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Retigabine suppresses loss of force in mouse models of hypokalaemic periodic paralysis

Brain (London, England : 1878), 2023-04, Vol.146 (4), p.1554-1560

2023

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Stac3 enhances expression of human CaV1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels

The Journal of general physiology, 2018-03, Vol.150 (3), p.475-489

2018

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Gating Pore Currents in DIII Hypopp Mutations of CaV1.1

Biophysical journal, 2018-02, Vol.114 (3), p.639a-639a

2018

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Inward rectifier potassium currents in mammalian skeletal muscle fibres

The Journal of physiology, 2015-03, Vol.593 (5), p.1213-1238

2015

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Creation of a Novel Humanized Dystrophic Mouse Model of Duchenne Muscular Dystrophy and Application of a CRISPR/Cas9 Gene Editing Therapy

Journal of neuromuscular diseases, 2017, Vol.4 (2), p.139-145

2017

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Voltage-dependent dynamic FRET signals from the transverse tubules in mammalian skeletal muscle fibers

The Journal of general physiology, 2007-12, Vol.130 (6), p.581-600

2007

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The delayed rectifier potassium conductance in the sarcolemma and the transverse tubular system membranes of mammalian skeletal muscle fibers

The Journal of general physiology, 2012-08, Vol.140 (2), p.109-137

2012

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Age‐dependent chloride channel expression in skeletal muscle fibres of normal and HSALR myotonic mice

The Journal of physiology, 2013-03, Vol.591 (5), p.1347-1371

2013

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Action potential-evoked calcium release is impaired in single skeletal muscle fibers from heart failure patients

PloS one, 2014-10, Vol.9 (10), p.e109309-e109309

2014

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Effects of membrane depolarization and changes in extracellular [K⁺] on the Ca²⁺ transients of fast skeletal muscle fibers. Implications for muscle fatigue

Journal of muscle research and cell motility, 2010, Vol.31 (1), p.13-33

2010

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Functional expression of transgenic 1sDHPR channels in adult mammalian skeletal muscle fibres

The Journal of physiology, 2011-03, Vol.589 (Pt 6), p.1421-1442

2011

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Gating pore currents occur in CaV1.1 domain III mutants associated with HypoPP

The Journal of general physiology, 2021-11, Vol.153 (11), p.1

2021

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Functional rescue of periodic paralysis by in vivo gene editing of CaV1.1

Biophysical journal, 2022-02, Vol.121 (3), p.177a-177a

2022

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A Skeletal Muscle Conditional KCNJ2 Knock-Out Mouse Model for Periodic Paralysis in Andersen-Tawil Syndrome

Biophysical journal, 2019-02, Vol.116 (3), p.242a-242a

2019

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The distinct role of the four voltage sensors of the skeletal CaV1.1 channel in voltage-dependent activation

The Journal of general physiology, 2021-11, Vol.153 (11), p.1

2021

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NaV1.4 Loss of Function Changes for Recessively Inherited Myopathy with Fluctuating Weakness

Biophysical journal, 2018-02, Vol.114 (3), p.632a-632a

2018

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Stac3 enhances expression of human Ca^sub V^1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels

The Journal of general physiology, 2018-03, Vol.150 (3), p.475

2018

Volltextzugriff (PDF)

Recovery from acidosis is a robust trigger for loss of force in murine hypokalemic periodic paralysis

The Journal of general physiology, 2019-04, Vol.151 (4), p.555-566

2019

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A Mutation Linked to Malignant Hyperthermia in the Skeletal CaV1.1 Channel Stabilizes the Resting State of Voltage Sensor I and Impairs Channel Activation

Biophysical journal, 2019-02, Vol.116 (3), p.111a-111a

2019

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Recovery from Intracellular Acidosis Triggers Loss of Force in Hypokalemic Periodic Paralysis

Biophysical journal, 2019-02, Vol.116 (3), p.32

2019

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The Contribution of the Individual Voltage Sensors to the Activation of Skeletal CaV1.1 Channels

Biophysical journal, 2020-02, Vol.118 (3), p.105a-105a

2020

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DNA transfection of mammalian skeletal muscles using in vivo electroporation

Journal of visualized experiments, 2009-10 (32)

2009

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Optically-Tracked Structural Rearrangements of the Voltage Sensing Domains in the Human CaV1.1 Channel

Biophysical journal, 2018-02, Vol.114 (3), p.39a-39a

2018

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A highly-selective chloride microelectrode based on a mercuracarborand anion carrier

Scientific reports, 2019-12, Vol.9 (1), p.18860-12, Article 18860

2019

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Myasthenic congenital myopathy from recessive mutations at a single residue in Na V 1.4

Neurology, 2019-03, Vol.92 (13), p.e1405

2019

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Retigabine suppresses loss of force in mouse models of hypokalaemic periodic paralysis

Stac3 enhances expression of human CaV1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels

Gating Pore Currents in DIII Hypopp Mutations of CaV1.1

Inward rectifier potassium currents in mammalian skeletal muscle fibres

Creation of a Novel Humanized Dystrophic Mouse Model of Duchenne Muscular Dystrophy and Application of a CRISPR/Cas9 Gene Editing Therapy

Voltage-dependent dynamic FRET signals from the transverse tubules in mammalian skeletal muscle fibers

The delayed rectifier potassium conductance in the sarcolemma and the transverse tubular system membranes of mammalian skeletal muscle fibers

Age‐dependent chloride channel expression in skeletal muscle fibres of normal and HSALR myotonic mice

Action potential-evoked calcium release is impaired in single skeletal muscle fibers from heart failure patients

Effects of membrane depolarization and changes in extracellular [K⁺] on the Ca²⁺ transients of fast skeletal muscle fibers. Implications for muscle fatigue

Functional expression of transgenic 1sDHPR channels in adult mammalian skeletal muscle fibres

Gating pore currents occur in CaV1.1 domain III mutants associated with HypoPP

Functional rescue of periodic paralysis by in vivo gene editing of CaV1.1

A Skeletal Muscle Conditional KCNJ2 Knock-Out Mouse Model for Periodic Paralysis in Andersen-Tawil Syndrome

The distinct role of the four voltage sensors of the skeletal CaV1.1 channel in voltage-dependent activation

NaV1.4 Loss of Function Changes for Recessively Inherited Myopathy with Fluctuating Weakness

Stac3 enhances expression of human Ca^sub V^1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels

Recovery from acidosis is a robust trigger for loss of force in murine hypokalemic periodic paralysis

A Mutation Linked to Malignant Hyperthermia in the Skeletal CaV1.1 Channel Stabilizes the Resting State of Voltage Sensor I and Impairs Channel Activation

Recovery from Intracellular Acidosis Triggers Loss of Force in Hypokalemic Periodic Paralysis

The Contribution of the Individual Voltage Sensors to the Activation of Skeletal CaV1.1 Channels

DNA transfection of mammalian skeletal muscles using in vivo electroporation

Optically-Tracked Structural Rearrangements of the Voltage Sensing Domains in the Human CaV1.1 Channel

A highly-selective chloride microelectrode based on a mercuracarborand anion carrier

Myasthenic congenital myopathy from recessive mutations at a single residue in Na V 1.4

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