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Protective effect of quercetin treatment on HFD-induced intestinal dysbiosis and barrier dysfunction in an in vivo model of non-alcoholic fatty liver disease
Journal of hepatology, 2017, Vol.66 (1), p.S169-S169
2017
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P0973 : Quercetin ameliorates MCD-induced non-alcoholic fatty liver disease in mice by modulating inflammatory, oxidative/nitrosative stress and lipid metabolism-related gene deregulation via the PI3K/AKT pathway
Journal of hepatology, 2015-04, Vol.62, p.S711-S711
2015
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868 LIVER X RECEPTOR a-MEDIATED LIPOGENESIS INDUCED BY HEPATITIS C VIRUS NS5A AND CORE PROTEINS REGULATES VIRAL REPLICATION IN AN IN VITRO MODEL
Journal of hepatology, 2012-04, Vol.56, p.S338-S338
2012
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804 EFFECT OF HCV NS5A AND CORE PROTEINS AND VIRAL REPLICATION ON LXRa-MEDIATED LIPOGENIC GENES INDUCTION IN AN IN VITRO MODEL
Journal of hepatology, 2011-03, Vol.54, p.S323-S323
2011
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786 ROLE OF LXRALPHA AND ITS LIPOGENIC TARGET GENES IN THE PATHOGENESIS OF HCV-RELATED STEATOSIS IN AN IN VITRO MODEL
Journal of hepatology, 2010, Vol.52, p.S305-S306
2010
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THU-386 - Protective effect of quercetin treatment on HFD-induced intestinal dysbiosis and barrier dysfunction in an in vivo model of non-alcoholic fatty liver disease
Journal of hepatology, 2017, Vol.66 (1), p.S169-S169
2017
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Protective effect of quercetin on high-fat diet-induced non-alcoholic fatty liver disease in mice is mediated by modulating intestinal microbiota imbalance and related gut-liver axis activation
Free radical biology & medicine, 2017-01, Vol.102, p.188-202
2017
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The TRAPP complex mediates secretion arrest induced by stress granule assembly
The EMBO journal, 2019-10, Vol.38 (19), p.e101704-n/a
2019
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Lysosomotropic Drugs: Pharmacological Tools to Study Lysosomal Function
Current drug metabolism, 2017-12, Vol.18 (12), p.1147
2017
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gene2drug: a computational tool for pathway-based rational drug repositioning
Bioinformatics, 2018-05, Vol.34 (9), p.1498-1505
2018
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Quercetin ameliorates dysregulation of lipid metabolism genes via the PI3K/AKT pathway in a diet‐induced mouse model of nonalcoholic fatty liver disease
Molecular nutrition & food research, 2015-05, Vol.59 (5), p.879-893
2015
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TFEB Modulates p21/WAF1/CIP1 during the DNA Damage Response
Cells (Basel, Switzerland), 2020-05, Vol.9 (5), p.1186
2020
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Flavonoids and Related Compounds in Non-Alcoholic Fatty Liver Disease Therapy
Current medicinal chemistry, 2015-01, Vol.22 (25), p.2991
2015
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Modulation of PI3K-LXRα-dependent lipogenesis mediated by oxidative/nitrosative stress contributes to inhibition of HCV replication by quercetin
Laboratory investigation, 2014-03, Vol.94 (3), p.262-274
2014
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The human liver fatty acid binding protein (FABP1) gene is activated by FOXA1 and PPARα; and repressed by C/EBPα: Implications in FABP1 down-regulation in nonalcoholic fatty liver disease
Biochimica et biophysica acta, 2013-04, Vol.1831 (4), p.803-818
2013
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Liver X receptor α-mediated regulation of lipogenesis by core and NS5A proteins contributes to HCV-induced liver steatosis and HCV replication
Laboratory investigation, 2012-08, Vol.92 (8), p.1191-1202
2012
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Repression of the nuclear receptor small heterodimer partner by steatotic drugs and in advanced nonalcoholic fatty liver disease
Molecular pharmacology, 2015-04, Vol.87 (4), p.582-594
2015
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Comparing structural and transcriptional drug networks reveals signatures of drug activity and toxicity in transcriptional responses
NPJ systems biology and applications, 2017-01, Vol.3 (1), p.23-12, Article 23
2017
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Modulation of PI3K-LXR[alpha]-dependent lipogenesis mediated by oxidative/nitrosative stress contributes to inhibition of HCV replication by quercetin
Laboratory investigation, 2014-03, Vol.94 (3), p.262
2014
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Liver X receptor [alpha]-mediated regulation of lipogenesis by core and NS5A proteins contributes to HCV-induced liver steatosis and HCV replication
Laboratory investigation, 2012-08, Vol.92 (8), p.1191
2012
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