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Endotoxin Uptake by S1 Proximal Tubular Segment Causes Oxidative Stress in the Downstream S2 Segment

Journal of the American Society of Nephrology, 2011-08, Vol.22 (8), p.1505-1516

2011

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The macrophage mediates the renoprotective effects of endotoxin preconditioning

Journal of the American Society of Nephrology, 2015-06, Vol.26 (6), p.1347-1362

2015

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p53 Is Renoprotective after Ischemic Kidney Injury by Reducing Inflammation

Journal of the American Society of Nephrology, 2013-01, Vol.24 (1), p.113-124

2013

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Endotoxin Preconditioning Reprograms S1 Tubules and Macrophages to Protect the Kidney

Journal of the American Society of Nephrology, 2018-01, Vol.29 (1), p.104-117

2018

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Novel application of complementary imaging techniques to examine in vivo glucose metabolism in the kidney

American journal of physiology. Renal physiology, 2016-04, Vol.310 (8), p.F717-F725

2016

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Sepsis induces changes in the expression and distribution of Toll-like receptor 4 in the rat kidney

American journal of physiology. Renal physiology, 2006-05, Vol.290 (5), p.F1034-F1043

2006

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Inhibition of Toll-Like Receptor 4 Signaling Mitigates Microvascular Loss but Not Fibrosis in a Model of Ischemic Acute Kidney Injury

International journal of molecular sciences, 2016-05, Vol.17 (5), p.647-647

2016

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Minocycline reduces renal microvascular leakage in a rat model of ischemic renal injury

American journal of physiology. Renal physiology, 2005-01, Vol.288 (1), p.F91-F97

2005

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Bacterial sepsis triggers an antiviral response that causes translation shutdown

The Journal of clinical investigation, 2019-01, Vol.129 (1), p.296-309

2019

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Guanine nucleosides and Jurkat cell death: roles of ATP depletion and accumulation of deoxyribonucleotides

American Journal of Physiology: Cell Physiology, 2001-12, Vol.281 (6), p.C1776-C1784

2001

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P53 mediates the apoptotic response to GTP depletion after renal ischemia-reperfusion: Protective role of a p53 inhibitor

Journal of the American Society of Nephrology, 2003, Vol.14 (1), p.128-138

2003

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The p53 inhibitor pifithrin-α can stimulate fibrosis in a rat model of ischemic acute kidney injury

American journal of physiology. Renal physiology, 2012-01, Vol.302 (2), p.F284-F291

2012

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Sepsis induces an increase in thick ascending limb Cox-2 that is TLR4 dependent

American journal of physiology. Renal physiology, 2007-10, Vol.293 (4), p.F1187-F1196

2007

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Guanosine supplementation reduces apoptosis and protects renal function in the setting of ischemic injury

The Journal of clinical investigation, 2001-11, Vol.108 (9), p.1291-1298

2001

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p53 regulates renal expression of HIF-1α and pVHL under physiological conditions and after ischemia-reperfusion injury

American Journal of Physiology - Renal Physiology, 2008-12, Vol.295 (6), p.F1666-F1677

2008

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The p53 inhibitor pifithrin-α can stimulate fibrosis in a rat model of ischemic acute kidney injury

American journal of physiology. Heart and circulatory physiology, 2012, Vol.71 (1)

2012

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The p53 inhibitor pifithrin-[alpha] can stimulate fibrosis in a rat model of ischemic acute kidney injury

American journal of physiology. Renal physiology, 2012-01, Vol.302 (2), p.F284

2012

Volltextzugriff (PDF)

p53 regulates renal expression of HIF-1{alpha} and pVHL under physiological conditions and after ischemia-reperfusion injury

American journal of physiology. Renal physiology, 2008-12, Vol.295 (6), p.F1666-F1677

2008

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Sepsis induces an increase in thick ascending limb Cox-2 that is TLR4 dependent

American journal of physiology. Renal physiology, 2007, Vol.62 (4)

2007

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Sepsis induces changes in the expression and distribution of Toll-like receptor 4 in the rat kidney

American journal of physiology. Renal physiology, 2006-05, Vol.59 (5), p.F1034

2006

Volltextzugriff (PDF)

P53 regulates renal expression of HIF-1α and pVHL under physiological conditions and after ischemia-reperfusion injury

American journal of physiology. Renal physiology, 2008, Vol.64 (6)

2008

Volltextzugriff (PDF)

Minocycline reduces renal microvascular leakage in a rat model of ischemic renal injury

American journal of physiology. Renal physiology, 2005, Vol.57 (1), p.F91-F97

2005

Volltextzugriff (PDF)

Guanine nucleosides and Jurkat cell death: roles of ATP depletion and accumulation of deoxyribonucleotides

American Journal of Physiology: Cell Physiology, 2001-12, Vol.50 (6), p.C1776-C1784

2001

Volltextzugriff (PDF)

The role of cytokines in polymyositis

Arthritis and rheumatism, 1994-06, Vol.37 (6), p.907-914

1994

Volltextzugriff (PDF)

Proliferative response of peripheral blood mononuclear cells to autologous and allogeneic muscle in patients with polymyositis/dermatomyositis

Arthritis and rheumatism, 1989-04, Vol.32 (4), p.446-453

1989

Volltextzugriff (PDF)

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Endotoxin Uptake by S1 Proximal Tubular Segment Causes Oxidative Stress in the Downstream S2 Segment

The macrophage mediates the renoprotective effects of endotoxin preconditioning

p53 Is Renoprotective after Ischemic Kidney Injury by Reducing Inflammation

Endotoxin Preconditioning Reprograms S1 Tubules and Macrophages to Protect the Kidney

Novel application of complementary imaging techniques to examine in vivo glucose metabolism in the kidney

Sepsis induces changes in the expression and distribution of Toll-like receptor 4 in the rat kidney

Inhibition of Toll-Like Receptor 4 Signaling Mitigates Microvascular Loss but Not Fibrosis in a Model of Ischemic Acute Kidney Injury

Minocycline reduces renal microvascular leakage in a rat model of ischemic renal injury

Bacterial sepsis triggers an antiviral response that causes translation shutdown

Guanine nucleosides and Jurkat cell death: roles of ATP depletion and accumulation of deoxyribonucleotides

P53 mediates the apoptotic response to GTP depletion after renal ischemia-reperfusion: Protective role of a p53 inhibitor

The p53 inhibitor pifithrin-α can stimulate fibrosis in a rat model of ischemic acute kidney injury

Sepsis induces an increase in thick ascending limb Cox-2 that is TLR4 dependent

Guanosine supplementation reduces apoptosis and protects renal function in the setting of ischemic injury

p53 regulates renal expression of HIF-1α and pVHL under physiological conditions and after ischemia-reperfusion injury

The p53 inhibitor pifithrin-α can stimulate fibrosis in a rat model of ischemic acute kidney injury

The p53 inhibitor pifithrin-[alpha] can stimulate fibrosis in a rat model of ischemic acute kidney injury

p53 regulates renal expression of HIF-1{alpha} and pVHL under physiological conditions and after ischemia-reperfusion injury

Sepsis induces an increase in thick ascending limb Cox-2 that is TLR4 dependent

Sepsis induces changes in the expression and distribution of Toll-like receptor 4 in the rat kidney

P53 regulates renal expression of HIF-1α and pVHL under physiological conditions and after ischemia-reperfusion injury

Minocycline reduces renal microvascular leakage in a rat model of ischemic renal injury

Guanine nucleosides and Jurkat cell death: roles of ATP depletion and accumulation of deoxyribonucleotides

The role of cytokines in polymyositis

Proliferative response of peripheral blood mononuclear cells to autologous and allogeneic muscle in patients with polymyositis/dermatomyositis

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