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Acetylcholinesterase Inhibition Attenuates the Development of Hypertension and Inflammation in Spontaneously Hypertensive Rats

American journal of hypertension, 2015-10, Vol.28 (10), p.1201-1208

2015

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Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure

American journal of physiology. Regulatory, integrative and comparative physiology, 2013-10, Vol.305 (8), p.R908-R916

2013

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Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis

PloS one, 2013-07, Vol.8 (7), p.e68809-e68809

2013

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Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure

PloS one, 2017-12, Vol.12 (12), p.e0189469

2017

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Pyridostigmine restores cardiac autonomic balance after small myocardial infarction in mice

PloS one, 2014-08, Vol.9 (8), p.e104476-e104476

2014

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Coronary microvascular disease in chronic Chagas cardiomyopathy including an overview on history, pathology, and other proposed pathogenic mechanisms

PLoS neglected tropical diseases, 2010-08, Vol.4 (8), p.e674

2010

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Aspirin treatment of mice infected with Trypanosoma cruzi and implications for the pathogenesis of Chagas disease

PloS one, 2011-02, Vol.6 (2), p.e16959

2011

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Matrix Metalloproteinase Inhibition Improves Cardiac Dysfunction and Remodeling in 2-Kidney, 1-Clip Hypertension

Journal of cardiac failure, 2010-07, Vol.16 (7), p.599-608

2010

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5-Lipoxygenase is a key determinant of acute myocardial inflammation and mortality during Trypanosoma cruzi infection

Microbes and infection, 2010-08, Vol.12 (8), p.587-597

2010

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MicroRNA Profiling Using a PCR-Based Method

Methods in molecular biology (Clifton, N.J.), 2023, Vol.2595, p.159

2023

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Sepsis: going to the heart of the matter

Pathobiology (Basel), 2013-01, Vol.80 (2), p.70-86

2013

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Imbalance between matrix metalloproteinases and tissue inhibitor of metalloproteinases in hypertensive vascular remodeling

Matrix biology, 2010-04, Vol.29 (3), p.194-201

2010

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Turbulent blood flow plays an essential localizing role in the development of atherosclerotic lesions in experimentally induced hypercholesterolaemia in rats

International journal of experimental pathology, 2008-02, Vol.89 (1), p.72-80

2008

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β1-Adrenergic blockers exert antioxidant effects, reduce matrix metalloproteinase activity, and improve renovascular hypertension-induced cardiac hypertrophy

Free radical biology & medicine, 2014-08, Vol.73, p.308-317

2014

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Elastase‐2, an angiotensin II‐generating enzyme, contributes to increased angiotensin II in resistance arteries of mice with myocardial infarction

British journal of pharmacology, 2017-05, Vol.174 (10), p.1104-1115

2017

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Changes in hemodynamic and neurohumoral control cause cardiac damage in one-kidney, one-clip hypertensive mice

American journal of physiology. Regulatory, integrative and comparative physiology, 2008-12, Vol.295 (6), p.R1904-R1913

2008

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Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-β in renovascular hypertension-induced cardiac hypertrophy

Experimental and molecular pathology, 2013-02, Vol.94 (1), p.1-9

2013

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Tempol inhibits TGF-β and MMPs upregulation and prevents cardiac hypertensive changes

International journal of cardiology, 2013-04, Vol.165 (1), p.165-173

2013

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Prospective analysis of myocardial strain through the evolution of Chagas disease in the hamster animal model

The International Journal of Cardiovascular Imaging, 2022-01, Vol.38 (1), p.117-129

2022

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Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery

Life sciences (1973), 2016-01, Vol.145, p.9-18

2016

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Dantrolene improves in vitro structural changes induced by serum from Trypanosoma cruzi-infected mice

Parasitology research (1987), 2017, Vol.116 (1), p.429-433

2017

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Myocardial structural changes in long-term human severe sepsis/septic shock may be responsible for cardiac dysfunction

Shock (Augusta, Ga.), 2007-01, Vol.27 (1), p.10-18

2007

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Reduced expression of adherens and gap junction proteins can have a fundamental role in the development of heart failure following cardiac hypertrophy in rats

Experimental and molecular pathology, 2016-02, Vol.100 (1), p.167-176

2016

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Chagas Heart Disease: Report on Recent Developments

Cardiology in review, 2012-03, Vol.20 (2), p.53-65

2012

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Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy

European journal of pharmacology, 2011-11, Vol.670 (2-3), p.541-553

2011

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Acetylcholinesterase Inhibition Attenuates the Development of Hypertension and Inflammation in Spontaneously Hypertensive Rats

Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure

Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis

Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure

Pyridostigmine restores cardiac autonomic balance after small myocardial infarction in mice

Coronary microvascular disease in chronic Chagas cardiomyopathy including an overview on history, pathology, and other proposed pathogenic mechanisms

Aspirin treatment of mice infected with Trypanosoma cruzi and implications for the pathogenesis of Chagas disease

Matrix Metalloproteinase Inhibition Improves Cardiac Dysfunction and Remodeling in 2-Kidney, 1-Clip Hypertension

5-Lipoxygenase is a key determinant of acute myocardial inflammation and mortality during Trypanosoma cruzi infection

MicroRNA Profiling Using a PCR-Based Method

Sepsis: going to the heart of the matter

Imbalance between matrix metalloproteinases and tissue inhibitor of metalloproteinases in hypertensive vascular remodeling

Turbulent blood flow plays an essential localizing role in the development of atherosclerotic lesions in experimentally induced hypercholesterolaemia in rats

β1-Adrenergic blockers exert antioxidant effects, reduce matrix metalloproteinase activity, and improve renovascular hypertension-induced cardiac hypertrophy

Elastase‐2, an angiotensin II‐generating enzyme, contributes to increased angiotensin II in resistance arteries of mice with myocardial infarction

Changes in hemodynamic and neurohumoral control cause cardiac damage in one-kidney, one-clip hypertensive mice

Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-β in renovascular hypertension-induced cardiac hypertrophy

Tempol inhibits TGF-β and MMPs upregulation and prevents cardiac hypertensive changes

Prospective analysis of myocardial strain through the evolution of Chagas disease in the hamster animal model

Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery

Dantrolene improves in vitro structural changes induced by serum from Trypanosoma cruzi-infected mice

Myocardial structural changes in long-term human severe sepsis/septic shock may be responsible for cardiac dysfunction

Reduced expression of adherens and gap junction proteins can have a fundamental role in the development of heart failure following cardiac hypertrophy in rats

Chagas Heart Disease: Report on Recent Developments

Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy

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