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Synaptic Deficits at Neuromuscular Junctions in Two Mouse Models of Charcot-Marie-Tooth Type 2d

The Journal of neuroscience, 2016-03, Vol.36 (11), p.3254-3267

2016

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Sodium channel slow inactivation as a therapeutic target for myotonia congenita

Annals of neurology, 2015-02, Vol.77 (2), p.320-332

2015

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Crucial Role of Sodium Channel Fast Inactivation in Muscle Fibre Inexcitability in a Rat Model of Critical Illness Myopathy

The Journal of physiology, 2003-03, Vol.547 (2), p.555-566

2003

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Reversible Recruitment of a Homeostatic Reserve Pool of Synaptic Vesicles Underlies Rapid Homeostatic Plasticity of Quantal Content

The Journal of neuroscience, 2016-01, Vol.36 (3), p.828-836

2016

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Gap Junctional Coupling and Patterns of Connexin Expression among Neonatal Rat Lumbar Spinal Motor Neurons

The Journal of neuroscience, 1999-12, Vol.19 (24), p.10813-10828

1999

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Altered terminal Schwann cell morphology precedes denervation in SOD1 mice

Experimental neurology, 2016-01, Vol.275 (1), p.172-181

2016

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Nerve terminal degeneration is independent of muscle fiber genotype in SOD1 mice

PloS one, 2010-03, Vol.5 (3), p.e9802-e9802

2010

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Sodium channel inactivation in an animal model of acute quadriplegic myopathy

Annals of neurology, 2001-07, Vol.50 (1), p.26-33

2001

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Permanent central synaptic disconnection of proprioceptors after nerve injury and regeneration. II. Loss of functional connectivity with motoneurons

Journal of neurophysiology, 2011-11, Vol.106 (5), p.2471-2485

2011

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Motor terminal degeneration unaffected by activity changes in SOD1G93A mice; a possible role for glycolysis

Neurobiology of disease, 2012-10, Vol.48 (1), p.132-140

2012

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Abnormal response of distal Schwann cells to denervation in a mouse model of motor neuron disease

Experimental neurology, 2016-04, Vol.278, p.116-126

2016

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Ca2+ Dependence of the Binomial Parameters p and n at the Mouse Neuromuscular Junction

Journal of neurophysiology, 2010-02, Vol.103 (2), p.659-666

2010

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Rat motoneuron properties recover following reinnervation in the absence of muscle activity and evoked acetylcholine release

The Journal of physiology, 2007-11, Vol.585 (1), p.47-56

2007

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Recovery of proprioceptive feedback from nerve crush

The Journal of physiology, 2011-10, Vol.589 (20), p.4935-4947

2011

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Motor terminal degeneration unaffected by activity changes in SOD1(G93A) mice; a possible role for glycolysis

Neurobiology of disease, 2012-10, Vol.48 (1), p.132-140

2012

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Reduced Endplate Currents Underlie Motor Unit Dysfunction in Canine Motor Neuron Disease

Journal of neurophysiology, 2002-12, Vol.88 (6), p.3293-3304

2002

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Role of Ca(2+) in injury-induced changes in sodium current in rat skeletal muscle

American Journal of Physiology: Cell Physiology, 2009-08, Vol.297 (2), p.C352-C359

2009

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Role of Ca2+ in injury-induced changes in sodium current in rat skeletal muscle

American Journal of Physiology: Cell Physiology, 2009-08, Vol.297 (2), p.C352-C359

2009

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Central Suppression of Regenerated Proprioceptive Afferents

The Journal of neuroscience, 2005-05, Vol.25 (19), p.4733-4742

2005

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Activity-dependent presynaptic regulation of quantal size at the mammalian neuromuscular junction in vivo

The Journal of neuroscience, 2005-01, Vol.25 (2), p.343-351

2005

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Nerve Terminal Degeneration Is Independent of Muscle Fiber Genotype in SOD1G93A Mice

PloS one, 2010-03, Vol.5 (3), p.e9802

2010

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Resting potential-dependent regulation of the voltage sensitivity of sodium channel gating in rat skeletal muscle in vivo

The Journal of general physiology, 2005-08, Vol.126 (2), p.161-172

2005

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Decreased Synaptic Activity Shifts the Calcium Dependence of Release at the Mammalian Neuromuscular Junction In Vivo

The Journal of neuroscience, 2004-11, Vol.24 (47), p.10687-10692

2004

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Enhanced Transmission at a Spinal Synapse Triggered In Vivo by an Injury Signal Independent of Altered Synaptic Activity

The Journal of neuroscience, 2007-11, Vol.27 (47), p.12851-12859

2007

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Nerve Injury Induces Gap Junctional Coupling among Axotomized Adult Motor Neurons

The Journal of neuroscience, 2000-01, Vol.20 (2), p.674-684

2000

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Synaptic Deficits at Neuromuscular Junctions in Two Mouse Models of Charcot-Marie-Tooth Type 2d

Sodium channel slow inactivation as a therapeutic target for myotonia congenita

Crucial Role of Sodium Channel Fast Inactivation in Muscle Fibre Inexcitability in a Rat Model of Critical Illness Myopathy

Reversible Recruitment of a Homeostatic Reserve Pool of Synaptic Vesicles Underlies Rapid Homeostatic Plasticity of Quantal Content

Gap Junctional Coupling and Patterns of Connexin Expression among Neonatal Rat Lumbar Spinal Motor Neurons

Altered terminal Schwann cell morphology precedes denervation in SOD1 mice

Nerve terminal degeneration is independent of muscle fiber genotype in SOD1 mice

Sodium channel inactivation in an animal model of acute quadriplegic myopathy

Permanent central synaptic disconnection of proprioceptors after nerve injury and regeneration. II. Loss of functional connectivity with motoneurons

Motor terminal degeneration unaffected by activity changes in SOD1G93A mice; a possible role for glycolysis

Abnormal response of distal Schwann cells to denervation in a mouse model of motor neuron disease

Ca2+ Dependence of the Binomial Parameters p and n at the Mouse Neuromuscular Junction

Rat motoneuron properties recover following reinnervation in the absence of muscle activity and evoked acetylcholine release

Recovery of proprioceptive feedback from nerve crush

Motor terminal degeneration unaffected by activity changes in SOD1(G93A) mice; a possible role for glycolysis

Reduced Endplate Currents Underlie Motor Unit Dysfunction in Canine Motor Neuron Disease

Role of Ca(2+) in injury-induced changes in sodium current in rat skeletal muscle

Role of Ca2+ in injury-induced changes in sodium current in rat skeletal muscle

Central Suppression of Regenerated Proprioceptive Afferents

Activity-dependent presynaptic regulation of quantal size at the mammalian neuromuscular junction in vivo

Nerve Terminal Degeneration Is Independent of Muscle Fiber Genotype in SOD1G93A Mice

Resting potential-dependent regulation of the voltage sensitivity of sodium channel gating in rat skeletal muscle in vivo

Decreased Synaptic Activity Shifts the Calcium Dependence of Release at the Mammalian Neuromuscular Junction In Vivo

Enhanced Transmission at a Spinal Synapse Triggered In Vivo by an Injury Signal Independent of Altered Synaptic Activity

Nerve Injury Induces Gap Junctional Coupling among Axotomized Adult Motor Neurons

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