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Apoptosis and efficient repair of DNA damage protect human keratinocytes against UVB

Cell death and differentiation, 2003-06, Vol.10 (6), p.754-756

2003

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The cockayne syndrome B protein is essential for neuronal differentiation and neuritogenesis

Cell death & disease, 2014-05, Vol.5 (5), p.e1268-e1268

2014

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Lack of effect of caffeine post-treatment on X-ray-induced chromosomal aberrations in Werner's syndrome lymphoblastoid cell lines: a preliminary report

International journal of radiation biology, 1999, Vol.75 (11), p.1349-1355

Franchitto, L. Proietti De Santis, P. Pichierri, P. Mosesso, F. Palitti, A.

1999

Volltextzugriff (PDF)

Chromosome radiosensitivity in human G2 lymphocytes and cell-cycle progression

International journal of radiation biology, 1999, Vol.75 (5), p.621-627

PALITTI, P. PICHIERRI, A. FRANCHITTO, L. PROIETTI DE SANTIS, P. MOSESSO, F.

1999

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Inhibition of p53, p21 and Bax by pifithrin-α does not affect UV induced apoptotic response in CS-B cells

DNA repair, 2003-08, Vol.2 (8), p.891-900

2003

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Relationship between DNA lesions, DNA repair and chromosomal damage induced by acetaldehyde

Mutation research, 2009-03, Vol.662 (1), p.3-9

2009

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The ATPase domain but not the acidic region of Cockayne syndrome group B gene product is essential for DNA repair

Molecular biology of the cell, 1999-11, Vol.10 (11), p.3583-3594

1999

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Werner's syndrome lymphoblastoid cells are hypersensitive to topoisomerase II inhibitors in the G2 phase of the cell cycle

Mutation research, 2000-03, Vol.459 (2), p.123-133

2000

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Role of the ATPase domain of the Cockayne syndrome group B protein in UV induced apoptosis

Oncogene, 2000-01, Vol.19 (4), p.477-489

2000

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TFIIH Transcription Factor, a Target for the Rift Valley Hemorrhagic Fever Virus

Cell, 2004-02, Vol.116 (4), p.541-550

2004

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Basal Transcription Defect Discriminates between Xeroderma Pigmentosum and Trichothiodystrophy in XPD Patients

Molecular cell, 2003-06, Vol.11 (6), p.1635-1646

2003

Volltextzugriff (PDF)

Transcription coupled repair deficiency results in increased chromosomal aberrations and apoptotic death in the UV61 cell line, the Chinese hamster homologue of Cockayne’s syndrome B

Mutation research, 2001-03, Vol.485 (2), p.121-132

2001

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p8/TTD-A as a Repair-Specific TFIIH Subunit

Molecular cell, 2006-01, Vol.21 (2), p.215-226

2006

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CSB Is a Component of RNA Pol I Transcription

Molecular cell, 2002-10, Vol.10 (4), p.819-829

2002

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The use of an epithelial liver cell line from Chinese hamster, competent for xenobiotic metabolism, for the detection of clastogenic events

Mutation Research/Environmental Mutagenesis and Related Subjects, 1992, Vol.271 (2), p.177-177

1992

Volltextzugriff (PDF)

Transcription coupled repair efficiency determines the cell cycle progression and apoptosis after UV exposure in hamster cells

DNA repair, 2002-03, Vol.1 (3), p.209-223

2002

Volltextzugriff (PDF)

Telomere length measurement in tumor and non‐tumor cells as a valuable prognostic for tumor progression

Cancer genetics, 2019-10, Vol.238, p.50-61

2019

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Cells, Cultured
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Cricetinae
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Apoptosis and efficient repair of DNA damage protect human keratinocytes against UVB

The cockayne syndrome B protein is essential for neuronal differentiation and neuritogenesis

Lack of effect of caffeine post-treatment on X-ray-induced chromosomal aberrations in Werner's syndrome lymphoblastoid cell lines: a preliminary report

Chromosome radiosensitivity in human G2 lymphocytes and cell-cycle progression

Inhibition of p53, p21 and Bax by pifithrin-α does not affect UV induced apoptotic response in CS-B cells

Relationship between DNA lesions, DNA repair and chromosomal damage induced by acetaldehyde

The ATPase domain but not the acidic region of Cockayne syndrome group B gene product is essential for DNA repair

Werner's syndrome lymphoblastoid cells are hypersensitive to topoisomerase II inhibitors in the G2 phase of the cell cycle

Role of the ATPase domain of the Cockayne syndrome group B protein in UV induced apoptosis

TFIIH Transcription Factor, a Target for the Rift Valley Hemorrhagic Fever Virus

Basal Transcription Defect Discriminates between Xeroderma Pigmentosum and Trichothiodystrophy in XPD Patients

Transcription coupled repair deficiency results in increased chromosomal aberrations and apoptotic death in the UV61 cell line, the Chinese hamster homologue of Cockayne’s syndrome B

p8/TTD-A as a Repair-Specific TFIIH Subunit

CSB Is a Component of RNA Pol I Transcription

The use of an epithelial liver cell line from Chinese hamster, competent for xenobiotic metabolism, for the detection of clastogenic events

Transcription coupled repair efficiency determines the cell cycle progression and apoptosis after UV exposure in hamster cells

Telomere length measurement in tumor and non‐tumor cells as a valuable prognostic for tumor progression

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