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Circulating tumor cell technologies

Molecular oncology, 2016-03, Vol.10 (3), p.374-394

2016

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Heparanase Regulates Secretion, Composition, and Function of Tumor Cell-derived Exosomes

The Journal of biological chemistry, 2013-04, Vol.288 (14), p.10093-10099

2013

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Targeting the tetraspanin CD81 reduces cancer invasion and metastasis

Proceedings of the National Academy of Sciences - PNAS, 2021-06, Vol.118 (24), p.1-8

2021

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The heparanase/syndecan‐1 axis in cancer: mechanisms and therapies

The FEBS journal, 2013-05, Vol.280 (10), p.2294-2306

2013

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Chemotherapy induces secretion of exosomes loaded with heparanase that degrades extracellular matrix and impacts tumor and host cell behavior

Matrix biology, 2018-01, Vol.65, p.104-118

2018

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SST0001, a Chemically Modified Heparin, Inhibits Myeloma Growth and Angiogenesis via Disruption of the Heparanase/Syndecan-1 Axis

Clinical cancer research, 2011-03, Vol.17 (6), p.1382-1393

2011

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Chemotherapy induces expression and release of heparanase leading to changes associated with an aggressive tumor phenotype

Matrix biology, 2016-09, Vol.55, p.22-34

2016

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Shed syndecan-1 translocates to the nucleus of cells delivering growth factors and inhibiting histone acetylation: a novel mechanism of tumor-host cross-talk

The Journal of biological chemistry, 2015-01, Vol.290 (2), p.941-949

2015

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Heparanase Plays a Dual Role in Driving Hepatocyte Growth Factor (HGF) Signaling by Enhancing HGF Expression and Activity

The Journal of biological chemistry, 2011-02, Vol.286 (8), p.6490-6499

2011

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Chemotherapy stimulates syndecan-1 shedding: A potentially negative effect of treatment that may promote tumor relapse

Matrix biology, 2014-04, Vol.35, p.215-222

2014

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Heparan Sulfate Chains of Syndecan-1 Regulate Ectodomain Shedding

The Journal of biological chemistry, 2012-03, Vol.287 (13), p.9952-9961

2012

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Targeting heparanase overcomes chemoresistance and diminishes relapse in myeloma

Oncotarget, 2016-01, Vol.7 (2), p.1598-1607

2016

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Heparanase is a host enzyme required for herpes simplex virus-1 release from cells

Nature communications, 2015-04, Vol.6 (1), p.6985-6985

2015

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Investigating circulating tumor cells and distant metastases in patient-derived orthotopic xenograft models of triple-negative breast cancer

Breast cancer research : BCR, 2019-08, Vol.21 (1), p.98-98, Article 98

2019

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Heparanase promotes myeloma stemness and in vivo tumorigenesis

Matrix biology, 2020-06, Vol.88, p.53-68

2020

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Desmoglein 2 is a substrate of kallikrein 7 in pancreatic cancer

BMC cancer, 2008-12, Vol.8 (1), p.373-373, Article 373

2008

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Real-Time Detection of Circulating Tumor Cells in Living Animals Using Functionalized Large Gold Nanorods

Nano letters, 2019-04, Vol.19 (4), p.2334-2342

2019

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Enumeration and targeted analysis of KRAS, BRAF and PIK3CA mutations in CTCs captured by a label-free platform: Comparison to ctDNA and tissue in metastatic colorectal cancer

Oncotarget, 2016-12, Vol.7 (51), p.85349-85364

2016

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Kallikrein 7 enhances pancreatic cancer cell invasion by shedding E‐cadherin

Cancer, 2007-05, Vol.109 (9), p.1811-1820

2007

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Proteolytic action of kallikrein-related peptidase 7 produces unique active matrix metalloproteinase-9 lacking the C-terminal hemopexin domains

Biochimica et biophysica acta, 2011-08, Vol.1813 (8), p.1525-1531

2011

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Tumor shedding and metastatic progression after tumor excision in patient-derived orthotopic xenograft models of triple-negative breast cancer

Clinical & experimental metastasis, 2020-06, Vol.37 (3), p.413-424

2020

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Heparanase is a host enzyme required for herpes simplex virus-1 release from cells

Nature communications, 2015-04, Vol.6 (1), Article 6985

2015

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Heparanase Enhances Local and Systemic Osteolysis in Multiple Myeloma by Upregulating the Expression and Secretion of RANKL

Cancer research (Chicago, Ill.), 2010-11, Vol.70 (21), p.8329-8338

2010

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The extracellular matrix protein fibronectin is a substrate for kallikrein 7

Biochemical and biophysical research communications, 2008-05, Vol.369 (4), p.1169-1173

2008

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Circulating tumor cell technologies

Heparanase Regulates Secretion, Composition, and Function of Tumor Cell-derived Exosomes

Targeting the tetraspanin CD81 reduces cancer invasion and metastasis

The heparanase/syndecan‐1 axis in cancer: mechanisms and therapies

Chemotherapy induces secretion of exosomes loaded with heparanase that degrades extracellular matrix and impacts tumor and host cell behavior

SST0001, a Chemically Modified Heparin, Inhibits Myeloma Growth and Angiogenesis via Disruption of the Heparanase/Syndecan-1 Axis

Chemotherapy induces expression and release of heparanase leading to changes associated with an aggressive tumor phenotype

Shed syndecan-1 translocates to the nucleus of cells delivering growth factors and inhibiting histone acetylation: a novel mechanism of tumor-host cross-talk

Heparanase Plays a Dual Role in Driving Hepatocyte Growth Factor (HGF) Signaling by Enhancing HGF Expression and Activity

Chemotherapy stimulates syndecan-1 shedding: A potentially negative effect of treatment that may promote tumor relapse

Heparan Sulfate Chains of Syndecan-1 Regulate Ectodomain Shedding

Targeting heparanase overcomes chemoresistance and diminishes relapse in myeloma

Heparanase is a host enzyme required for herpes simplex virus-1 release from cells

Investigating circulating tumor cells and distant metastases in patient-derived orthotopic xenograft models of triple-negative breast cancer

Heparanase promotes myeloma stemness and in vivo tumorigenesis

Desmoglein 2 is a substrate of kallikrein 7 in pancreatic cancer

Real-Time Detection of Circulating Tumor Cells in Living Animals Using Functionalized Large Gold Nanorods

Enumeration and targeted analysis of KRAS, BRAF and PIK3CA mutations in CTCs captured by a label-free platform: Comparison to ctDNA and tissue in metastatic colorectal cancer

Immune-stimulating antibody conjugates elicit robust myeloid activation and durable antitumor immunity

Kallikrein 7 enhances pancreatic cancer cell invasion by shedding E‐cadherin

Proteolytic action of kallikrein-related peptidase 7 produces unique active matrix metalloproteinase-9 lacking the C-terminal hemopexin domains

Tumor shedding and metastatic progression after tumor excision in patient-derived orthotopic xenograft models of triple-negative breast cancer

Heparanase is a host enzyme required for herpes simplex virus-1 release from cells

Heparanase Enhances Local and Systemic Osteolysis in Multiple Myeloma by Upregulating the Expression and Secretion of RANKL

The extracellular matrix protein fibronectin is a substrate for kallikrein 7

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