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G.P.1.10 DHPR α1 S subunit controls skeletal muscle mass and morphogenesis

Neuromuscular disorders : NMD, 2009-09, Vol.19 (8), p.559-559

2009

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P.4.10 Exon skipping as a therapeutic strategy applied to a RyR1 mutation causing severe core myopathy

Neuromuscular disorders : NMD, 2013-10, Vol.23 (9), p.762-762

2013

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Thiol reagents increase the affinity of the inositol 1,4,5-trisphosphate receptor

The Journal of biological chemistry, 1993-08, Vol.268 (22), p.16488-16494

1993

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DMD TREATMENT: ANIMAL MODELS

Neuromuscular disorders : NMD, 2018-10, Vol.28, p.S94

2018

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DMD TREATMENT: ANIMAL MODELS: P.204R-DMDdel52, a novel preclinical rat model of Duchenne muscular dystrophy

Neuromuscular disorders : NMD, 2018-10, Vol.28, p.S94-S94

2018

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P.288Multi-exon skipping by CRISPR/Cas9-mediated genome editing in GRMD dog

Neuromuscular disorders : NMD, 2019-10, Vol.29, p.S151-S151

2019

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DMD TREATMENT

Neuromuscular disorders : NMD, 2019-10, Vol.29, p.S164-S165

2019

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DMD TREATMENT: P.334PPMO pre-treatment is beneficial for AAV-based gene therapy in Duchenne muscular dystrophy

Neuromuscular disorders : NMD, 2019-10, Vol.29, p.S164-S165

2019

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P.447 - From voltage sensing to gene expression in the control of muscle mass homeostasis

Neuromuscular disorders : NMD, 2017-10, Vol.27, p.S230-S230

2017

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From voltage sensing to gene expression in the control of muscle mass homeostasis

Neuromuscular disorders : NMD, 2017-10, Vol.27, p.S230

2017

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P.20.11 Dystrophin rescue needed to recover a correct location of nNOS and the return to a normalized RyR1 status in treated GRMD dogs

Neuromuscular disorders : NMD, 2013-10, Vol.23 (9), p.841-842

2013

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G.P.93

Neuromuscular disorders : NMD, 2014-10, Vol.24 (9), p.822-823

2014

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Blockade of ActRIIB Signaling Triggers Muscle Fatigability and Metabolic Myopathy

Molecular therapy, 2014-08, Vol.22 (8), p.1423-1433

2014

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P.237 - MicroRNAs involved in nNOS regulation in dystrophic context

Neuromuscular disorders : NMD, 2017-10, Vol.27, p.S166-S167

2017

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MicroRNAs involved in nNOS regulation in dystrophic context

Neuromuscular disorders : NMD, 2017-10, Vol.27, p.S166-S167

2017

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G.P.75 Variable phenotype of del45–55 Becker patients correlated to nNOSμ mislocalization and RYR1 hypernitrosylation

Neuromuscular disorders : NMD, 2012-10, Vol.22 (9), p.834-834

2012

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Pharmacological characteristics and species-related variations of β3-adrenergic receptors

Fundamental & clinical pharmacology, 1995-05, Vol.9 (3), p.211-218

1995

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P1.37 DHPR α1S subunit controls skeletal muscle mass and morphogenesis

Neuromuscular disorders : NMD, 2010-10, Vol.20 (9), p.611-612

2010

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Actin scaffolding by clathrin heavy chain is required for skeletal muscle sarcomere organization

The Journal of general physiology, 2014-06, Vol.143 (6), p.1436

2014

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FP.44 Exploring the role of MuscleBlind-Like proteins in the regulation of CaVB1 isoform expression in adult skeletal muscle

Neuromuscular disorders : NMD, 2022-10, Vol.32, p.S129

2022

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Human immortalized brown adipocytes express functional beta3-adrenoceptor coupled to lipolysis

Journal of cell science, 1997-04, Vol.110 ( Pt 7), p.801-807

1997

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Human adipose cells express CD4, CXCR4, and CCR5 receptors: a new target cell type for the immunodeficiency virus‐1?

The FASEB journal, 2002-08, Vol.16 (10), p.1254-1256

2002

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New series of N-substituted phenyl ketone oxime ethers: synthesis and bovine β3-adrenergic agonistic activities

Pharmazie, 2003-01, Vol.58 (1), p.13-17

2003

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DMD – BIOMARKERS & OUTCOME MEASURES

Neuromuscular disorders : NMD, 2020-10, Vol.30, p.S81

2020

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DMD – BIOMARKERS & OUTCOME MEASURES: P.120 Phenotype and genotype characterization as predictors of DMD 45 to 55 multi-exon skipping therapy

Neuromuscular disorders : NMD, 2020-10, Vol.30, p.S81-S81

2020

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G.P.1.10 DHPR α1 S subunit controls skeletal muscle mass and morphogenesis

P.4.10 Exon skipping as a therapeutic strategy applied to a RyR1 mutation causing severe core myopathy

Thiol reagents increase the affinity of the inositol 1,4,5-trisphosphate receptor

DMD TREATMENT: ANIMAL MODELS

DMD TREATMENT: ANIMAL MODELS: P.204R-DMDdel52, a novel preclinical rat model of Duchenne muscular dystrophy

P.288Multi-exon skipping by CRISPR/Cas9-mediated genome editing in GRMD dog

DMD TREATMENT

DMD TREATMENT: P.334PPMO pre-treatment is beneficial for AAV-based gene therapy in Duchenne muscular dystrophy

P.447 - From voltage sensing to gene expression in the control of muscle mass homeostasis

From voltage sensing to gene expression in the control of muscle mass homeostasis

P.20.11 Dystrophin rescue needed to recover a correct location of nNOS and the return to a normalized RyR1 status in treated GRMD dogs

G.P.93

Blockade of ActRIIB Signaling Triggers Muscle Fatigability and Metabolic Myopathy

P.237 - MicroRNAs involved in nNOS regulation in dystrophic context

MicroRNAs involved in nNOS regulation in dystrophic context

G.P.75 Variable phenotype of del45–55 Becker patients correlated to nNOSμ mislocalization and RYR1 hypernitrosylation

Pharmacological characteristics and species-related variations of β3-adrenergic receptors

P1.37 DHPR α1S subunit controls skeletal muscle mass and morphogenesis

Actin scaffolding by clathrin heavy chain is required for skeletal muscle sarcomere organization

FP.44 Exploring the role of MuscleBlind-Like proteins in the regulation of CaVB1 isoform expression in adult skeletal muscle

Human immortalized brown adipocytes express functional beta3-adrenoceptor coupled to lipolysis

Human adipose cells express CD4, CXCR4, and CCR5 receptors: a new target cell type for the immunodeficiency virus‐1?

New series of N-substituted phenyl ketone oxime ethers: synthesis and bovine β3-adrenergic agonistic activities

DMD – BIOMARKERS & OUTCOME MEASURES

DMD – BIOMARKERS & OUTCOME MEASURES: P.120 Phenotype and genotype characterization as predictors of DMD 45 to 55 multi-exon skipping therapy

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