Physiology: Cell Physiology, 2015-09, Vol.309 (5), p.C286-C295

2015

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Targeting Cardiac Myocyte Na + -K + Pump Function With β3 Adrenergic Agonist in Rabbit Model of Severe Congestive Heart Failure

Circulation. Heart failure, 2020-09, Vol.13 (9), p.e006753-e006753

2020

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FXYD Proteins Reverse Inhibition of the Na+-K+ Pump Mediated by Glutathionylation of Its β1 Subunit

The Journal of biological chemistry, 2011-05, Vol.286 (21), p.18562-18572

2011

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Silencing overexpression of FXYD3 protein in breast cancer cells amplifies effects of doxorubicin and γ-radiation on Na+/K+-ATPase and cell survival

Breast cancer research and treatment, 2016-01, Vol.155 (2), p.203-213

2016

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Glutathionylation Mediates Angiotensin II–Induced eNOS Uncoupling, Amplifying NADPH Oxidase‐Dependent Endothelial Dysfunction

Journal of the American Heart Association, 2014-04, Vol.3 (2), p.e000731-n/a

2014

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Membrane accessibility of glutathione

Biochimica et biophysica acta, 2015-10, Vol.1848 (10), p.2430-2436

2015

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Susceptibility of β1 Na+-K+ Pump Subunit to Glutathionylation and Oxidative Inhibition Depends on Conformational State of Pump

The Journal of biological chemistry, 2012-04, Vol.287 (15), p.12353-12364

2012

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Stimulation of the cardiac myocyte Na+-K+ pump due to reversal of its constitutive oxidative inhibition

American Journal of Physiology: Cell Physiology, 2015-08, Vol.309 (4), p.C239-C250

2015

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Field triage to primary angioplasty combined with emergency department bypass reduces treatment delays and is associated with improved outcome

European heart journal, 2007-10, Vol.28 (19), p.2313-2319

2007

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Angiotensin II inhibits the Na+-K+ pump via PKC-dependent activation of NADPH oxidase

American Journal of Physiology: Cell Physiology, 2009-04, Vol.296 (4), p.C693-C700

2009

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Reversible Oxidative Modification: Implications for Cardiovascular Physiology and Pathophysiology

Trends in cardiovascular medicine, 2010-04, Vol.20 (3), p.85-90

2010

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β-Adrenergic Regulation of the Cardiac Na+ -K+ ATPase Mediated by Oxidative Signaling

Trends in cardiovascular medicine, 2012-05, Vol.22 (4), p.83-87

2012

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The first‐in‐man randomized trial of a beta3 adrenoceptor agonist in chronic heart failure: the BEAT‐HF trial

Glutathionylation-Dependence of Na+-K+-Pump Currents Can Mimic Reduced Subsarcolemmal Na+ Diffusion

Displacement of Native FXYD Protein From Na + /K + -ATPase With Novel FXYD Peptide Derivatives: Effects on Doxorubicin Cytotoxicity

β3 Adrenergic Stimulation of the Cardiac Na+-K+ Pump by Reversal of an Inhibitory Oxidative Modification

Identification of Electric-Field-Dependent Steps in the Na+,K+-Pump Cycle

Extracellular Allosteric Na+ Binding to the Na+,K+-ATPase in Cardiac Myocytes

Kinetic Comparisons of Heart and Kidney Na+,K+-ATPases

S-glutathionylation of the Na+-K+ Pump: A Novel Redox Mechanism in Preeclampsia

Exaggeration of nonculprit stenosis severity during acute myocardial infarction: implications for immediate multivessel revascularization

The nitric oxide donor sodium nitroprusside stimulates the Na+âK+ pump in isolated rabbit cardiac myocytes

Reversible Oxidative Modification: A Key Mechanism of Na+-K+ Pump Regulation

Oxidative regulation of the Na+–K+ pump in the cardiovascular system

Hemodynamic Effects of Cyclic Guanosine Monophosphate-Dependent Signaling Through β3 Adrenoceptor Stimulation in Patients With Advanced Heart Failure: A Randomized Invasive Clinical Trial

β3-Adrenoceptor activation relieves oxidative inhibition of the cardiac Na+-K+ pump in hyperglycemia induced by insulin receptor blockade

Targeting Cardiac Myocyte Na + -K + Pump Function With β3 Adrenergic Agonist in Rabbit Model of Severe Congestive Heart Failure

FXYD Proteins Reverse Inhibition of the Na+-K+ Pump Mediated by Glutathionylation of Its β1 Subunit

Silencing overexpression of FXYD3 protein in breast cancer cells amplifies effects of doxorubicin and γ-radiation on Na+/K+-ATPase and cell survival

Glutathionylation Mediates Angiotensin II–Induced eNOS Uncoupling, Amplifying NADPH Oxidase‐Dependent Endothelial Dysfunction

Membrane accessibility of glutathione

Susceptibility of β1 Na+-K+ Pump Subunit to Glutathionylation and Oxidative Inhibition Depends on Conformational State of Pump

Stimulation of the cardiac myocyte Na+-K+ pump due to reversal of its constitutive oxidative inhibition

Field triage to primary angioplasty combined with emergency department bypass reduces treatment delays and is associated with improved outcome

Angiotensin II inhibits the Na+-K+ pump via PKC-dependent activation of NADPH oxidase

Reversible Oxidative Modification: Implications for Cardiovascular Physiology and Pathophysiology

β-Adrenergic Regulation of the Cardiac Na+ -K+ ATPase Mediated by Oxidative Signaling

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