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IP3 receptor function and localization in myotubes: an unexplored Ca2+ signaling pathway in skeletal muscle

Journal of cell science, 2001-10, Vol.114 (20), p.3673-3683

2001

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Absence of the β Subunit (cchb1) of the Skeletal Muscle Dihydropyridine Receptor Alters Expression of the α1Subunit and Eliminates Excitation-Contraction Coupling

Proceedings of the National Academy of Sciences - PNAS, 1996-11, Vol.93 (24), p.13961-13966

1996

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Restoration of excitation-contraction coupling and slow calcium current in dysgenic muscle by dihydropyridine receptor complementary DNA

Nature (London), 1988-11, Vol.336 (6195), p.134-139

1988

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Dihydropyridine receptors as voltage sensors for a depolarization-evoked, IP3R-mediated, slow calcium signal in skeletal muscle cells

The Journal of general physiology, 2003-01, Vol.121 (1), p.3-16

2003

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IP3 receptors, IP3 transients, and nucleus-associated Ca2+ signals in cultured skeletal muscle

American Journal of Physiology: Cell Physiology, 2000-05, Vol.278 (5), p.C998

2000

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A lethal mutation in mice eliminates the slow calcium current in skeletal muscle cells

Nature (London), 1986-03, Vol.320 (6058), p.168-170

1986

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IP3 Receptors and Associated Ca2+ Signals Localize to Satellite Cells and to Components of the Neuromuscular Junction in Skeletal Muscle

The Journal of neuroscience, 2003-09, Vol.23 (23), p.8185-8192

2003

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Formation of Triads without the Dihydropyridine Receptor α Subunits in Cell Lines from Dysgenic Skeletal Muscle

The Journal of cell biology, 1996-07, Vol.134 (2), p.375-387

1996

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Muscular dysgenesis : a model system for studying skeletal muscle development

The FASEB journal, 1990-07, Vol.4 (10), p.2798-2808

POWELL, J. A

1990

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The development of motoneurons in the embryonic spinal cord of the mouse mutant, muscular dysgenesis ( [formula omitted]): Survival, morphology, and biochemical differentiation

Developmental biology, 1986-04, Vol.114 (2), p.426-436

1986

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IP 3 Receptors and Associated Ca 2+ Signals Localize to Satellite Cells and to Components of the Neuromuscular Junction in Skeletal Muscle

The Journal of neuroscience, 2003-09, Vol.23 (23), p.8185-8192

2003

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Electrical properties of normal and dysgenib mouse skeletal muscle in culture

Journal of cellular physiology, 1973-08, Vol.82 (1), p.21-38

1973

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Differences in both inositol 1,4,5-trisphosphate mass and inositol 1,4,5-trisphosphate receptors between normal and dystrophic skeletal muscle cell lines

Muscle & nerve, 1998-07, Vol.21 (7), p.902-909

1998

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Triad Formation: Organization and Function of the Sarcoplasmic Reticulum Calcium Release Channel and Triadin in Normal and Dysgenic Muscle in Vitro

The Journal of cell biology, 1993-12, Vol.123 (5), p.1161-1174

1993

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IP 3 receptors, IP 3 transients, and nucleus-associated Ca 2+ signals in cultured skeletal muscle

American Journal of Physiology: Cell Physiology, 2000-05, Vol.278 (5), p.C998-C1010

2000

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Fine structure of prenatal and early postnatal dystrophic mouse muscle

Journal of the neurological sciences, 1975, Vol.24 (1), p.109-126

1975

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IP3 receptors, IP3 transients, and nucleus-associated Ca2+ signals in cultured skeletal muscle

American Journal of Physiology: Cell Physiology, 2000-05, Vol.47 (5), p.C998-C1010

2000

Volltextzugriff (PDF)

Muscular dysgenesis: a model system for studying skeletal muscle development

The FASEB journal, 1990-07, Vol.4 (10), p.2798-2808

Powell, Jeanne A.

1990

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Molecular Origin of the L-Type Ca2+ Current of Skeletal Muscle Myotubes Selectively Deficient in Dihydropyridine Receptor β1a Subunit

Biophysical journal, 1998-07, Vol.75 (1), p.207-217

1998

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Coordinated development of myofibrils, sarcoplasmic reticulum and transverse tubules in normal and dysgenic mouse skeletal muscle, in vivo and in vitro

Developmental biology, 1992-04, Vol.150 (2), p.266-280

1992

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Molecular Origin of the L-Type Ca 2+ Current of Skeletal Muscle Myotubes Selectively Deficient in Dihydropyridine Receptor β 1a Subunit

Biophysical journal, 1998, Vol.75 (1), p.207-217

1998

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Modification of mitochondrial metabolism in fibroblasts from mice with a skeletal muscle mutation (muscular dysgenesis)

Differentiation (London), 2000-06, Vol.65 (5), p.261-270

2000

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Modification of mitochondrial metabolism in fibroblasts from mice with a skeletal muscle mutation (muscular dysgenesis) : Evidence of embryonic communication between myoblasts and fibroblasts

Differentiation (London), 2000-05, Vol.65 (5), p.261-270

2000

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Rescue of excitation-contraction coupling in dysgenic muscle by addition of fibroblasts in vitro

Neuron (Cambridge, Mass.), 1989-04, Vol.2 (4), p.1341-1350

1989

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Absence of the beta subunit (cchb1) of the skeletal muscle dihydropyridine receptor alters expression of the alpha 1 subunit and eliminates excitation-contraction coupling

Proceedings of the National Academy of Sciences - PNAS, 1996-11, Vol.93 (24), p.13961-13966

1996

Volltextzugriff (PDF)

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IP3 receptor function and localization in myotubes: an unexplored Ca2+ signaling pathway in skeletal muscle

Absence of the β Subunit (cchb1) of the Skeletal Muscle Dihydropyridine Receptor Alters Expression of the α1Subunit and Eliminates Excitation-Contraction Coupling

Restoration of excitation-contraction coupling and slow calcium current in dysgenic muscle by dihydropyridine receptor complementary DNA

Dihydropyridine receptors as voltage sensors for a depolarization-evoked, IP3R-mediated, slow calcium signal in skeletal muscle cells

IP3 receptors, IP3 transients, and nucleus-associated Ca2+ signals in cultured skeletal muscle

A lethal mutation in mice eliminates the slow calcium current in skeletal muscle cells

IP3 Receptors and Associated Ca2+ Signals Localize to Satellite Cells and to Components of the Neuromuscular Junction in Skeletal Muscle

Formation of Triads without the Dihydropyridine Receptor α Subunits in Cell Lines from Dysgenic Skeletal Muscle

Muscular dysgenesis : a model system for studying skeletal muscle development

The development of motoneurons in the embryonic spinal cord of the mouse mutant, muscular dysgenesis ( [formula omitted]): Survival, morphology, and biochemical differentiation

IP 3 Receptors and Associated Ca 2+ Signals Localize to Satellite Cells and to Components of the Neuromuscular Junction in Skeletal Muscle

Electrical properties of normal and dysgenib mouse skeletal muscle in culture

Differences in both inositol 1,4,5-trisphosphate mass and inositol 1,4,5-trisphosphate receptors between normal and dystrophic skeletal muscle cell lines

Triad Formation: Organization and Function of the Sarcoplasmic Reticulum Calcium Release Channel and Triadin in Normal and Dysgenic Muscle in Vitro

IP 3 receptors, IP 3 transients, and nucleus-associated Ca 2+ signals in cultured skeletal muscle

Fine structure of prenatal and early postnatal dystrophic mouse muscle

IP3 receptors, IP3 transients, and nucleus-associated Ca2+ signals in cultured skeletal muscle

Muscular dysgenesis: a model system for studying skeletal muscle development

Molecular Origin of the L-Type Ca2+ Current of Skeletal Muscle Myotubes Selectively Deficient in Dihydropyridine Receptor β1a Subunit

Coordinated development of myofibrils, sarcoplasmic reticulum and transverse tubules in normal and dysgenic mouse skeletal muscle, in vivo and in vitro

Molecular Origin of the L-Type Ca 2+ Current of Skeletal Muscle Myotubes Selectively Deficient in Dihydropyridine Receptor β 1a Subunit

Modification of mitochondrial metabolism in fibroblasts from mice with a skeletal muscle mutation (muscular dysgenesis)

Modification of mitochondrial metabolism in fibroblasts from mice with a skeletal muscle mutation (muscular dysgenesis) : Evidence of embryonic communication between myoblasts and fibroblasts

Rescue of excitation-contraction coupling in dysgenic muscle by addition of fibroblasts in vitro

Absence of the beta subunit (cchb1) of the skeletal muscle dihydropyridine receptor alters expression of the alpha 1 subunit and eliminates excitation-contraction coupling

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