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An effective QCD Lagrangian in the presence of an axial chemical potential

The European physical journal. C, Particles and fields, 2013, Vol.73 (1), p.1-10, Article 2294

2013

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Chemical potentials and parity breaking: the Nambu–Jona-Lasinio model

The European physical journal. C, Particles and fields, 2014-02, Vol.74 (2), p.2776-11, Article 2776

2014

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Dilepton excess from local parity breaking in baryon matter

Physics letters. B, 2012-03, Vol.710 (1), p.230-235

2012

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Dilepton excess from local parity breaking in baryon matter

Physics letters. B, 2012, Vol.710 (1), p.230-235

2012

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Searching for P- and CP-odd effects in heavy ion collisions

AIP Conference Proceedings, 2016, Vol.1701 (1)

2016

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Analysis of dilepton angular distributions in a parity breaking medium

Physical review. D, Particles, fields, gravitation, and cosmology, 2014-08, Vol.90 (3), Article 034024

2014

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Abnormal enhancement of dilepton yield in central heavy-ion collisions from local parity breaking

Theoretical and mathematical physics, 2012-01, Vol.170 (1), p.17-25

2012

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Abnormal dilepton yield from parity breaking in dense nuclear matter

AIP conference proceedings, 2011, Vol.1343 (1)

2011

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Аномальное усиление выхода дилептонов в центральных столкновениях тяжелых ионов вследствие локального нарушения четности

Teoretičeskaja i matematičeskaja fizika, 2012, Vol.170 (1), p.22-33

2012

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BRG1/SMARCA4 is essential for neuroblastoma cell viability through modulation of cell death and survival pathways

Oncogene, 2016-09, Vol.35 (39), p.5179-5190

2016

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Fas apoptosis inhibitory molecules: more than death‐receptor antagonists in the nervous system

Journal of neurochemistry, 2016-10, Vol.139 (1), p.11-21

2016

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FAIM-L regulation of XIAP degradation modulates Synaptic Long-Term Depression and Axon Degeneration

Scientific reports, 2016-10, Vol.6 (1), p.35775-35775, Article 35775

2016

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Identification and characterization of new isoforms of human fas apoptotic inhibitory molecule (FAIM)

PloS one, 2017-10, Vol.12 (10), p.e0185327-e0185327

2017

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TNFα induces survival through the FLIP-L-dependent activation of the MAPK/ERK pathway

Cell death & disease, 2013-02, Vol.4 (2), p.e493-e493

2013

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MYCN repression of Lifeguard/FAIM2 enhances neuroblastoma aggressiveness

Cell death & disease, 2014-09, Vol.5 (9), p.e1401-e1401

2014

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Lifeguard Inhibits Fas Ligand-mediated Endoplasmic Reticulum-Calcium Release Mandatory for Apoptosis in Type II Apoptotic Cells

The Journal of biological chemistry, 2016-01, Vol.291 (3), p.1221-1234

2016

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SIVA-1 regulates apoptosis and synaptic function by modulating XIAP interaction with the death receptor antagonist FAIM-L

Cell death & disease, 2020-02, Vol.11 (2), p.82-82, Article 82

2020

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FAIM-L is an IAP-binding protein that inhibits XIAP ubiquitinylation and protects from Fas-induced apoptosis

The Journal of neuroscience, 2013-12, Vol.33 (49), p.19262-19275

2013

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TNFα sensitizes neuroblastoma cells to FasL-, cisplatin- and etoposide-induced cell death by NF-κB-mediated expression of Fas

Molecular cancer, 2015-03, Vol.14 (1), p.62, Article 62

2015

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Synthesis and optical characterisation of triphenylamine-based hole extractor materials for CdSe quantum dotsElectronic supplementary information (ESI) available: Powder XRD of CdSe QDs. Cyclic voltammetry at different scan rates and differential pulse voltammetry of neat TPA derivatives. Single-photon counting fitting. Transient absorption spectrum. Reorganisation energies table. See DOI: 10.1039/c3cp50980j

2013-05

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Synthesis and optical characterisation of triphenylamine-based hole extractor materials for CdSe quantum dots

Physical chemistry chemical physics : PCCP, 2013-05, Vol.15 (20), p.7679-7684

2013

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Synthesis and optical characterisation of triphenylamine-based hole extractor materials for CdSe quantum dotst

Physical chemistry chemical physics : PCCP, 2013, Vol.15 (20), p.7679-7684

2013

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FAIM-L regulation of XIAP degradation modulates Synaptic Long-Term Depression and Axon Degeneration

Scientific reports, 2016-10

2016

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TNF[alpha] induces survival through the FLIP-L-dependent activation of the MAPK/ERK pathway

Cell death & disease, 2013-02, Vol.4, p.e493

2013

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An effective QCD Lagrangian in the presence of an axial chemical potential

Chemical potentials and parity breaking: the Nambu–Jona-Lasinio model

Dilepton excess from local parity breaking in baryon matter

Dilepton excess from local parity breaking in baryon matter

Searching for P- and CP-odd effects in heavy ion collisions

Analysis of dilepton angular distributions in a parity breaking medium

Abnormal enhancement of dilepton yield in central heavy-ion collisions from local parity breaking

Abnormal dilepton yield from parity breaking in dense nuclear matter

Аномальное усиление выхода дилептонов в центральных столкновениях тяжелых ионов вследствие локального нарушения четности

BRG1/SMARCA4 is essential for neuroblastoma cell viability through modulation of cell death and survival pathways

Fas apoptosis inhibitory molecules: more than death‐receptor antagonists in the nervous system

FAIM-L regulation of XIAP degradation modulates Synaptic Long-Term Depression and Axon Degeneration

Identification and characterization of new isoforms of human fas apoptotic inhibitory molecule (FAIM)

TNFα induces survival through the FLIP-L-dependent activation of the MAPK/ERK pathway

MYCN repression of Lifeguard/FAIM2 enhances neuroblastoma aggressiveness

Lifeguard Inhibits Fas Ligand-mediated Endoplasmic Reticulum-Calcium Release Mandatory for Apoptosis in Type II Apoptotic Cells

SIVA-1 regulates apoptosis and synaptic function by modulating XIAP interaction with the death receptor antagonist FAIM-L

FAIM-L is an IAP-binding protein that inhibits XIAP ubiquitinylation and protects from Fas-induced apoptosis

TNFα sensitizes neuroblastoma cells to FasL-, cisplatin- and etoposide-induced cell death by NF-κB-mediated expression of Fas

Synthesis and optical characterisation of triphenylamine-based hole extractor materials for CdSe quantum dots

Synthesis and optical characterisation of triphenylamine-based hole extractor materials for CdSe quantum dotst

FAIM-L regulation of XIAP degradation modulates Synaptic Long-Term Depression and Axon Degeneration

TNF[alpha] induces survival through the FLIP-L-dependent activation of the MAPK/ERK pathway

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