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XBP1 deficiency promotes hepatocyte pyroptosis by impairing mitophagy to activate mtDNA-cGAS-STING signaling in macrophages during acute liver injury

Redox biology, 2022-06, Vol.52, p.102305-102305, Article 102305

2022

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miR-139-5p sponged by LncRNA NEAT1 regulates liver fibrosis via targeting β-catenin/SOX9/TGF-β1 pathway

Cell death discovery, 2021-09, Vol.7 (1), p.243-15, Article 243

2021

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XBP1-mediated activation of the STING signalling pathway in macrophages contributes to liver fibrosis progression

JHEP reports, 2022-11, Vol.4 (11), p.100555-100555, Article 100555

2022

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Macrophage ATG16L1 expression suppresses metabolic dysfunction-associated steatohepatitis progression by promoting lipophagy

Clinical and molecular hepatology, 2024-07, Vol.30 (3), p.515-538

2024

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TAK1 deficiency promotes liver injury and tumorigenesis via ferroptosis and macrophage cGAS-STING signalling

JHEP reports, 2023-05, Vol.5 (5), p.100695-100695, Article 100695

2023

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Letter to the Editor: Scavenger receptor a is a major homeostatic regulator that restrains drug-induced liver injury

Hepatology (Baltimore, Md.), 2024-08, Vol.80 (2), p.E24

2024

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Reply to: "Short-term activation of PERK alleviates the progression of experimental non-alcoholic steatohepatitis"

Journal of hepatology, 2023-12, Vol.79 (6), p.e237-e238

2023

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Role of XBP1 in regulating the progression of non-alcoholic steatohepatitis

Journal of hepatology, 2022-08, Vol.77 (2), p.312-325

2022

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The role of XBP1 in regulating the progression of non-alcoholic steatohepatitis

Journal of hepatology, 2022-07, Vol.77, p.S695-S696

2022

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Correspondence on Editorial regarding“Macrophage ATG16L1: potential candidate for MASH treatment”

Clinical and molecular hepatology, 2024-07

2024

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Single-cell analysis reveals hypoxia-induced immunosuppressive microenvironment in intrahepatic cholangiocarcinoma

Biochimica et biophysica acta. Molecular basis of disease, 2024-10, Vol.1870 (7), p.167276, Article 167276

2024

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Macrophage ATG16L1 expression suppresses metabolic dysfunction-associated steatohepatitis progression by promoting lipophagy

Clinical and molecular hepatology, 2024-07, Vol.30 (3), p.515

2024

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Integrated single‐cell transcriptomics reveals the hypoxia‐induced inflammation‐cancer transformation in NASH‐derived hepatocellular carcinoma

Cell proliferation, 2024-04, Vol.57 (4), p.e13576-n/a

2024

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Macrophage ATG16L1 expression suppresses MASH progression by promoting lipophagy

Clinical and molecular hepatology, 2024-05

2024

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Reply to: “Short-term activation of PERK alleviates the progression of experimental nonalcoholic steatohepatitis”

Journal of hepatology

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XBP1 deficiency promotes hepatocyte pyroptosis by impairing mitophagy to activate mtDNA-cGAS-STING signaling in macrophages during acute liver injury

miR-139-5p sponged by LncRNA NEAT1 regulates liver fibrosis via targeting β-catenin/SOX9/TGF-β1 pathway

XBP1-mediated activation of the STING signalling pathway in macrophages contributes to liver fibrosis progression

Macrophage ATG16L1 expression suppresses metabolic dysfunction-associated steatohepatitis progression by promoting lipophagy

TAK1 deficiency promotes liver injury and tumorigenesis via ferroptosis and macrophage cGAS-STING signalling

Letter to the Editor: Scavenger receptor a is a major homeostatic regulator that restrains drug-induced liver injury

Reply to: "Short-term activation of PERK alleviates the progression of experimental non-alcoholic steatohepatitis"

Role of XBP1 in regulating the progression of non-alcoholic steatohepatitis

The role of XBP1 in regulating the progression of non-alcoholic steatohepatitis

Correspondence on Editorial regarding“Macrophage ATG16L1: potential candidate for MASH treatment”

Single-cell analysis reveals hypoxia-induced immunosuppressive microenvironment in intrahepatic cholangiocarcinoma

Macrophage ATG16L1 expression suppresses metabolic dysfunction-associated steatohepatitis progression by promoting lipophagy

Integrated single‐cell transcriptomics reveals the hypoxia‐induced inflammation‐cancer transformation in NASH‐derived hepatocellular carcinoma

Macrophage ATG16L1 expression suppresses MASH progression by promoting lipophagy

Reply to: “Short-term activation of PERK alleviates the progression of experimental nonalcoholic steatohepatitis”

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