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Mouse models of metabolic liver injury

Laboratory animals (London), 2015-04, Vol.49 (1_suppl), p.47-58

2015

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P0965 : High fat diet treatment of conditional c-met KO-mice develop an inflammatory phenotype and progress to fibrosis independently from steatosis

Journal of hepatology, 2015-04, Vol.62, p.S708-S708

2015

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Caspase-8 Deficiency Ameliorates Hepatic Steatosis, but not Apoptosis in Alcoholic Liver Injury

Journal of hepatology, 2016, Vol.64 (2), p.S230-S231

2016

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P113 DEFICIENT MICRORNA-21 INDUCTION PROMOTES SMALL-FOR-SIZE SYNDROME FOLLOWING EXTENDED HEPATECTOMY IN MICE

Journal of hepatology, 2014-04, Vol.60 (1), p.S104-S104

2014

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1288 miR-21 DEFICIENCY IMPROVES GLUCOSE TOLERANCE AND HEPATIC LIPID CATABOLISM IN MICE FED A HIGH-FAT DIET

Journal of hepatology, 2013-04, Vol.58, p.S520-S521

2013

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Guidelines and Considerations for Metabolic Tolerance Tests in Mice

Diabetes, metabolic syndrome and obesity, 2020-02, Vol.13, p.439-450

2020

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Hepatocyte specific deletion of c-Met leads to the development of severe non-alcoholic steatohepatitis in mice

Journal of hepatology, 2014-10, Vol.61 (4), p.883-890

2014

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Neutrophil recruitment in the reperfused-injured rat liver was effectively attenuated by repertaxin, a novel allosteric non-competitive inhibitor of CXCL8 receptors: A therapeutic approach for the treatment of post-ischemic hepatic syndromes

International journal of immunopathology and pharmacology, 2005-09, Vol.18 (3), p.475-486

2005

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Neutrophil Recruitment in the Reperfused-Injured Rat Liver was Effectively Attenuated by Repertaxin, a Novel Allosteric Noncompetitive Inhibitor of Cxcl8 Receptors: A Therapeutic Approach for the Treatment of Post-Ischemic Hepatic Syndromes

International journal of immunopathology and pharmacology, 2005-07, Vol.18 (3), p.475-486

2005

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THU-287 - Caspase-8 Deficiency Ameliorates Hepatic Steatosis, but not Apoptosis in Alcoholic Liver Injury

Journal of hepatology, 2016, Vol.64 (2), p.S230-S231

2016

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Role of the Hypoxia Inducible Factors HIF-1α and HIF-2α during hepatic repair in a rat model of thioacetamide (TAA)-induced chronic liver damage

Zeitschrift für Gastroenterologie, 2014, Vol.52 (1)

2014

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Interleukin–6 plays a central role in the hepatic erythropoietin expression during acute phase response: Analysis of two murine models

Zeitschrift für Gastroenterologie, 2010, Vol.48 (1)

2010

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Hepatic Nrf2 overexpression inhibits the deleterious effects induced by c-met deficiency in the progression of NASH

Zeitschrift für Gastroenterologie, 2016, Vol.54 (12), p.1343-1404

2016

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Characterization of the erythropoietin/erythropoietin receptor axis in a rat model of liver damage and cholangiocarcinoma development

Zeitschrift für Gastroenterologie, 2013, Vol.51 (1)

2013

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Lack of hepatic keap-1 ameliorates steatosis through a negative LXR modulation without altering the innate immune response in a diet-induced model of nonalcoholic steato-hepatitis

Zeitschrift für Gastroenterologie, 2015, Vol.53 (1)

2015

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Lactoferrin gene expression upregulation in liver during turpentine oil- or bacterial LPS-induced acute phase reaction

Zeitschrift für Gastroenterologie, 2012, Vol.50 (1)

2012

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Melanocortin receptors in rat liver cells: change of gene expression and intracellular localization during acute-phase-response

Zeitschrift für Gastroenterologie, 2012, Vol.50 (1)

2012

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Transcription regulators of hepatic heme-oxygenase-1 gene expression in IL-6 knock out mice: Comparison of two different models of acute phase reaction

Zeitschrift für Gastroenterologie, 2009, Vol.47 (9)

2009

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Enhanced expression of c-myc in hepatocytes promotes initiation and progression of alcoholic liver disease

Zeitschrift für Gastroenterologie, 2015, Vol.53 (12)

2015

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Lipocalin-2 gene expression and production in a model of acute phase response reaction

Zeitschrift für Gastroenterologie, 2011, Vol.49 (1)

2011

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Ischaemic preconditioning modulates the activity of Kupffer cells during in vivo reperfusion injury of rat liver

Cell biochemistry and function, 2003-12, Vol.21 (4), p.299-305

2003

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29 The novel IL-8 inhibitor repertaxin reduces the neutrophil recruitment in the rat liver submitted to ischemia/reperfusion injury

Digestive and liver disease, 2003-02, Vol.35, p.A18-A18

2003

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The behavior of the gonadotropins FSH and LH during and after treatment with estrogenic and progestational drugs

Patologia e clinica ostetrica e ginecologica, 1973, Vol.1 (1), p.26

1973

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The topical use of fluocortolene esters (trimethylacetate and capronate) in the treatment of proctitis caused by irradiation

Clinica terapeutica, 1967-10, Vol.43 (1), p.35

1967

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Auto-aggressive CXCR6 + CD8 T cells cause liver immune pathology in NASH

Nature (London), 2021-04, Vol.592 (7854), p.444-449

2021

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Mouse models of metabolic liver injury

P0965 : High fat diet treatment of conditional c-met KO-mice develop an inflammatory phenotype and progress to fibrosis independently from steatosis

Caspase-8 Deficiency Ameliorates Hepatic Steatosis, but not Apoptosis in Alcoholic Liver Injury

P113 DEFICIENT MICRORNA-21 INDUCTION PROMOTES SMALL-FOR-SIZE SYNDROME FOLLOWING EXTENDED HEPATECTOMY IN MICE

1288 miR-21 DEFICIENCY IMPROVES GLUCOSE TOLERANCE AND HEPATIC LIPID CATABOLISM IN MICE FED A HIGH-FAT DIET

Guidelines and Considerations for Metabolic Tolerance Tests in Mice

Hepatocyte specific deletion of c-Met leads to the development of severe non-alcoholic steatohepatitis in mice

Neutrophil recruitment in the reperfused-injured rat liver was effectively attenuated by repertaxin, a novel allosteric non-competitive inhibitor of CXCL8 receptors: A therapeutic approach for the treatment of post-ischemic hepatic syndromes

Neutrophil Recruitment in the Reperfused-Injured Rat Liver was Effectively Attenuated by Repertaxin, a Novel Allosteric Noncompetitive Inhibitor of Cxcl8 Receptors: A Therapeutic Approach for the Treatment of Post-Ischemic Hepatic Syndromes

THU-287 - Caspase-8 Deficiency Ameliorates Hepatic Steatosis, but not Apoptosis in Alcoholic Liver Injury

Role of the Hypoxia Inducible Factors HIF-1α and HIF-2α during hepatic repair in a rat model of thioacetamide (TAA)-induced chronic liver damage

Interleukin–6 plays a central role in the hepatic erythropoietin expression during acute phase response: Analysis of two murine models

Hepatic Nrf2 overexpression inhibits the deleterious effects induced by c-met deficiency in the progression of NASH

Characterization of the erythropoietin/erythropoietin receptor axis in a rat model of liver damage and cholangiocarcinoma development

Lack of hepatic keap-1 ameliorates steatosis through a negative LXR modulation without altering the innate immune response in a diet-induced model of nonalcoholic steato-hepatitis

Lactoferrin gene expression upregulation in liver during turpentine oil- or bacterial LPS-induced acute phase reaction

Melanocortin receptors in rat liver cells: change of gene expression and intracellular localization during acute-phase-response

Transcription regulators of hepatic heme-oxygenase-1 gene expression in IL-6 knock out mice: Comparison of two different models of acute phase reaction

Enhanced expression of c-myc in hepatocytes promotes initiation and progression of alcoholic liver disease

Lipocalin-2 gene expression and production in a model of acute phase response reaction

Ischaemic preconditioning modulates the activity of Kupffer cells during in vivo reperfusion injury of rat liver

29 The novel IL-8 inhibitor repertaxin reduces the neutrophil recruitment in the rat liver submitted to ischemia/reperfusion injury

The behavior of the gonadotropins FSH and LH during and after treatment with estrogenic and progestational drugs

The topical use of fluocortolene esters (trimethylacetate and capronate) in the treatment of proctitis caused by irradiation

Auto-aggressive CXCR6 + CD8 T cells cause liver immune pathology in NASH

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