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Enhancing the Cytotoxic Effects of PARP Inhibitors with DNA Demethylating Agents – A Potential Therapy for Cancer

Cancer cell, 2016-10, Vol.30 (4), p.637-650

2016

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HDAC inhibitors: roles of DNA damage and repair

Advances in cancer research, 2012, Vol.116, p.87-129

2012

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CHD4 Has Oncogenic Functions in Initiating and Maintaining Epigenetic Suppression of Multiple Tumor Suppressor Genes

Cancer cell, 2017-05, Vol.31 (5), p.653-668.e7

2017

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Phase I Clinical Trial of DNA Methyltransferase Inhibitor Decitabine and PARP Inhibitor Talazoparib Combination Therapy in Relapsed/Refractory Acute Myeloid Leukemia

Clinical cancer research, 2022-04, Vol.28 (7), p.1313-1322

2022

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An Effective Epigenetic-PARP Inhibitor Combination Therapy for Breast and Ovarian Cancers Independent of BRCA Mutations

Clinical cancer research, 2018-07, Vol.24 (13), p.3163-3175

2018

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DNA double‐strand break response in stem cells: Mechanisms to maintain genomic integrity

Biochimica et biophysica acta, 2013-02, Vol.1830 (2), p.2345-2353

2013

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c-MYC Generates Repair Errors via Increased Transcription of Alternative-NHEJ Factors, LIG3 and PARP1, in Tyrosine Kinase-Activated Leukemias

Molecular cancer research, 2015-04, Vol.13 (4), p.699-712

2015

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Pharmacologic Induction of BRCAness in BRCA -Proficient Cancers: Expanding PARP Inhibitor Use

Cancers, 2022-05, Vol.14 (11), p.2640

2022

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Activating STING1-dependent immune signaling in TP53 mutant and wild-type acute myeloid leukemia

Proceedings of the National Academy of Sciences - PNAS, 2022-07, Vol.119 (27), p.e2123227119

2022

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DNA methyltransferase inhibitors induce a BRCAness phenotype that sensitizes NSCLC to PARP inhibitor and ionizing radiation

Proceedings of the National Academy of Sciences - PNAS, 2019-11, Vol.116 (45), p.22609-22618

2019

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Personalized synthetic lethality induced by targeting RAD52 in leukemias identified by gene mutation and expression profile

Blood, 2013-08, Vol.122 (7), p.1293-1304

2013

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The Epigenetic Hallmarks of Cancer

Cancer discovery, 2024-10, Vol.14 (10), p.1783-1809

2024

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PARP1 PARylates and stabilizes STAT5 in FLT3-ITD acute myeloid leukemia and other STAT5-activated cancers

Translational oncology, 2022-01, Vol.15 (1), p.101283-101283, Article 101283

2022

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Partnering with PARP inhibitors in acute myeloid leukemia with FLT3-ITD

Cancer letters, 2019-07, Vol.454, p.171-178

2019

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Cells expressing FLT3/ITD mutations exhibit elevated repair errors generated through alternative NHEJ pathways: implications for genomic instability and therapy

Blood, 2010-12, Vol.116 (24), p.5298-5305

2010

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How Epigenetic Therapy Beats Adverse Genetics in Monosomy Karyotype AML

Cancer research (Chicago, Ill.), 2021-02, Vol.81 (4), p.813-815

2021

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Pharmacologic induction of innate immune signaling directly drives homologous recombination deficiency

Proceedings of the National Academy of Sciences - PNAS, 2020-07, Vol.117 (30), p.17785-17795

2020

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Internal tandem duplication of FLT3 (FLT3/ITD) induces increased ROS production, DNA damage, and misrepair: implications for poor prognosis in AML

Blood, 2008-03, Vol.111 (6), p.3173-3182

2008

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Histone demethylase JMJD1A promotes expression of DNA repair factors and radio-resistance of prostate cancer cells

Cell death & disease, 2020-04, Vol.11 (4), p.214-214, Article 214

2020

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Genomic instability in myeloid malignancies: Increased reactive oxygen species (ROS), DNA double strand breaks (DSBs) and error-prone repair

Cancer letters, 2008-10, Vol.270 (1), p.1-9

2008

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Histone deacetylase inhibitors decrease NHEJ both by acetylation of repair factors and trapping of PARP1 at DNA double-strand breaks in chromatin

Leukemia research, 2016-06, Vol.45, p.14-23

2016

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Oxidative stress leads to increased mutation frequency in a murine model of myelodysplastic syndrome

Leukemia research, 2014-01, Vol.38 (1), p.95-102

2014

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Targeting abnormal DNA double strand break repair in cancer

Cellular and molecular life sciences : CMLS, 2010-11, Vol.67 (21), p.3699-3710

2010

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Targeting abnormal DNA repair in therapy-resistant breast cancers

Molecular cancer research, 2012-01, Vol.10 (1), p.96-107

2012

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Enhancing the Cytotoxic Effects of PARP Inhibitors with DNA Demethylating Agents – A Potential Therapy for Cancer

HDAC inhibitors: roles of DNA damage and repair

CHD4 Has Oncogenic Functions in Initiating and Maintaining Epigenetic Suppression of Multiple Tumor Suppressor Genes

Phase I Clinical Trial of DNA Methyltransferase Inhibitor Decitabine and PARP Inhibitor Talazoparib Combination Therapy in Relapsed/Refractory Acute Myeloid Leukemia

An Effective Epigenetic-PARP Inhibitor Combination Therapy for Breast and Ovarian Cancers Independent of BRCA Mutations

DNA double‐strand break response in stem cells: Mechanisms to maintain genomic integrity

c-MYC Generates Repair Errors via Increased Transcription of Alternative-NHEJ Factors, LIG3 and PARP1, in Tyrosine Kinase-Activated Leukemias

Transient Low Doses of DNA-Demethylating Agents Exert Durable Antitumor Effects on Hematological and Epithelial Tumor Cells

Pharmacologic Induction of BRCAness in BRCA -Proficient Cancers: Expanding PARP Inhibitor Use

Activating STING1-dependent immune signaling in TP53 mutant and wild-type acute myeloid leukemia

DNA methyltransferase inhibitors induce a BRCAness phenotype that sensitizes NSCLC to PARP inhibitor and ionizing radiation

Personalized synthetic lethality induced by targeting RAD52 in leukemias identified by gene mutation and expression profile

The Epigenetic Hallmarks of Cancer

PARP1 PARylates and stabilizes STAT5 in FLT3-ITD acute myeloid leukemia and other STAT5-activated cancers

Partnering with PARP inhibitors in acute myeloid leukemia with FLT3-ITD

Cells expressing FLT3/ITD mutations exhibit elevated repair errors generated through alternative NHEJ pathways: implications for genomic instability and therapy

How Epigenetic Therapy Beats Adverse Genetics in Monosomy Karyotype AML

Pharmacologic induction of innate immune signaling directly drives homologous recombination deficiency

Internal tandem duplication of FLT3 (FLT3/ITD) induces increased ROS production, DNA damage, and misrepair: implications for poor prognosis in AML

Histone demethylase JMJD1A promotes expression of DNA repair factors and radio-resistance of prostate cancer cells

Genomic instability in myeloid malignancies: Increased reactive oxygen species (ROS), DNA double strand breaks (DSBs) and error-prone repair

Histone deacetylase inhibitors decrease NHEJ both by acetylation of repair factors and trapping of PARP1 at DNA double-strand breaks in chromatin

Oxidative stress leads to increased mutation frequency in a murine model of myelodysplastic syndrome

Targeting abnormal DNA double strand break repair in cancer

Targeting abnormal DNA repair in therapy-resistant breast cancers

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