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The case for periodontitis in the pathogenesis of rheumatoid arthritis

Nature reviews. Rheumatology, 2017-10, Vol.13 (10), p.606

2017

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Pros and cons of causative association between periodontitis and rheumatoid arthritis

Periodontology 2000, 2022-06, Vol.89 (1), p.83-98

2022

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Manipulation of Neutrophils by Porphyromonas gingivalis in the Development of Periodontitis

Frontiers in cellular and infection microbiology, 2017-05, Vol.7, p.197-197

2017

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Epigenetic regulation in bacterial infections: targeting histone deacetylases

Critical reviews in microbiology, 2018-05, Vol.44 (3), p.336-350

2018

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Antibodies to Porphyromonas gingivalis Indicate Interaction Between Oral Infection, Smoking, and Risk Genes in Rheumatoid Arthritis Etiology

Arthritis & rheumatology (Hoboken, N.J.), 2016-03, Vol.68 (3), p.604-613

2016

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The Type IX Secretion System (T9SS): Highlights and Recent Insights into Its Structure and Function

Frontiers in cellular and infection microbiology, 2017-05, Vol.7, p.215-215

2017

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Host cell-surface proteins as substrates of gingipains, the main proteases of Porphyromonas gingivalis

Biological chemistry, 2018-11, Vol.399 (12), p.1353

2018

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Triggering NETosis via protease-activated receptor (PAR)-2 signaling as a mechanism of hijacking neutrophils function for pathogen benefits

PLoS pathogens, 2019-05, Vol.15 (5), p.e1007773-e1007773

2019

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Mechanisms of vascular damage by systemic dissemination of the oral pathogen Porphyromonas gingivalis

The FEBS journal, 2021-03, Vol.288 (5), p.1479-1495

2021

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Complement Activation as a Helping Hand for Inflammophilic Pathogens and Cancer

Frontiers in immunology, 2019-01, Vol.9, p.3125-3125

2019

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Epigenetic regulation of inflammation in periodontitis: cellular mechanisms and therapeutic potential

Clinical epigenetics, 2020-11, Vol.12 (1), p.186-186, Article 186

2020

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Identification of bacterial biofilm and the Staphylococcus aureus derived protease, staphopain, on the skin surface of patients with atopic dermatitis

Scientific reports, 2017-08, Vol.7 (1), p.8689-12, Article 8689

2017

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Citrullination as a plausible link to periodontitis, rheumatoid arthritis, atherosclerosis and Alzheimer's disease

Journal of oral microbiology, 2018-01, Vol.10 (1), p.1487742-8

2018

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Uncovering the Oral Dysbiotic Microbiota as Masters of Neutrophil Responses in the Pathobiology of Periodontitis

Frontiers in microbiology, 2021-10, Vol.12, p.729717-729717

2021

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Structure and Mechanism of Cysteine Peptidase Gingipain K (Kgp), a Major Virulence Factor of Porphyromonas gingivalis in Periodontitis

The Journal of biological chemistry, 2014-11, Vol.289 (46), p.32291-32302

2014

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Corruption of Innate Immunity by Bacterial Proteases

Journal of innate immunity, 2009-01, Vol.1 (2), p.70-87

2009

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Porphyromonas gingivalis Peptidyl Arginine Deiminase (PPAD) in the Context of the Feed-Forward Loop of Inflammation in Periodontitis

International journal of molecular sciences, 2023-08, Vol.24 (16), p.12922

2023

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Structural determinants of inhibition of Porphyromonas gingivalis gingipain K by KYT-36, a potent, selective, and bioavailable peptidase inhibitor

Scientific reports, 2019-03, Vol.9 (1), p.4935, Article 4935

2019

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A natural anti-periodontitis agent, epimedokoreanin B, inhibits virulence activities of gingipains from Porphyromonas gingivalis

Bioscience, biotechnology, and biochemistry, 2019-07, Vol.83 (7), p.1382-1384

2019

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Proteolytic Activity-Independent Activation of the Immune Response by Gingipains from Porphyromonas gingivalis

mBio, 2022-06, Vol.13 (3), p.e0378721-e0378721

2022

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Peptidylarginine Deiminase of Porphyromonas gingivalis Modulates the Interactions between Candida albicans Biofilm and Human Plasminogen and High-Molecular-Mass Kininogen

International journal of molecular sciences, 2020-04, Vol.21 (7), p.2495

2020

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Strategies for the inhibition of gingipains for the potential treatment of periodontitis and associated systemic diseases

Journal of Oral Microbiology, 2014-01, Vol.6 (1), p.24800-12

2014

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Structural and functional probing of PorZ, an essential bacterial surface component of the type-IX secretion system of human oral-microbiomic Porphyromonas gingivalis

Scientific reports, 2016-11, Vol.6 (1), p.37708-37708, Article 37708

2016

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Antibodies against Porphyromonas gingivalis in serum and saliva and their association with rheumatoid arthritis and periodontitis. Data from two rheumatoid arthritis cohorts in Sweden

Frontiers in immunology, 2023, Vol.14, p.1183194-1183194

2023

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The case for periodontitis in the pathogenesis of rheumatoid arthritis

Pros and cons of causative association between periodontitis and rheumatoid arthritis

Manipulation of Neutrophils by Porphyromonas gingivalis in the Development of Periodontitis

Epigenetic regulation in bacterial infections: targeting histone deacetylases

Antibodies to Porphyromonas gingivalis Indicate Interaction Between Oral Infection, Smoking, and Risk Genes in Rheumatoid Arthritis Etiology

The Type IX Secretion System (T9SS): Highlights and Recent Insights into Its Structure and Function

Host cell-surface proteins as substrates of gingipains, the main proteases of Porphyromonas gingivalis

Triggering NETosis via protease-activated receptor (PAR)-2 signaling as a mechanism of hijacking neutrophils function for pathogen benefits

Mechanisms of vascular damage by systemic dissemination of the oral pathogen Porphyromonas gingivalis

Complement Activation as a Helping Hand for Inflammophilic Pathogens and Cancer

Epigenetic regulation of inflammation in periodontitis: cellular mechanisms and therapeutic potential

Identification of bacterial biofilm and the Staphylococcus aureus derived protease, staphopain, on the skin surface of patients with atopic dermatitis

Citrullination as a plausible link to periodontitis, rheumatoid arthritis, atherosclerosis and Alzheimer's disease

Uncovering the Oral Dysbiotic Microbiota as Masters of Neutrophil Responses in the Pathobiology of Periodontitis

Structure and Mechanism of Cysteine Peptidase Gingipain K (Kgp), a Major Virulence Factor of Porphyromonas gingivalis in Periodontitis

Corruption of Innate Immunity by Bacterial Proteases

Porphyromonas gingivalis Peptidyl Arginine Deiminase (PPAD) in the Context of the Feed-Forward Loop of Inflammation in Periodontitis

Porphyromonas gingivalis facilitates the development and progression of destructive arthritis through its unique bacterial peptidylarginine deiminase (PAD)

Structural determinants of inhibition of Porphyromonas gingivalis gingipain K by KYT-36, a potent, selective, and bioavailable peptidase inhibitor

A natural anti-periodontitis agent, epimedokoreanin B, inhibits virulence activities of gingipains from Porphyromonas gingivalis

Proteolytic Activity-Independent Activation of the Immune Response by Gingipains from Porphyromonas gingivalis

Peptidylarginine Deiminase of Porphyromonas gingivalis Modulates the Interactions between Candida albicans Biofilm and Human Plasminogen and High-Molecular-Mass Kininogen

Strategies for the inhibition of gingipains for the potential treatment of periodontitis and associated systemic diseases

Structural and functional probing of PorZ, an essential bacterial surface component of the type-IX secretion system of human oral-microbiomic Porphyromonas gingivalis

Antibodies against Porphyromonas gingivalis in serum and saliva and their association with rheumatoid arthritis and periodontitis. Data from two rheumatoid arthritis cohorts in Sweden

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