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Epigenetic changes at Il12rb2 and Tbx21 in relation to plasticity behavior of Th17 cells

The Journal of immunology (1950), 2011-03, Vol.186 (6), p.3373-3382

2011

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Highly purified Th17 cells from BDC2.5NOD mice convert into Th1-like cells in NOD/SCID recipient mice

The Journal of clinical investigation, 2009-03, Vol.119 (3), p.565-572

2009

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Correction: Loss of Invariant Chain Protects Nonobese Diabetic Mice against Type 1 Diabetes

The Journal of immunology (1950), 2022-03, Vol.208 (6), p.1512-1512

2022

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Validated germline-competent embryonic stem cell lines from nonobese diabetic mice

Nature medicine, 2009-07, Vol.15 (7), p.814-818

2009

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Patients With Chronic Pancreatitis Have Islet Progenitor Cells in Their Ducts, but Reversal of Overt Diabetes in NOD Mice by Anti-CD3 Shows No Evidence for Islet Regeneration

Diabetes (New York, N.Y.), 2007-03, Vol.56 (3), p.634-640

2007

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Diabetes in non-obese diabetic mice is not associated with quantitative changes in CD4⁺ CD25⁺ Foxp3⁺ regulatory T cells

Immunology, 2007-05, Vol.121 (1), p.15-28

2007

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Cutting Edge: Interactions Through the IL-10 Receptor Regulate Autoimmune Diabetes

The Journal of immunology (1950), 2001-12, Vol.167 (11), p.6087-6091

2001

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PD‐L1 blockade overrides Salmonella typhimurium‐mediated diabetes prevention in NOD mice: No role for Tregs

European journal of immunology, 2011-10, Vol.41 (10), p.2966-2976

2011

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Type 1 diabetes development requires both CD4+ and CD8+ T cells and can be reversed by non-depleting antibodies targeting both T cell populations

The review of diabetic studies, 2009, Vol.6 (2), p.97-103

2009

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Roles for TGF-beta and programmed cell death 1 ligand 1 in regulatory T cell expansion and diabetes suppression by zymosan in nonobese diabetic mice

The Journal of immunology (1950), 2010-09, Vol.185 (5), p.2754-2762

2010

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early age-related increase in the frequency of CD4⁺ Foxp3⁺ cells in BDC2·5NOD mice

Immunology, 2007-08, Vol.121 (4), p.565-576

2007

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An early age-related increase in the frequency of CD4+ Foxp3+ cells in BDC2.5NOD mice

Immunology, 2007-08, Vol.121 (4), p.565-576

2007

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MAdCAM‐1 is needed for diabetes development mediated by the T cell clone, BDC‐2·5

Immunology, 2005-12, Vol.116 (4), p.525-531

2005

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Infection and autoimmunity: are we winning the war, only to lose the peace?

Trends in parasitology, 2004-07, Vol.20 (7), p.316-321

2004

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Loss of Invariant Chain Protects Nonobese Diabetic Mice against Type 1 Diabetes

Journal of Immunology, 2006-12, Vol.177 (11), p.7588-7598

2006

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Both central and peripheral tolerance mechanisms play roles in diabetes prevention in NOD-E transgenic mice

Autoimmunity (Chur, Switzerland), 2008-01, Vol.41 (5), p.383-394

2008

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Nondepleting anti-CD4 and soluble interleukin-1 receptor prevent autoimmune destruction of syngeneic islet grafts in diabetic NOD mice

Transplantation, 2002-09, Vol.74 (5), p.611

2002

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alpha-Cell neogenesis in an animal model of IDDM

Diabetes (New York, N.Y.), 1997-04, Vol.46 (4), p.599-606

1997

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Tolerogenic strategies to halt or prevent type 1 diabetes

Nature immunology, 2001-09, Vol.2 (9), p.810-815

2001

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Nondepleting anti-CD4 and soluble interleukin-1 receptor prevent autoimmune destruction of syngeneic islet grafts in diabetic NOD mice1

Transplantation, 2002-09, Vol.74 (5), p.611-619

2002

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MAdCAM-1 is needed for diabetes development mediated by the T cell clone, BDC-2.5

Immunology, 2005-12, Vol.116 (4), p.525-531

2005

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ORIGINAL ARTICLE: MAdCAM-1 is needed for diabetes development mediated by the T cell clone, BDC-2.5

Immunology, 2005-12, Vol.116 (4), p.525-531

2005

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Diabetes in non-obese diabetic mice is not associated with quantitative changes in CD4 super(+) CD25 super(+) Foxp3 super(+) regulatory T cells

Immunology, 2007-05, Vol.121 (1), p.15-28

2007

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TOLERANCE AND SUPPRESSION IN A PRIMED IMMUNE SYSTEM1

Transplantation, 1996-12, Vol.62 (11), p.1614-1621

1996

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An early age-related increase in the frequency of CD4 super(+) Foxp3 super(+) cells in BDC2.5NOD mice

Immunology, 2007-08, Vol.121 (4), p.565-576

2007

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Epigenetic changes at Il12rb2 and Tbx21 in relation to plasticity behavior of Th17 cells

Highly purified Th17 cells from BDC2.5NOD mice convert into Th1-like cells in NOD/SCID recipient mice

Correction: Loss of Invariant Chain Protects Nonobese Diabetic Mice against Type 1 Diabetes

Validated germline-competent embryonic stem cell lines from nonobese diabetic mice

Patients With Chronic Pancreatitis Have Islet Progenitor Cells in Their Ducts, but Reversal of Overt Diabetes in NOD Mice by Anti-CD3 Shows No Evidence for Islet Regeneration

Diabetes in non-obese diabetic mice is not associated with quantitative changes in CD4⁺ CD25⁺ Foxp3⁺ regulatory T cells

Cutting Edge: Interactions Through the IL-10 Receptor Regulate Autoimmune Diabetes

PD‐L1 blockade overrides Salmonella typhimurium‐mediated diabetes prevention in NOD mice: No role for Tregs

Type 1 diabetes development requires both CD4+ and CD8+ T cells and can be reversed by non-depleting antibodies targeting both T cell populations

Roles for TGF-beta and programmed cell death 1 ligand 1 in regulatory T cell expansion and diabetes suppression by zymosan in nonobese diabetic mice

early age-related increase in the frequency of CD4⁺ Foxp3⁺ cells in BDC2·5NOD mice

An early age-related increase in the frequency of CD4+ Foxp3+ cells in BDC2.5NOD mice

MAdCAM‐1 is needed for diabetes development mediated by the T cell clone, BDC‐2·5

Infection and autoimmunity: are we winning the war, only to lose the peace?

Loss of Invariant Chain Protects Nonobese Diabetic Mice against Type 1 Diabetes

Both central and peripheral tolerance mechanisms play roles in diabetes prevention in NOD-E transgenic mice

Nondepleting anti-CD4 and soluble interleukin-1 receptor prevent autoimmune destruction of syngeneic islet grafts in diabetic NOD mice

alpha-Cell neogenesis in an animal model of IDDM

Tolerogenic strategies to halt or prevent type 1 diabetes

Nondepleting anti-CD4 and soluble interleukin-1 receptor prevent autoimmune destruction of syngeneic islet grafts in diabetic NOD mice1

MAdCAM-1 is needed for diabetes development mediated by the T cell clone, BDC-2.5

ORIGINAL ARTICLE: MAdCAM-1 is needed for diabetes development mediated by the T cell clone, BDC-2.5

Diabetes in non-obese diabetic mice is not associated with quantitative changes in CD4 super(+) CD25 super(+) Foxp3 super(+) regulatory T cells

TOLERANCE AND SUPPRESSION IN A PRIMED IMMUNE SYSTEM1

An early age-related increase in the frequency of CD4 super(+) Foxp3 super(+) cells in BDC2.5NOD mice

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