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Aging-Associated miR-217 Aggravates Atherosclerosis and Promotes Cardiovascular Dysfunction

Arteriosclerosis, thrombosis, and vascular biology, 2020-10, Vol.40 (10), p.2408-2424

2020

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MicroRNAs Prevent the Generation of Autoreactive Antibodies

Immunity (Cambridge, Mass.), 2010-11, Vol.33 (5), p.713-722

2010

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A broad atlas of somatic hypermutation allows prediction of activation-induced deaminase targets

The Journal of experimental medicine, 2018-03, Vol.215 (3), p.761-771

2018

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Activation-induced deaminase expression defines mature B cell lymphoma in the mouse

Frontiers in immunology, 2023-09, Vol.14, p.1268930-1268930

2023

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Estrogen directly activates AID transcription and function

The Journal of experimental medicine, 2009-01, Vol.206 (1), p.99-111

2009

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MicroRNA control of lymphocyte differentiation and function

Current opinion in immunology, 2011-06, Vol.23 (3), p.368-373

2011

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miR-181b negatively regulates activation-induced cytidine deaminase in B cells

The Journal of experimental medicine, 2008-09, Vol.205 (10), p.2199-2206

2008

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Regulation of B-cell development and function by microRNAs

Immunological reviews, 2013-05, Vol.253 (1), p.25-39

2013

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miR-28-based combination therapy impairs aggressive B cell lymphoma growth by rewiring DNA replication

Cell death & disease, 2023-10, Vol.14 (10), p.687-687, Article 687

2023

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CTCF orchestrates the germinal centre transcriptional program and prevents premature plasma cell differentiation

Nature communications, 2017-07, Vol.8 (1), p.16067-16067, Article 16067

2017

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A new role for circulating T follicular helper cells in humoral response to anti-PD-1 therapy

Journal for immunotherapy of cancer, 2020-09, Vol.8 (2), p.e001187

2020

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Transcription enhances AID-mediated cytidine deamination by exposing single-stranded DNA on the nontemplate strand

Nature immunology, 2003-05, Vol.4 (5), p.452-456

2003

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Role of genomic instability and p53 in AID-induced c-myc – Igh translocations

Nature, 2006-03, Vol.440 (7080), p.105-109

2006

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CCCTC-Binding Factor Locks Premature IgH Germline Transcription and Restrains Class Switch Recombination

Frontiers in immunology, 2017-09, Vol.8, p.1076-1076

2017

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miRNA-Based Therapies in B Cell Non-Hodgkin Lymphoma

Trends in immunology, 2020-10, Vol.41 (10), p.932-947

2020

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Low-affinity CTCF binding drives transcriptional regulation whereas high-affinity binding encompasses architectural functions

iScience, 2023-03, Vol.26 (3), p.106106-106106, Article 106106

2023

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Assessing the impact of an antigen-specific antibody response on atherosclerosis development in mice

STAR protocols, 2023-06, Vol.4 (2), p.102274-102274, Article 102274

2023

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C-Terminal Deletion of AID Uncouples Class Switch Recombination from Somatic Hypermutation and Gene Conversion

Molecular cell, 2003-08, Vol.12 (2), p.501-508

2003

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Somatic hypermutation is limited by CRM1-dependent nuclear export of activation-induced deaminase

The Journal of experimental medicine, 2004-05, Vol.199 (9), p.1235-1244

2004

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UNG shapes the specificity of AID-induced somatic hypermutation

The Journal of experimental medicine, 2012-07, Vol.209 (7), p.1379-1389

2012

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Activation-induced cytidine deaminase targets SUV4-20-mediated histone H4K20 trimethylation to class-switch recombination sites

Scientific reports, 2017-08, Vol.7 (1), p.7594-13, Article 7594

2017

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ALDH4A1 is an atherosclerosis auto-antigen targeted by protective antibodies

Nature (London), 2021-01, Vol.589 (7841), p.287-292

2021

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GPR55 is a key player for B-cell-mediated atheroprotection

Nature Cardiovascular Research, 2022-11, Vol.1 (11), p.982-983

Ramiro, Almudena R.

2022

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Haploinsufficiency of activation-induced deaminase for antibody diversification and chromosome translocations both in vitro and in vivo

PloS one, 2008-12, Vol.3 (12), p.e3927-e3927

2008

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Aging-Associated miR-217 Aggravates Atherosclerosis and Promotes Cardiovascular Dysfunction

MicroRNAs Prevent the Generation of Autoreactive Antibodies

A broad atlas of somatic hypermutation allows prediction of activation-induced deaminase targets

Activation-induced deaminase expression defines mature B cell lymphoma in the mouse

Estrogen directly activates AID transcription and function

MicroRNA control of lymphocyte differentiation and function

miR-181b negatively regulates activation-induced cytidine deaminase in B cells

Regulation of B-cell development and function by microRNAs

Infectious stimuli promote malignant B-cell acute lymphoblastic leukemia in the absence of AID

miR-28-based combination therapy impairs aggressive B cell lymphoma growth by rewiring DNA replication

CTCF orchestrates the germinal centre transcriptional program and prevents premature plasma cell differentiation

A new role for circulating T follicular helper cells in humoral response to anti-PD-1 therapy

Transcription enhances AID-mediated cytidine deamination by exposing single-stranded DNA on the nontemplate strand

Role of genomic instability and p53 in AID-induced c-myc – Igh translocations

CCCTC-Binding Factor Locks Premature IgH Germline Transcription and Restrains Class Switch Recombination

miRNA-Based Therapies in B Cell Non-Hodgkin Lymphoma

Low-affinity CTCF binding drives transcriptional regulation whereas high-affinity binding encompasses architectural functions

Assessing the impact of an antigen-specific antibody response on atherosclerosis development in mice

C-Terminal Deletion of AID Uncouples Class Switch Recombination from Somatic Hypermutation and Gene Conversion

Somatic hypermutation is limited by CRM1-dependent nuclear export of activation-induced deaminase

UNG shapes the specificity of AID-induced somatic hypermutation

Activation-induced cytidine deaminase targets SUV4-20-mediated histone H4K20 trimethylation to class-switch recombination sites

ALDH4A1 is an atherosclerosis auto-antigen targeted by protective antibodies

GPR55 is a key player for B-cell-mediated atheroprotection

Haploinsufficiency of activation-induced deaminase for antibody diversification and chromosome translocations both in vitro and in vivo

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