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PABPN1-Dependent mRNA Processing Induces Muscle Wasting

PLoS genetics, 2016-05, Vol.12 (5), p.e1006031-e1006031

2016

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Sensitive and reliable evaluation of single-cut sgRNAs to restore dystrophin by a GFP-reporter assay

PloS one, 2020-09, Vol.15 (9), p.e0239468-e0239468

2020

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New function of the myostatin/activin type I receptor (ALK4) as a mediator of muscle atrophy and muscle regeneration

The FASEB journal, 2017-01, Vol.31 (1), p.238-255

2017

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Low human dystrophin levels prevent cardiac electrophysiological and structural remodelling in a Duchenne mouse model

Scientific reports, 2021-05, Vol.11 (1), p.9779-9779, Article 9779

2021

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Influence of full-length dystrophin on brain volumes in mouse models of Duchenne muscular dystrophy

PloS one, 2018-03, Vol.13 (3), p.e0194636-e0194636

2018

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The predictive value of models of neuromuscular disorders to potentiate clinical translation

Disease models & mechanisms, 2022-07, Vol.15 (7)

van Putten, Maaike

2022

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Assessing functional performance in the mdx mouse model

Journal of visualized experiments, 2014-03 (85)

2014

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The use of genetically humanized animal models for personalized medicine approaches

Disease models & mechanisms, 2020-02, Vol.13 (2), p.dmm041673

2020

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Timing and localization of human dystrophin isoform expression provide insights into the cognitive phenotype of Duchenne muscular dystrophy

Scientific reports, 2017-10, Vol.7 (1), p.12575-12, Article 12575

2017

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Long-term Exon Skipping Studies With 2′-O-Methyl Phosphorothioate Antisense Oligonucleotides in Dystrophic Mouse Models

Molecular therapy. Nucleic acids, 2012-09, Vol.1 (9), p.e44-e44, Article e44

2012

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Environmental 24-hr Cycles Are Essential for Health

Current biology, 2016-07, Vol.26 (14), p.1843-1853

2016

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Learning, memory and blood–brain barrier pathology in Duchenne muscular dystrophy mice lacking Dp427, or Dp427 and Dp140

Genes, brain and behavior, 2024-06, Vol.23 (3), p.e12895-n/a

2024

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Characterization of neuromuscular synapse function abnormalities in multiple Duchenne muscular dystrophy mouse models

The European journal of neuroscience, 2016-06, Vol.43 (12), p.1623-1635

2016

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Detailed genetic and functional analysis of the hDMDdel52/mdx mouse model

PloS one, 2020-12, Vol.15 (12), p.e0244215

2020

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Antisense Oligonucleotide-Mediated Downregulation of IGFBPs Enhances IGF-1 Signaling

Journal of neuromuscular diseases, 2024-03, Vol.11 (2), p.299-314

2024

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Discovering fiber type architecture over the entire muscle using data‐driven analysis

Cytometry. Part A, 2021-12, Vol.99 (12), p.1240-1249

2021

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A dystrophic Duchenne mouse model for testing human antisense oligonucleotides

PloS one, 2018-02, Vol.13 (2), p.e0193289-e0193289

2018

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Nonclinical Exon Skipping Studies with 2′- O -Methyl Phosphorothioate Antisense Oligonucleotides in mdx and mdx-utrn−/− Mice Inspired by Clinical Trial Results

Nucleic acid therapeutics, 2019-04, Vol.29 (2), p.92-103

2019

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Mouse models for muscular dystrophies: an overview

Disease models & mechanisms, 2020-02, Vol.13 (2), p.dmm043562

2020

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On the use of D2.B10-Dmdmdx/J (D2.mdx) Versus C57BL/10ScSn-Dmdmdx/J (mdx) Mouse Models for Preclinical Studies on Duchenne Muscular Dystrophy: A Cautionary Note from Members of the TREAT-NMD Advisory Committee on Therapeutics

Journal of neuromuscular diseases, 2023, Vol.10 (1), p.155-158

2023

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Assessment of Behavioral Characteristics With Procedures of Minimal Human Interference in the mdx Mouse Model for Duchenne Muscular Dystrophy

Frontiers in behavioral neuroscience, 2021-01, Vol.14, p.629043-629043

2021

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Moving neuromuscular disorders research forward: from novel models to clinical studies

Disease models & mechanisms, 2020-02, Vol.13 (2)

2020

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Cross-sectional study into age-related pathology of mouse models for limb girdle muscular dystrophy types 2D and 2F

PloS one, 2019-08, Vol.14 (8), p.e0220665-e0220665

2019

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The effects of low levels of dystrophin on mouse muscle function and pathology

PloS one, 2012-02, Vol.7 (2), p.e31937-e31937

2012

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A modified diet does not ameliorate muscle pathology in a mouse model for Duchenne muscular dystrophy

PloS one, 2019-04, Vol.14 (4), p.e0215335-e0215335

2019

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PABPN1-Dependent mRNA Processing Induces Muscle Wasting

Sensitive and reliable evaluation of single-cut sgRNAs to restore dystrophin by a GFP-reporter assay

New function of the myostatin/activin type I receptor (ALK4) as a mediator of muscle atrophy and muscle regeneration

Low human dystrophin levels prevent cardiac electrophysiological and structural remodelling in a Duchenne mouse model

Influence of full-length dystrophin on brain volumes in mouse models of Duchenne muscular dystrophy

The predictive value of models of neuromuscular disorders to potentiate clinical translation

Assessing functional performance in the mdx mouse model

The use of genetically humanized animal models for personalized medicine approaches

Timing and localization of human dystrophin isoform expression provide insights into the cognitive phenotype of Duchenne muscular dystrophy

Long-term Exon Skipping Studies With 2′-O-Methyl Phosphorothioate Antisense Oligonucleotides in Dystrophic Mouse Models

Environmental 24-hr Cycles Are Essential for Health

Learning, memory and blood–brain barrier pathology in Duchenne muscular dystrophy mice lacking Dp427, or Dp427 and Dp140

Characterization of neuromuscular synapse function abnormalities in multiple Duchenne muscular dystrophy mouse models

Detailed genetic and functional analysis of the hDMDdel52/mdx mouse model

Antisense Oligonucleotide-Mediated Downregulation of IGFBPs Enhances IGF-1 Signaling

Discovering fiber type architecture over the entire muscle using data‐driven analysis

A dystrophic Duchenne mouse model for testing human antisense oligonucleotides

Nonclinical Exon Skipping Studies with 2′- O -Methyl Phosphorothioate Antisense Oligonucleotides in mdx and mdx-utrn−/− Mice Inspired by Clinical Trial Results

Mouse models for muscular dystrophies: an overview

On the use of D2.B10-Dmdmdx/J (D2.mdx) Versus C57BL/10ScSn-Dmdmdx/J (mdx) Mouse Models for Preclinical Studies on Duchenne Muscular Dystrophy: A Cautionary Note from Members of the TREAT-NMD Advisory Committee on Therapeutics

Assessment of Behavioral Characteristics With Procedures of Minimal Human Interference in the mdx Mouse Model for Duchenne Muscular Dystrophy

Moving neuromuscular disorders research forward: from novel models to clinical studies

Cross-sectional study into age-related pathology of mouse models for limb girdle muscular dystrophy types 2D and 2F

The effects of low levels of dystrophin on mouse muscle function and pathology

A modified diet does not ameliorate muscle pathology in a mouse model for Duchenne muscular dystrophy

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