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The Italian Registry of GH Treatment: electronic Clinical Report Form (e-CRF) and web-based platform for the national database of GH prescriptions

Journal of endocrinological investigation, 2019-07, Vol.42 (7), p.769-777

2019

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Role of galectin-3 as a receptor for advanced glycosylation end products

Kidney international, 2000-09, Vol.58 (S77), p.S31-S39

2000

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Upregulation of mesangial growth factor and extracellular matrix synthesis by advanced glycation end products via a receptor-mediated mechanism

Diabetes (New York, N.Y.), 1997-11, Vol.46 (11), p.1881-1887

1997

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Burden of T1DM and T2DM among adolescents in Western Europe, 1990-2019: results from the GBD 2019

European journal of public health, 2023-10, Vol.33 (Supplement_2)

2023

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The diabetic milieu modulates the advanced glycation end product-receptor complex in the mesangium by inducing or upregulating galectin-3 expression

Diabetes (New York, N.Y.), 2000-07, Vol.49 (7), p.1249-1257

2000

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Long-COVID in Italy: a country perspective

European journal of public health, 2023-10, Vol.33 (Supplement_2)

2023

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Mechanisms of glucose-enhanced extracellular matrix accumulation in rat glomerular mesangial cells

Diabetes (New York, N.Y.), 1994-03, Vol.43 (3), p.478-490

1994

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Mechanisms of glucose-enhanced extracellular matrix accumulation in rat glomerular mesangial cells

Diabetes (New York, N.Y.), 1994-03, Vol.43 (3), p.478-490

1994

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The Burden of Type 1 and Type 2 Diabetes Among Adolescents and Young Adults in 24 Western European Countries, 1990-2019: Results From the Global Burden of Disease Study 2019

International journal of public health, 2024-02, Vol.68, p.1606491-1606491

2024

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Galectin‐3/AGE‐receptor 3 knockout mice show accelerated AGE‐induced glomerular injury: evidence for a protective role of galectin‐3 as an AGE receptor

The FASEB journal, 2004-11, Vol.18 (14), p.1773-1775

2004

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Accelerated diabetic glomerulopathy in galectin‐3/AGE receptor 3 knockout mice

The FASEB journal, 2001-11, Vol.15 (13), p.2471-2479

2001

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Early, but not advanced, glomerulopathy is reversed by pancreatic islet transplants in experimental diabetic rats: correlation with glomerular extracellular matrix mRNA levels

Diabetes (New York, N.Y.), 1997-07, Vol.46 (7), p.1198-1206

1997

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Increased activity of the insulin-like growth factor system in mesangial cells cultured in high glucose conditions. Relation to glucose-enhanced extracellular matrix production

Diabetologia, 1996-07, Vol.39 (7), p.775-784

1996

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High glucose inhibits cytosolic calcium signaling in cultured rat mesangial cells

Kidney international, 1993-03, Vol.43 (3), p.585-591

1993

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Development of age-dependent glomerular lesions in galectin-3/AGE-receptor-3 knockout mice

American journal of physiology. Renal physiology, 2005-09, Vol.289 (3), p.F611-F621

2005

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Oxidative stress in diabetes-induced endothelial dysfunction involvement of nitric oxide and protein kinase C

Free radical biology & medicine, 2003-09, Vol.35 (6), p.683-694

2003

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Purinergic modulation of mesangial extracellular matrix production: Role in diabetic and other glomerular diseases

Kidney international, 2005-03, Vol.67 (3), p.875-885

2005

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Role of galectin-3 in diabetic nephropathy

Journal of the American Society of Nephrology, 2003-08, Vol.14 (8 Suppl 3), p.S264-S270

2003

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Eicosanoid synthesis in peripheral blood monocytes: A marker of disease activity in lupus nephritis

American journal of kidney diseases, 1998-11, Vol.32 (5), p.778-784

1998

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Upregulation of mesangial growth factor and extracellular matrix synthesis by advanced glycation end products via a receptor-mediated mechanism

Diabetes (New York, N.Y.), 1997-11, Vol.46 (11), p.1881-1887

1997

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High glucose level inhibits capacitative Ca 2 + influx in cultured rat mesangial cells by a protein kinase C-dependent mechanism

Diabetologia, 1997-04, Vol.40 (5), p.521-527

1997

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Regulatory role of eicosanoids in extracellular matrix overproduction induced by long-term exposure to high glucose in cultured rat mesangial cells

Diabetologia, 1996-09, Vol.39 (9), p.1055-1062

1996

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Autocrine and paracrine mechanisms in the early stages of diabetic nephropathy

Journal of endocrinological investigation, 1999-10, Vol.22 (9), p.708-735

1999

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Development of diabetic nephropathy in the Milan normotensive strain, but not in the Milan hypertensive strain: Possible permissive role of hemodynamics

Kidney international, 2005-04, Vol.67 (4), p.1440-1452

2005

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D-lysine effectively decreases the non-enzymic glycation of proteins in vitro

Clinical chemistry (Baltimore, Md.), 1989-03, Vol.35 (3), p.384-387

1989

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The Italian Registry of GH Treatment: electronic Clinical Report Form (e-CRF) and web-based platform for the national database of GH prescriptions

Role of galectin-3 as a receptor for advanced glycosylation end products

Upregulation of mesangial growth factor and extracellular matrix synthesis by advanced glycation end products via a receptor-mediated mechanism

Burden of T1DM and T2DM among adolescents in Western Europe, 1990-2019: results from the GBD 2019

The diabetic milieu modulates the advanced glycation end product-receptor complex in the mesangium by inducing or upregulating galectin-3 expression

Long-COVID in Italy: a country perspective

Mechanisms of glucose-enhanced extracellular matrix accumulation in rat glomerular mesangial cells

Mechanisms of glucose-enhanced extracellular matrix accumulation in rat glomerular mesangial cells

The Burden of Type 1 and Type 2 Diabetes Among Adolescents and Young Adults in 24 Western European Countries, 1990-2019: Results From the Global Burden of Disease Study 2019

Galectin‐3/AGE‐receptor 3 knockout mice show accelerated AGE‐induced glomerular injury: evidence for a protective role of galectin‐3 as an AGE receptor

Accelerated diabetic glomerulopathy in galectin‐3/AGE receptor 3 knockout mice

Early, but not advanced, glomerulopathy is reversed by pancreatic islet transplants in experimental diabetic rats: correlation with glomerular extracellular matrix mRNA levels

Increased activity of the insulin-like growth factor system in mesangial cells cultured in high glucose conditions. Relation to glucose-enhanced extracellular matrix production

High glucose inhibits cytosolic calcium signaling in cultured rat mesangial cells

Development of age-dependent glomerular lesions in galectin-3/AGE-receptor-3 knockout mice

Oxidative stress in diabetes-induced endothelial dysfunction involvement of nitric oxide and protein kinase C

Purinergic modulation of mesangial extracellular matrix production: Role in diabetic and other glomerular diseases

Role of galectin-3 in diabetic nephropathy

Eicosanoid synthesis in peripheral blood monocytes: A marker of disease activity in lupus nephritis

Upregulation of mesangial growth factor and extracellular matrix synthesis by advanced glycation end products via a receptor-mediated mechanism

High glucose level inhibits capacitative Ca 2 + influx in cultured rat mesangial cells by a protein kinase C-dependent mechanism

Regulatory role of eicosanoids in extracellular matrix overproduction induced by long-term exposure to high glucose in cultured rat mesangial cells

Autocrine and paracrine mechanisms in the early stages of diabetic nephropathy

Development of diabetic nephropathy in the Milan normotensive strain, but not in the Milan hypertensive strain: Possible permissive role of hemodynamics

D-lysine effectively decreases the non-enzymic glycation of proteins in vitro

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