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Immune escape as a fundamental trait of cancer: focus on IDO

Oncogene, 2008-06, Vol.27 (28), p.3889-3900

Prendergast, G C

2008

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Indoleamine 2,3-dioxygenase as a modifier of pathogenic inflammation in cancer and other inflammation-associated diseases

Current medicinal chemistry, 2011, Vol.18 (15), p.2257-2262

2011

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A key in vivo antitumor mechanism of action of natural product-based brassinins is inhibition of indoleamine 2,3-dioxygenase

Oncogene, 2008-05, Vol.27 (20), p.2851-2857

2008

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RhoB in cancer suppression

Histology and histopathology, 2006-02, Vol.21 (2), p.213

2006

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RhoB Is Dispensable for Mouse Development, but It Modifies Susceptibility to Tumor Formation as Well as Cell Adhesion and Growth Factor Signaling in Transformed Cells

Molecular and Cellular Biology, 2001-10, Vol.21 (20), p.6906-6912

2001

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Necrostatin-1 analogues: critical issues on the specificity, activity and in vivo use in experimental disease models

Cell death & disease, 2012-11, Vol.3 (11), p.e437-e437

2012

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RhoB facilitates c-Myc turnover by supporting efficient nuclear accumulation of GSK-3

Oncogene, 2006-03, Vol.25 (9), p.1281-1289

2006

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The polyproline region of p53 is required to activate apoptosis but not growth arrest

Oncogene, 1997-08, Vol.15 (8), p.887-898

1997

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A role for candidate tumor-suppressor gene TCEAL7 in the regulation of c-Myc activity, cyclin D1 levels and cellular transformation

Oncogene, 2008-12, Vol.27 (58), p.7223-7234

2008

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Inhibition of indoleamine 2,3-dioxygenase, an immunoregulatory target of the cancer suppression gene Bin1 , potentiates cancer chemotherapy

Nature medicine, 2005-03, Vol.11 (3), p.312-319

2005

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Farnesyltransferase inhibitors: mechanism and applications

Expert opinion on investigational drugs, 2001-12, Vol.10 (12), p.2105-2116

2001

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RhoB Alteration Is Necessary for Apoptotic and Antineoplastic Responses to Farnesyltransferase Inhibitors

Molecular and Cellular Biology, 2000-08, Vol.20 (16), p.6105-6113

2000

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BIN1 is a novel MYC-interacting protein with features of a tumour suppressor

Nature genetics, 1996-09, Vol.14 (1), p.69-77

1996

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Mechanisms of apoptosis by c-Myc

Oncogene, 1999-05, Vol.18 (19), p.2967-2987

Prendergast, G C

1999

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Recognition by Max of its cognate DNA through a dimeric b/HLH/Z domain

Nature (London), 1993-05, Vol.363 (6424), p.38-45

1993

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Actin' up: RhoB in cancer and apoptosis

Nature reviews. Cancer, 2001-11, Vol.1 (2), p.162-168

Prendergast, George C

2001

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Farnesyl transferase inhibitors induce apoptosis of Ras-transformed cells denied substratum attachment

Cancer research (Chicago, Ill.), 1997-02, Vol.57 (4), p.708-713

1997

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RhoB and Actin Polymerization Coordinate Src Activation with Endosome-Mediated Delivery to the Membrane

Developmental cell, 2004-12, Vol.7 (6), p.855-869

2004

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Methylation-Sensitive Sequence-Specific DNA Binding by the c-Myc Basic Region

Science (American Association for the Advancement of Science), 1991-01, Vol.251 (4990), p.186-189

1991

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Evidence that farnesyltransferase inhibitors suppress Ras transformation by interfering with Rho activity

Molecular and Cellular Biology, 1995-12, Vol.15 (12), p.6613-6622

1995

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Bin1 functionally interacts with Myc and inhibits cell proliferation via multiple mechanisms

Oncogene, 1999-06, Vol.18 (24), p.3564-3573

1999

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Association of Myn, the murine homolog of Max, with c-Myc stimulates methylation-sensitive DNA binding and ras cotransformation

Cell, 1991-05, Vol.65 (3), p.395-407

1991

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Bin1 mediates apoptosis by c-Myc in transformed primary cells

Cancer research (Chicago, Ill.), 2001-04, Vol.61 (7), p.3151-3156

2001

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Farnesyltransferase inhibitors define a role for RhoB in controlling neoplastic pathophysiology

Histology and histopathology, 2001-01, Vol.16 (1), p.269

Prendergast, G C

2001

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Geranylgeranylated RhoB mediates suppression of human tumor cell growth by farnesyltransferase inhibitors

Cancer research (Chicago, Ill.), 1999-11, Vol.59 (21), p.5492-5496

1999

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Immune escape as a fundamental trait of cancer: focus on IDO

Indoleamine 2,3-dioxygenase as a modifier of pathogenic inflammation in cancer and other inflammation-associated diseases

A key in vivo antitumor mechanism of action of natural product-based brassinins is inhibition of indoleamine 2,3-dioxygenase

RhoB in cancer suppression

RhoB Is Dispensable for Mouse Development, but It Modifies Susceptibility to Tumor Formation as Well as Cell Adhesion and Growth Factor Signaling in Transformed Cells

Necrostatin-1 analogues: critical issues on the specificity, activity and in vivo use in experimental disease models

RhoB facilitates c-Myc turnover by supporting efficient nuclear accumulation of GSK-3

The polyproline region of p53 is required to activate apoptosis but not growth arrest

A role for candidate tumor-suppressor gene TCEAL7 in the regulation of c-Myc activity, cyclin D1 levels and cellular transformation

Inhibition of indoleamine 2,3-dioxygenase, an immunoregulatory target of the cancer suppression gene Bin1 , potentiates cancer chemotherapy

Farnesyltransferase inhibitors: mechanism and applications

RhoB Alteration Is Necessary for Apoptotic and Antineoplastic Responses to Farnesyltransferase Inhibitors

BIN1 is a novel MYC-interacting protein with features of a tumour suppressor

Mechanisms of apoptosis by c-Myc

Recognition by Max of its cognate DNA through a dimeric b/HLH/Z domain

Actin' up: RhoB in cancer and apoptosis

Farnesyl transferase inhibitors induce apoptosis of Ras-transformed cells denied substratum attachment

RhoB and Actin Polymerization Coordinate Src Activation with Endosome-Mediated Delivery to the Membrane

Methylation-Sensitive Sequence-Specific DNA Binding by the c-Myc Basic Region

Evidence that farnesyltransferase inhibitors suppress Ras transformation by interfering with Rho activity

Bin1 functionally interacts with Myc and inhibits cell proliferation via multiple mechanisms

Association of Myn, the murine homolog of Max, with c-Myc stimulates methylation-sensitive DNA binding and ras cotransformation

Bin1 mediates apoptosis by c-Myc in transformed primary cells

Farnesyltransferase inhibitors define a role for RhoB in controlling neoplastic pathophysiology

Geranylgeranylated RhoB mediates suppression of human tumor cell growth by farnesyltransferase inhibitors

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