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HDL promotes rapid atherosclerosis regression in mice and alters inflammatory properties of plaque monocyte-derived cells

Proceedings of the National Academy of Sciences - PNAS, 2011-04, Vol.108 (17), p.7166-7171

2011

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Adipose Mitochondrial Biogenesis Is Suppressed in db/db and High-Fat Diet–Fed Mice and Improved by Rosiglitazone

Diabetes (New York, N.Y.), 2007-07, Vol.56 (7), p.1751-1760

2007

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Transdifferentiation of mouse aortic smooth muscle cells to a macrophage-like state after cholesterol loading

Proceedings of the National Academy of Sciences - PNAS, 2003-11, Vol.100 (23), p.13531-13536

2003

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The endoplasmic reticulum is the site of cholesterol-induced cytotoxicity in macrophages

Nature cell biology, 2003-09, Vol.5 (9), p.781-792

2003

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ACAT inhibition reduces the progression of preexisting, advanced atherosclerotic mouse lesions without plaque or systemic toxicity

Arteriosclerosis, thrombosis, and vascular biology, 2013-01, Vol.33 (1), p.4

2013

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Reversal of Hyperlipidemia With a Genetic Switch Favorably Affects the Content and Inflammatory State of Macrophages in Atherosclerotic Plaques

Circulation (New York, N.Y.), 2011-03, Vol.123 (9), p.989-998

2011

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Eliminating atherogenesis in mice by switching off hepatic lipoprotein secretion

Circulation (New York, N.Y.), 2003-03, Vol.107 (9), p.1315-1321

2003

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Electrophysiological characterization of human and mouse sodium-dependent citrate transporters (NaCT/SLC13A5) reveal species differences with respect to substrate sensitivity and cation dependence

The Journal of pharmacology and experimental therapeutics, 2015-11, Vol.355 (2), p.247

2015

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Dramatic remodeling of advanced atherosclerotic plaques of the apolipoprotein E–deficient mouse in a novel transplantation model

Journal of vascular surgery, 2001-09, Vol.34 (3), p.541,2A-547,2A

2001

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Lysophosphatidylcholine stimulates monocyte chemoattractant protein-1 gene expression in rat aortic smooth muscle cells

Arteriosclerosis, thrombosis, and vascular biology, 2002-10, Vol.22 (10), p.1617

2002

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Rosiglitazone Induces Mitochondrial Biogenesis in Differentiated Murine 3T3-L1 and C3H/10T1/2 Adipocytes

PPAR Research, 2011-01, Vol.2011, p.179454-11

2011

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Dyslipidemia Associated with Atherosclerotic Disease Systemically Alters Dendritic Cell Mobilization

Immunity (Cambridge, Mass.), 2004-10, Vol.21 (4), p.561-574

2004

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Acyl-coenzymeA (CoA):cholesterol acyltransferase inhibition in rat and human aortic smooth muscle cells is nontoxic and retards foam cell formation

Arteriosclerosis, thrombosis, and vascular biology, 2005-01, Vol.25 (1), p.122

2005

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Comparison of mitochondrial and macrophage content between subcutaneous and visceral fat in db/ db mice

Experimental and molecular pathology, 2007-08, Vol.83 (1), p.73-83

2007

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Elevating high-density lipoprotein cholesterol in apolipoprotein E-deficient mice remodels advanced atherosclerotic lesions by decreasing macrophage and increasing smooth muscle cell content

Circulation (New York, N.Y.), 2001-11, Vol.104 (20), p.2447-2452

2001

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High-density lipoproteins retard the progression of atherosclerosis and favorably remodel lesions without suppressing indices of inflammation or oxidation

Arteriosclerosis, thrombosis, and vascular biology, 2004-10, Vol.24 (10), p.1904

2004

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Atherosclerotic lesions in genetically modified mice quantified in vivo by non-invasive high-resolution magnetic resonance microscopy

Atherosclerosis, 2002-06, Vol.162 (2), p.315-321

2002

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Serial, noninvasive, in vivo magnetic resonance microscopy detects the development of atherosclerosis in apolipoprotein E-deficient mice and its progression by arterial wall remodeling

Journal of magnetic resonance imaging, 2003-02, Vol.17 (2), p.184-189

2003

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Arterial injury by cholesterol oxidation products causes endothelial dysfunction and arterial wall cholesterol accumulation

Arteriosclerosis, thrombosis, and vascular biology, 1998-12, Vol.18 (12), p.1885

1998

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Cholesterol oxidation products induce vascular foam cell lesion formation in hypercholesterolemic New Zealand white rabbits

Arteriosclerosis, thrombosis, and vascular biology, 1999-09, Vol.19 (9), p.2179

1999

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Abstract 3967: CRISPR/Cas9 screening identified novel membrane targets sensitizing hepatocellular carcinoma cells to natural killer cell-mediated cytotoxicity

Cancer research (Chicago, Ill.), 2023-04, Vol.83 (7_Supplement), p.3967-3967

2023

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High-density lipoprotein: gene-based approaches to the prevention of atherosclerosis

Annals of medicine (Helsinki), 2000, Vol.32 (9), p.642-651

2000

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Elevating high-density lipoprotein cholesterol in apolipoprotein e-deficient mice remodels advanced atherosclerotic lesions by decreasing macrophage and increasing smooth muscle

Circulation (New York, N.Y.), 2001-11, Vol.104 (20), p.2447

2001

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Elevating high-density lipoprotein cholesterol in apolipoprotein E-deficient mice remodels advanced atherosclerotic lesions by decreasing macrophage and increasing smooth muscle cell content. Editorial

Circulation (New York, N.Y.), 2001, Vol.104 (20), p.2447-2387

2001

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HDL promotes rapid atherosclerosis regression in mice and alters inflammatory properties of plaque monocyte-derived cells

Selective class IIa histone deacetylase inhibition via a nonchelating zinc-binding group

Adipose Mitochondrial Biogenesis Is Suppressed in db/db and High-Fat Diet–Fed Mice and Improved by Rosiglitazone

Transdifferentiation of mouse aortic smooth muscle cells to a macrophage-like state after cholesterol loading

The endoplasmic reticulum is the site of cholesterol-induced cytotoxicity in macrophages

ACAT inhibition reduces the progression of preexisting, advanced atherosclerotic mouse lesions without plaque or systemic toxicity

Reversal of Hyperlipidemia With a Genetic Switch Favorably Affects the Content and Inflammatory State of Macrophages in Atherosclerotic Plaques

Eliminating atherogenesis in mice by switching off hepatic lipoprotein secretion

Electrophysiological characterization of human and mouse sodium-dependent citrate transporters (NaCT/SLC13A5) reveal species differences with respect to substrate sensitivity and cation dependence

Dramatic remodeling of advanced atherosclerotic plaques of the apolipoprotein E–deficient mouse in a novel transplantation model

Lysophosphatidylcholine stimulates monocyte chemoattractant protein-1 gene expression in rat aortic smooth muscle cells

Rosiglitazone Induces Mitochondrial Biogenesis in Differentiated Murine 3T3-L1 and C3H/10T1/2 Adipocytes

Dyslipidemia Associated with Atherosclerotic Disease Systemically Alters Dendritic Cell Mobilization

Acyl-coenzymeA (CoA):cholesterol acyltransferase inhibition in rat and human aortic smooth muscle cells is nontoxic and retards foam cell formation

Comparison of mitochondrial and macrophage content between subcutaneous and visceral fat in db/ db mice

Elevating high-density lipoprotein cholesterol in apolipoprotein E-deficient mice remodels advanced atherosclerotic lesions by decreasing macrophage and increasing smooth muscle cell content

High-density lipoproteins retard the progression of atherosclerosis and favorably remodel lesions without suppressing indices of inflammation or oxidation

Atherosclerotic lesions in genetically modified mice quantified in vivo by non-invasive high-resolution magnetic resonance microscopy

Serial, noninvasive, in vivo magnetic resonance microscopy detects the development of atherosclerosis in apolipoprotein E-deficient mice and its progression by arterial wall remodeling

Arterial injury by cholesterol oxidation products causes endothelial dysfunction and arterial wall cholesterol accumulation

Cholesterol oxidation products induce vascular foam cell lesion formation in hypercholesterolemic New Zealand white rabbits

Abstract 3967: CRISPR/Cas9 screening identified novel membrane targets sensitizing hepatocellular carcinoma cells to natural killer cell-mediated cytotoxicity

High-density lipoprotein: gene-based approaches to the prevention of atherosclerosis

Elevating high-density lipoprotein cholesterol in apolipoprotein e-deficient mice remodels advanced atherosclerotic lesions by decreasing macrophage and increasing smooth muscle

Elevating high-density lipoprotein cholesterol in apolipoprotein E-deficient mice remodels advanced atherosclerotic lesions by decreasing macrophage and increasing smooth muscle cell content. Editorial

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