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The biology and therapeutic targeting of the proprotein convertases

Nature reviews. Drug discovery, 2012-05, Vol.11 (5), p.367-383

2012

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The Proprotein Convertases in Hypercholesterolemia and Cardiovascular Diseases: Emphasis on Proprotein Convertase Subtilisin/Kexin 9

Pharmacological reviews, 2017-01, Vol.69 (1), p.33-52

2017

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Transcriptome Analysis Reveals Nonfoamy Rather Than Foamy Plaque Macrophages Are Proinflammatory in Atherosclerotic Murine Models

Circulation research, 2018-10, Vol.123 (10), p.1127-1142

2018

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Proprotein convertase subtilisin/kexin type 9 (PCSK9) can mediate degradation of the low density lipoprotein receptor-related protein 1 (LRP-1)

PloS one, 2013-05, Vol.8 (5), p.e64145-e64145

2013

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Gene Inactivation of Proprotein Convertase Subtilisin/Kexin Type 9 Reduces Atherosclerosis in Mice

Circulation (New York, N.Y.), 2012-02, Vol.125 (7), p.894-901

2012

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Circulating proprotein convertase subtilisin/kexin 9 (PCSK9) regulates VLDLR protein and triglyceride accumulation in visceral adipose tissue

Arteriosclerosis, thrombosis, and vascular biology, 2011-04, Vol.31 (4), p.785

2011

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Proprotein convertase subtilisin/kexin type 9 (PCSK9): Hepatocyte‐specific low‐density lipoprotein receptor degradation and critical role in mouse liver regeneration

Hepatology (Baltimore, Md.), 2008-08, Vol.48 (2), p.646-654

2008

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The Proprotein Convertase PCSK9 Induces the Degradation of Low Density Lipoprotein Receptor (LDLR) and Its Closest Family Members VLDLR and ApoER2

The Journal of biological chemistry, 2008-01, Vol.283 (4), p.2363-2372

2008

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Sortilin enhances secretion of apolipoprotein(a) through effects on apolipoprotein B secretion and promotes uptake of lipoprotein(a)

Journal of lipid research, 2022-06, Vol.63 (6), p.100216-100216, Article 100216

2022

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Implication of the proprotein convertases in iron homeostasis: Proprotein convertase 7 sheds human transferrin receptor 1 and furin activates hepcidin

Hepatology (Baltimore, Md.), 2013-06, Vol.57 (6), p.2514-2524

2013

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PCSK9-deficient mice exhibit impaired glucose tolerance and pancreatic islet abnormalities

FEBS letters, 2010-02, Vol.584 (4), p.701-706

2010

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Proprotein Convertase Subtilisin/Kexin Type 9 Deficiency Reduces Melanoma Metastasis in Liver

Neoplasia (New York, N.Y.), 2012-12, Vol.14 (12), p.1122-IN5

2012

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Proprotein convertase 7 (PCSK7) reduces apoA‐V levels

The FEBS journal, 2020-08, Vol.287 (16), p.3565-3578

2020

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PCSK9 deficiency unmasks a sex- and tissue-specific subcellular distribution of the LDL and VLDL receptors in mice[S]

Journal of lipid research, 2015-11, Vol.56 (11), p.2133-2142

2015

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PCSK9 reduces the protein levels of the LDL receptor in mouse brain during development and after ischemic stroke[S]

Journal of lipid research, 2011-07, Vol.52 (7), p.1383-1391

2011

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Corrigendum: PCSK9 is not secreted from mature differentiated intestinal cells

Journal of lipid research, 2022-04, Vol.63 (4), p.100186-100186, Article 100186

2022

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A single domain antibody against the Cys- and His-rich domain of PCSK9 and evolocumab exhibit different inhibition mechanisms in humanized PCSK9 mice

Biological chemistry, 2018-11, Vol.399 (12), p.1363

2018

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Proprotein Convertase Subtilisin/Kexin Type 9 (PCSK9) Single Domain Antibodies Are Potent Inhibitors of Low Density Lipoprotein Receptor Degradation

The Journal of biological chemistry, 2016-08, Vol.291 (32), p.16659-16671

2016

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Endoplasmic Reticulum Stress and Ca2+ Depletion Differentially Modulate the Sterol Regulatory Protein PCSK9 to Control Lipid Metabolism

The Journal of biological chemistry, 2017-01, Vol.292 (4), p.1510-1523

2017

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Disruption of the expression of the proprotein convertase PC7 reduces BDNF production and affects learning and memory in mice

Proceedings of the National Academy of Sciences - PNAS, 2013-10, Vol.110 (43), p.17362-17367

2013

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The Cellular Trafficking of the Secretory Proprotein Convertase PCSK9 and Its Dependence on the LDLR

Traffic (Copenhagen, Denmark), 2007-06, Vol.8 (6), p.718-732

2007

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PCSK9 is not secreted from mature differentiated intestinal cells

Journal of lipid research, 2021-01, Vol.62, p.100096-100096, Article 100096

2021

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Identification and characterization of new gain-of-function mutations in the PCSK9 gene responsible for autosomal dominant hypercholesterolemia

Atherosclerosis, 2012-08, Vol.223 (2), p.394-400

2012

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Pcsk9 knockout exacerbates diet-induced non-alcoholic steatohepatitis, fibrosis and liver injury in mice

JHEP reports, 2019-12, Vol.1 (6), p.418-429

2019

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The LDLR deficient mouse as a model for aortic calcification and quantification by micro-computed tomography

Atherosclerosis, 2011-12, Vol.219 (2), p.455-462

2011

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The biology and therapeutic targeting of the proprotein convertases

The Proprotein Convertases in Hypercholesterolemia and Cardiovascular Diseases: Emphasis on Proprotein Convertase Subtilisin/Kexin 9

Transcriptome Analysis Reveals Nonfoamy Rather Than Foamy Plaque Macrophages Are Proinflammatory in Atherosclerotic Murine Models

Proprotein convertase subtilisin/kexin type 9 (PCSK9) can mediate degradation of the low density lipoprotein receptor-related protein 1 (LRP-1)

Gene Inactivation of Proprotein Convertase Subtilisin/Kexin Type 9 Reduces Atherosclerosis in Mice

Circulating proprotein convertase subtilisin/kexin 9 (PCSK9) regulates VLDLR protein and triglyceride accumulation in visceral adipose tissue

Proprotein convertase subtilisin/kexin type 9 (PCSK9): Hepatocyte‐specific low‐density lipoprotein receptor degradation and critical role in mouse liver regeneration

The Proprotein Convertase PCSK9 Induces the Degradation of Low Density Lipoprotein Receptor (LDLR) and Its Closest Family Members VLDLR and ApoER2

Sortilin enhances secretion of apolipoprotein(a) through effects on apolipoprotein B secretion and promotes uptake of lipoprotein(a)

Implication of the proprotein convertases in iron homeostasis: Proprotein convertase 7 sheds human transferrin receptor 1 and furin activates hepcidin

PCSK9-deficient mice exhibit impaired glucose tolerance and pancreatic islet abnormalities

Proprotein Convertase Subtilisin/Kexin Type 9 Deficiency Reduces Melanoma Metastasis in Liver

Proprotein convertase 7 (PCSK7) reduces apoA‐V levels

PCSK9 deficiency unmasks a sex- and tissue-specific subcellular distribution of the LDL and VLDL receptors in mice[S]

PCSK9 reduces the protein levels of the LDL receptor in mouse brain during development and after ischemic stroke[S]

Corrigendum: PCSK9 is not secreted from mature differentiated intestinal cells

A single domain antibody against the Cys- and His-rich domain of PCSK9 and evolocumab exhibit different inhibition mechanisms in humanized PCSK9 mice

Proprotein Convertase Subtilisin/Kexin Type 9 (PCSK9) Single Domain Antibodies Are Potent Inhibitors of Low Density Lipoprotein Receptor Degradation

Endoplasmic Reticulum Stress and Ca2+ Depletion Differentially Modulate the Sterol Regulatory Protein PCSK9 to Control Lipid Metabolism

Disruption of the expression of the proprotein convertase PC7 reduces BDNF production and affects learning and memory in mice

The Cellular Trafficking of the Secretory Proprotein Convertase PCSK9 and Its Dependence on the LDLR

PCSK9 is not secreted from mature differentiated intestinal cells

Identification and characterization of new gain-of-function mutations in the PCSK9 gene responsible for autosomal dominant hypercholesterolemia

Pcsk9 knockout exacerbates diet-induced non-alcoholic steatohepatitis, fibrosis and liver injury in mice

The LDLR deficient mouse as a model for aortic calcification and quantification by micro-computed tomography

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