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The role of reactive oxygen species in homeostasis and degradation of cartilage

Osteoarthritis and cartilage, 2003-10, Vol.11 (10), p.747-755

2003

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Rhein, the metabolite of diacerhein, reduces the proliferation of osteoarthritic chondrocytes and synoviocytes without inducing apoptosis

Scandinavian journal of rheumatology, 2009-01, Vol.38 (2), p.104-111

2009

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Immunohistochemical analysis of transforming growth factor beta isoforms and their receptors in human cartilage from normal and osteoarthritic femoral heads

Rheumatology international, 2005-03, Vol.25 (2), p.118-124

2005

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Reactive oxygen species downregulate the expression of pro-inflammatory genes by human chondrocytes

Inflammation research, 2003-03, Vol.52 (3), p.111-118

2003

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Rhein, a diacerhein-derived metabolite, modulates the expression of matrix degrading enzymes and the cell proliferation of articular chondrocytes by inhibiting ERK and JNK-AP-1 dependent pathways

Clinical and experimental rheumatology, 2007-07, Vol.25 (4), p.546

2007

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Comparative effects of IL-1β and hydrogen peroxide (H 2O 2) on catabolic and anabolic gene expression in juvenile bovine chondrocytes

Osteoarthritis and cartilage, 2005-10, Vol.13 (10), p.915-924

2005

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Transforming growth factor β stimulates collagen and glycosaminoglycan biosynthesis in cultured rabbit articular chondrocytes

FEBS letters, 1988-07, Vol.234 (1), p.172-175

1988

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Semiquantitative reverse transcription-polymerase chain reaction analysis of syndecan-1 and -4 messages in cartilage and cultured chondrocytes from osteoarthritic joints

Osteoarthritis and cartilage, 2000-01, Vol.8 (1), p.34-43

2000

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Differentiation potential of human muscle-derived cells towards chondrogenic phenotype in alginate beads culture

Osteoarthritis and cartilage, 2008-12, Vol.16 (12), p.1509-1518

2008

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Sp1 and Sp3 Transcription Factors Mediate Interleukin-1β Down-regulation of Human Type II Collagen Gene Expression in Articular Chondrocytes

The Journal of biological chemistry, 2003-10, Vol.278 (41), p.39762-39772

2003

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Expression of TGF-βs and their receptors is differentially modulated by reactive oxygen species and nitric oxide in human articular chondrocytes

Osteoarthritis and cartilage, 2002-05, Vol.10 (5), p.344-352

2002

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L'agrécanase, enzyme-clé de la dégradation du cartilage dans l'arthrose ?

M.S. Médecine sciences, 2000, Vol.16 (2), p.276

Pujol, JP

2000

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For intra-articular delivery of chondroitin sulfate

Glycobiology (Oxford), 2009-08, Vol.19 (8), p.813-815

2009

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NFκB mediates IL‐1β‐induced down‐regulation of TβRII through the modulation of Sp3 expression

Journal of cellular and molecular medicine, 2008-09, Vol.12 (5a), p.1754-1766

2008

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17 -Oestradiol up-regulates the expression of a functional UDP-glucose dehydrogenase in articular chondrocytes: comparison with effects of cytokines and growth factors

Rheumatology (Oxford, England), 2008-03, Vol.47 (3), p.281-288

2008

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17β-Oestradiol up-regulates the expression of a functional UDP-glucose dehydrogenase in articular chondrocytes: comparison with effects of cytokines and growth factors

Rheumatology (Oxford, England), 2008-03, Vol.47 (3), p.281-288

2008

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Sp3 represses the Sp1-mediated transactivation of the human COL2A1 gene in primary and de-differentiated chondrocytes

The Journal of biological chemistry, 2001-10, Vol.276 (40), p.36881-36895

2001

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Cell density-dependent proliferative effects of transforming growth factor (TGF)-beta 1, beta 2, and beta 3 in human chondrosarcoma cells HCS-2/8 are associated with changes in the expression of TGF-beta receptor type I

Cancer investigation, 2001-01, Vol.19 (5), p.475-486

2001

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Hypoxia affects mesenchymal stromal cell osteogenic differentiation and angiogenic factor expression

Bone (New York, N.Y.), 2007-04, Vol.40 (4), p.1078-1087

2007

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Regulation by reactive oxygen species of interleukin-1β, nitric oxide and prostaglandin E2 production by human chondrocytes

Osteoarthritis and cartilage, 2002-07, Vol.10 (7), p.547-555

2002

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Modulation of rabbit articular chondrocyte (RAC) proliferation by TGF-β isoforms

Cell proliferation, 1995-04, Vol.28 (4), p.221-234

1995

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SOX9 exerts a bifunctional effect on type II collagen gene (COL2A1) expression in chondrocytes depending on the differentiation state

DNA and cell biology, 2003-02, Vol.22 (2), p.119-129

2003

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Rabbit Articular Chondrocytes (RAC) Express Distinct Transforming Growth Factor-β Receptor Phenotypes as a Function of Cell Cycle Phases

Experimental cell research, 1993-03, Vol.205 (1), p.165-170

1993

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Characterization of proteoglycans synthesized by rabbit articular chondrocytes in response to transforming growth factor-β (TGF-β)

Biochimica et biophysica acta, 1991-07, Vol.1093 (2), p.196-206

1991

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Rhein inhibits interleukin-1 beta-induced activation of MEK/ERK pathway and DNA binding of NF-kappa B and AP-1 in chondrocytes cultured in hypoxia: a potential mechanism for its disease-modifying effect in osteoarthritis

Inflammation, 2003-08, Vol.27 (4), p.233-246

2003

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The role of reactive oxygen species in homeostasis and degradation of cartilage

Rhein, the metabolite of diacerhein, reduces the proliferation of osteoarthritic chondrocytes and synoviocytes without inducing apoptosis

Immunohistochemical analysis of transforming growth factor beta isoforms and their receptors in human cartilage from normal and osteoarthritic femoral heads

Reactive oxygen species downregulate the expression of pro-inflammatory genes by human chondrocytes

Rhein, a diacerhein-derived metabolite, modulates the expression of matrix degrading enzymes and the cell proliferation of articular chondrocytes by inhibiting ERK and JNK-AP-1 dependent pathways

Comparative effects of IL-1β and hydrogen peroxide (H 2O 2) on catabolic and anabolic gene expression in juvenile bovine chondrocytes

Transforming growth factor β stimulates collagen and glycosaminoglycan biosynthesis in cultured rabbit articular chondrocytes

Semiquantitative reverse transcription-polymerase chain reaction analysis of syndecan-1 and -4 messages in cartilage and cultured chondrocytes from osteoarthritic joints

Differentiation potential of human muscle-derived cells towards chondrogenic phenotype in alginate beads culture

Sp1 and Sp3 Transcription Factors Mediate Interleukin-1β Down-regulation of Human Type II Collagen Gene Expression in Articular Chondrocytes

Expression of TGF-βs and their receptors is differentially modulated by reactive oxygen species and nitric oxide in human articular chondrocytes

L'agrécanase, enzyme-clé de la dégradation du cartilage dans l'arthrose ?

For intra-articular delivery of chondroitin sulfate

NFκB mediates IL‐1β‐induced down‐regulation of TβRII through the modulation of Sp3 expression

17 -Oestradiol up-regulates the expression of a functional UDP-glucose dehydrogenase in articular chondrocytes: comparison with effects of cytokines and growth factors

17β-Oestradiol up-regulates the expression of a functional UDP-glucose dehydrogenase in articular chondrocytes: comparison with effects of cytokines and growth factors

Sp3 represses the Sp1-mediated transactivation of the human COL2A1 gene in primary and de-differentiated chondrocytes

Cell density-dependent proliferative effects of transforming growth factor (TGF)-beta 1, beta 2, and beta 3 in human chondrosarcoma cells HCS-2/8 are associated with changes in the expression of TGF-beta receptor type I

Hypoxia affects mesenchymal stromal cell osteogenic differentiation and angiogenic factor expression

Regulation by reactive oxygen species of interleukin-1β, nitric oxide and prostaglandin E2 production by human chondrocytes

Modulation of rabbit articular chondrocyte (RAC) proliferation by TGF-β isoforms

SOX9 exerts a bifunctional effect on type II collagen gene (COL2A1) expression in chondrocytes depending on the differentiation state

Rabbit Articular Chondrocytes (RAC) Express Distinct Transforming Growth Factor-β Receptor Phenotypes as a Function of Cell Cycle Phases

Characterization of proteoglycans synthesized by rabbit articular chondrocytes in response to transforming growth factor-β (TGF-β)

Rhein inhibits interleukin-1 beta-induced activation of MEK/ERK pathway and DNA binding of NF-kappa B and AP-1 in chondrocytes cultured in hypoxia: a potential mechanism for its disease-modifying effect in osteoarthritis

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