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Selective Protein Covalent Binding and Target Organ Toxicity

Toxicology and applied pharmacology, 1997-03, Vol.143 (1), p.1-12

1997

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A Protective Role for Cyclooxygenase-2 in Drug-Induced Liver Injury in Mice

Chemical research in toxicology, 2001-12, Vol.14 (12), p.1620-1628

2001

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Powerful Prose : How Textual Features Impact Readers (Edition 1)

1, 2021

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Protective Role of Kupffer Cells in Acetaminophen-Induced Hepatic Injury in Mice

Chemical research in toxicology, 2002-12, Vol.15 (12), p.1504-1513

2002

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Hepatotoxicity after desflurane anesthesia

Anesthesiology (Philadelphia), 1995-11, Vol.83 (5), p.1125-1129

1995

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Role of interleukin-6 in hepatic heat shock protein expression and protection against acetaminophen-induced liver disease

Biochemical and biophysical research communications, 2003-04, Vol.304 (1), p.207-212

2003

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Epidemic of liver disease caused by hydrochlorofluorocarbons used as ozone-sparing substitutes of chlorofluorocarbons

The Lancet (British edition), 1997-08, Vol.350 (9077), p.556-559

1997

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Hepatoprotective Role of Endogenous Interleukin-13 in a Murine Model of Acetaminophen-Induced Liver Disease

Chemical research in toxicology, 2007-05, Vol.20 (5), p.734-744

2007

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Metabolic basis for a drug hypersensitivity: antibodies in sera from patients with halothane hepatitis recognize liver neoantigens that contain the trifluoroacetyl group derived from halothane

The Journal of pharmacology and experimental therapeutics, 1988-06, Vol.245 (3), p.1103

1988

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Immunohistochemical Detection of Protein Adducts of 2,4-Dinitrochlorobenzene in Antigen Presenting Cells and Lymphocytes after Oral Administration to Mice: Lack of a Role of Kupffer Cells in Oral Tolerance

Chemical research in toxicology, 2001-09, Vol.14 (9), p.1209-1217

2001

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Tolerogenic role of Kupffer cells in immune-mediated adverse drug reactions

Toxicology (Amsterdam), 2005-04, Vol.209 (2), p.109-112

2005

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Human Anti-Endoplasmic Reticulum Antibodies in Sera of Patients with Halothane-Induced Hepatitis are Directed against a Trifluoroacetylated Carboxylesterase

Proceedings of the National Academy of Sciences - PNAS, 1989-01, Vol.86 (1), p.322-326

1989

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Metabolic Activation of Diclofenac by Human Cytochrome P450 3A4: Role of 5-Hydroxydiclofenac

Chemical research in toxicology, 1999-02, Vol.12 (2), p.214-222

1999

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Proteomic Identification of Potential Susceptibility Factors in Drug-Induced Liver Disease

Chemical research in toxicology, 2005-06, Vol.18 (6), p.924-933

2005

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The immunologic and metabolic basis of drug hypersensitivities

Annual review of pharmacology and toxicology, 1988, Vol.28 (1), p.367-387

1988

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Immunochemical detection of liver protein adducts of the nonsteroidal antiinflammatory drug diclofenac

Chemical research in toxicology, 1993-03, Vol.6 (2), p.147-150

1993

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An immunochemical approach of identifying and characterizing protein targets of toxic reactive metabolites

Chemical research in toxicology, 1993-11, Vol.6 (6), p.786-793

Pohl, Lance R

1993

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Expression Profiling of Acetaminophen Liver Toxicity in Mice Using Microarray Technology

Biochemical and biophysical research communications, 2001-03, Vol.282 (1), p.321-328

2001

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Genomic Identification of Potential Risk Factors during Acetaminophen-Induced Liver Disease in Susceptible and Resistant Strains of Mice

Chemical research in toxicology, 2006-02, Vol.19 (2), p.223-233

2006

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Factors affecting the expression of trifluoroacetylated liver microsomal protein neoantigens in rats treated with halothane

Drug metabolism and disposition, 1990-09, Vol.18 (5), p.788

1990

Volltextzugriff (PDF)

Tissue Acylation by the Chlorofluorocarbon Substitute 2,2-Dichloro-1,1,1- Trifluoroethane

Proceedings of the National Academy of Sciences - PNAS, 1991-02, Vol.88 (4), p.1407-1410

1991

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Detection of cysteine conjugate metabolite adduct formation with specific mitochondrial proteins using antibodies raised against halothane metabolite adducts

The Journal of biological chemistry, 1991-10, Vol.266 (28), p.18415-18418

1991

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Lymphocyte Loss and Immunosuppression Following Acetaminophen-Induced Hepatotoxicity in Mice as a Potential Mechanism of Tolerance

Chemical research in toxicology, 2007-01, Vol.20 (1), p.20-26

2007

Volltextzugriff (PDF)

CONSULTATION TO PEDIATRICIANS

Journal of the American Academy of Child and Adolescent Psychiatry, 1998-11, Vol.37 (11), p.1129-1130

Pohl, Richard L.

1998

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Halothane hepatitis patients have serum antibodies that react with protein disulfide isomerase

Hepatology (Baltimore, Md.), 1993-10, Vol.18 (4), p.858-863

1993

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Selective Protein Covalent Binding and Target Organ Toxicity

A Protective Role for Cyclooxygenase-2 in Drug-Induced Liver Injury in Mice

Powerful Prose : How Textual Features Impact Readers (Edition 1)

Protective Role of Kupffer Cells in Acetaminophen-Induced Hepatic Injury in Mice

Hepatotoxicity after desflurane anesthesia

Role of interleukin-6 in hepatic heat shock protein expression and protection against acetaminophen-induced liver disease

Epidemic of liver disease caused by hydrochlorofluorocarbons used as ozone-sparing substitutes of chlorofluorocarbons

Hepatoprotective Role of Endogenous Interleukin-13 in a Murine Model of Acetaminophen-Induced Liver Disease

Metabolic basis for a drug hypersensitivity: antibodies in sera from patients with halothane hepatitis recognize liver neoantigens that contain the trifluoroacetyl group derived from halothane

Immunohistochemical Detection of Protein Adducts of 2,4-Dinitrochlorobenzene in Antigen Presenting Cells and Lymphocytes after Oral Administration to Mice: Lack of a Role of Kupffer Cells in Oral Tolerance

Tolerogenic role of Kupffer cells in immune-mediated adverse drug reactions

Human Anti-Endoplasmic Reticulum Antibodies in Sera of Patients with Halothane-Induced Hepatitis are Directed against a Trifluoroacetylated Carboxylesterase

Metabolic Activation of Diclofenac by Human Cytochrome P450 3A4: Role of 5-Hydroxydiclofenac

Proteomic Identification of Potential Susceptibility Factors in Drug-Induced Liver Disease

The immunologic and metabolic basis of drug hypersensitivities

Immunochemical detection of liver protein adducts of the nonsteroidal antiinflammatory drug diclofenac

An immunochemical approach of identifying and characterizing protein targets of toxic reactive metabolites

Expression Profiling of Acetaminophen Liver Toxicity in Mice Using Microarray Technology

Genomic Identification of Potential Risk Factors during Acetaminophen-Induced Liver Disease in Susceptible and Resistant Strains of Mice

Factors affecting the expression of trifluoroacetylated liver microsomal protein neoantigens in rats treated with halothane

Tissue Acylation by the Chlorofluorocarbon Substitute 2,2-Dichloro-1,1,1- Trifluoroethane

Detection of cysteine conjugate metabolite adduct formation with specific mitochondrial proteins using antibodies raised against halothane metabolite adducts

Lymphocyte Loss and Immunosuppression Following Acetaminophen-Induced Hepatotoxicity in Mice as a Potential Mechanism of Tolerance

CONSULTATION TO PEDIATRICIANS

Halothane hepatitis patients have serum antibodies that react with protein disulfide isomerase

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