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Single atom substitution in mouse protein kinase G eliminates oxidant sensing to cause hypertension

Nature medicine, 2012-02, Vol.18 (2), p.286-290

2012

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Blood Pressure–Lowering by the Antioxidant Resveratrol Is Counterintuitively Mediated by Oxidation of cGMP-Dependent Protein Kinase

Circulation (New York, N.Y.), 2019-07, Vol.140 (2), p.126-137

2019

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Vascular dysfunction caused by loss of Brn-3b/POU4F2 transcription factor in aortic vascular smooth muscle cells is linked to deregulation of calcium signalling pathways

Cell death & disease, 2023-11, Vol.14 (11), p.770-770, Article 770

2023

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Protection from hypertension in mice by the Mediterranean diet is mediated by nitro fatty acid inhibition of soluble epoxide hydrolase

Proceedings of the National Academy of Sciences - PNAS, 2014-06, Vol.111 (22), p.8167-8172

2014

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Distinct Regulatory Effects of Myeloid Cell and Endothelial Cell NAPDH Oxidase 2 on Blood Pressure

Circulation (New York, N.Y.), 2017-05, Vol.135 (22), p.2163-2177

2017

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Protein kinase G oxidation is a major cause of injury during sepsis

Proceedings of the National Academy of Sciences - PNAS, 2013-06, Vol.110 (24), p.9909-9913

2013

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Disulfide-activated protein kinase G Iα regulates cardiac diastolic relaxation and fine-tunes the Frank–Starling response

Nature communications, 2016-10, Vol.7 (1), p.13187-11, Article 13187

2016

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PKG1α oxidation negatively regulates food seeking behaviour and reward

Redox biology, 2019-02, Vol.21, p.101077-101077, Article 101077

2019

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BS17 Oxidation of protein kinase a regulatory subunit PKARIα alpha regulates vasodilation and blood pressure lowering

Heart (British Cardiac Society), 2023-06, Vol.109 (Suppl 3), p.A257-A258

2023

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UK-5099 does not inhibit the mitochondrial pyruvate carrier through irreversible adduction to cysteine 54 in MPC2

Free radical biology & medicine, 2023-11, Vol.208, p.S66

2023

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Protein Kinase G Iα Oxidation Paradoxically Underlies Blood Pressure Lowering by the Reductant Hydrogen Sulfide

Hypertension (Dallas, Tex. 1979), 2014-12, Vol.64 (6), p.1344-1351

2014

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cGMP-Dependent Activation of Protein Kinase G Precludes Disulfide Activation: Implications for Blood Pressure Control

Hypertension (Dallas, Tex. 1979), 2012-11, Vol.60 (5), p.1301-1308

2012

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Phosphodiesterase 5 Inhibition Limits Doxorubicin-induced Heart Failure by Attenuating Protein Kinase G Iα Oxidation

The Journal of biological chemistry, 2016-08, Vol.291 (33), p.17427-17436

2016

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Proof of Principle for a Novel Class of Antihypertensives That Target the Oxidative Activation of PKG Iα (Protein Kinase G Iα)

Hypertension (Dallas, Tex. 1979), 2017-09, Vol.70 (3), p.577-586

2017

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The role of oxidative stress in the development of myocardial diastolic dysfunction with diabetes mellitus

Journal of molecular and cellular cardiology, 2007-06, Vol.42 (6), p.S59-S59

2007

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Singlet molecular oxygen regulates vascular tone and blood pressure in inflammation

Nature (London), 2019-02, Vol.566 (7745), p.548-552

2019

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Long-lasting blood pressure lowering effects of nitrite are NO-independent and mediated by hydrogen peroxide, persulfides, and oxidation of protein kinase G1α redox signalling

Cardiovascular research, 2020-01, Vol.116 (1), p.51-62

2020

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Sulforaphane-mediated inhibition of SHP2 as a potential pharmacotherapy for Noonan syndrome

Free radical biology & medicine, 2018-11, Vol.128, p.S42

2018

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Nitroglycerin Fails to Lower Blood Pressure in Redox-Dead Cys42Ser PKG1α Knock-In Mouse

Circulation (New York, N.Y.), 2012-07, Vol.126 (3), p.287-295

2012

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Redox regulation of cGMP-dependent protein kinase Iα in the cardiovascular system

Frontiers in pharmacology, 2015-07, Vol.6, p.139-139

2015

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Oxidation of PKGIα mediates an endogenous adaptation to pulmonary hypertension

Proceedings of the National Academy of Sciences - PNAS, 2019-06, Vol.116 (26), p.13016-13025

2019

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Mitochondrial pyruvate carrier abundance mediates pathological cardiac hypertrophy

Nature metabolism, 2020-11, Vol.2 (11), p.1223-1231

2020

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Redox-Dead Protein Kinase a RIα knock-in Mouse Is Not Hypertensive But Has Increased Vascular Reactivity to Oxidants

Free radical biology & medicine, 2013-11, Vol.65, p.S82

2013

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PSS171 - Redox-Dead Protein Kinase a RIα knock-in Mouse Is Not Hypertensive But Has Increased Vascular Reactivity to Oxidants

Free radical biology & medicine, 2013-11, Vol.65, p.S82-S82

2013

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Response by Prysyazhna et al to Letter Regarding Article, “Blood Pressure–Lowering by the Antioxidant Resveratrol Is Counterintuitively Mediated by Oxidation of cGMP-Dependent Protein Kinase”

Circulation (New York, N.Y.), 2019-11, Vol.140 (22), p.e810-e811

2019

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Single atom substitution in mouse protein kinase G eliminates oxidant sensing to cause hypertension

Blood Pressure–Lowering by the Antioxidant Resveratrol Is Counterintuitively Mediated by Oxidation of cGMP-Dependent Protein Kinase

Vascular dysfunction caused by loss of Brn-3b/POU4F2 transcription factor in aortic vascular smooth muscle cells is linked to deregulation of calcium signalling pathways

Protection from hypertension in mice by the Mediterranean diet is mediated by nitro fatty acid inhibition of soluble epoxide hydrolase

Distinct Regulatory Effects of Myeloid Cell and Endothelial Cell NAPDH Oxidase 2 on Blood Pressure

Protein kinase G oxidation is a major cause of injury during sepsis

Disulfide-activated protein kinase G Iα regulates cardiac diastolic relaxation and fine-tunes the Frank–Starling response

PKG1α oxidation negatively regulates food seeking behaviour and reward

BS17 Oxidation of protein kinase a regulatory subunit PKARIα alpha regulates vasodilation and blood pressure lowering

UK-5099 does not inhibit the mitochondrial pyruvate carrier through irreversible adduction to cysteine 54 in MPC2

Protein Kinase G Iα Oxidation Paradoxically Underlies Blood Pressure Lowering by the Reductant Hydrogen Sulfide

cGMP-Dependent Activation of Protein Kinase G Precludes Disulfide Activation: Implications for Blood Pressure Control

Phosphodiesterase 5 Inhibition Limits Doxorubicin-induced Heart Failure by Attenuating Protein Kinase G Iα Oxidation

Proof of Principle for a Novel Class of Antihypertensives That Target the Oxidative Activation of PKG Iα (Protein Kinase G Iα)

The role of oxidative stress in the development of myocardial diastolic dysfunction with diabetes mellitus

Singlet molecular oxygen regulates vascular tone and blood pressure in inflammation

Long-lasting blood pressure lowering effects of nitrite are NO-independent and mediated by hydrogen peroxide, persulfides, and oxidation of protein kinase G1α redox signalling

Sulforaphane-mediated inhibition of SHP2 as a potential pharmacotherapy for Noonan syndrome

Nitroglycerin Fails to Lower Blood Pressure in Redox-Dead Cys42Ser PKG1α Knock-In Mouse

Redox regulation of cGMP-dependent protein kinase Iα in the cardiovascular system

Oxidation of PKGIα mediates an endogenous adaptation to pulmonary hypertension

Mitochondrial pyruvate carrier abundance mediates pathological cardiac hypertrophy

Redox-Dead Protein Kinase a RIα knock-in Mouse Is Not Hypertensive But Has Increased Vascular Reactivity to Oxidants

PSS171 - Redox-Dead Protein Kinase a RIα knock-in Mouse Is Not Hypertensive But Has Increased Vascular Reactivity to Oxidants

Response by Prysyazhna et al to Letter Regarding Article, “Blood Pressure–Lowering by the Antioxidant Resveratrol Is Counterintuitively Mediated by Oxidation of cGMP-Dependent Protein Kinase”

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