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Carcinoma-associated fibroblasts: orchestrating the composition of malignancy

Genes & development, 2016-05, Vol.30 (9), p.1002-1019

2016

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An Oral Small Molecule VEGFR2 Inhibitor, Apatinib, in Patients with Recurrent or Refractory Cervical Cancer: A Real World Study

Journal of oncology, 2020-06, Vol.2020, p.1-6

2020

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Clinical Outcome of Carotid Body Paraganglioma Management: A Review of 10-Year Experience

Journal of oncology, 2020, Vol.2020, p.6081273-6

2020

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Stromal directives can control cancer

Science (American Association for the Advancement of Science), 2019-07, Vol.365 (6449), p.122-123

2019

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Rare somatic cells from human breast tissue exhibit extensive lineage plasticity

Proceedings of the National Academy of Sciences - PNAS, 2013-03, Vol.110 (12), p.4598-4603

2013

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Basal Subtype and MAPK/ERK Kinase (MEK)-Phosphoinositide 3-Kinase Feedback Signaling Determine Susceptibility of Breast Cancer Cells to MEK Inhibition

Cancer research (Chicago, Ill.), 2009-01, Vol.69 (2), p.565-572

2009

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Intermediate DNA methylation is a conserved signature of genome regulation

Nature communications, 2015-02, Vol.6 (1), p.6363-6363, Article 6363

2015

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Epigenetic and transcriptional determinants of the human breast

Nature communications, 2015-02, Vol.6 (1), p.6351-6351, Article 6351

2015

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Coordinate Transcriptional and Translational Repression of p53 by TGF-β1 Impairs the Stress Response

Molecular cell, 2013-05, Vol.50 (4), p.552-564

2013

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Regulatory network decoded from epigenomes of surface ectoderm-derived cell types

Nature communications, 2014-11, Vol.5 (1), p.5442-5442, Article 5442

2014

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Stromal directives can control cancer: Could reprogramming the pretumor microenvironment transform cancer care?

Science (American Association for the Advancement of Science), 2019-07, Vol.365 (6449), p.122-123

2019

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Insights into the Function of the Unstructured N-Terminal Domain of Proteins 4.1R and 4.1G in Erythropoiesis

International Journal of Cell Biology, 2011, Vol.2011, p.943272-13

2011

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Similarities and differences in the structure and function of 4.1G and 4.1R135, two protein 4.1 paralogues expressed in erythroid cells

Biochemical journal, 2010-12, Vol.432 (2), p.407-416

2010

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Identification of a third Protein 4.1 tumor suppressor, Protein 4.1R, in meningioma pathogenesis

Neurobiology of disease, 2003-08, Vol.13 (3), p.191-202

2003

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Integrative analysis of 111 reference human epigenomes

Nature (London), 2015-02, Vol.518 (7539), p.317-330

2015

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Differential domain evolution and complex RNA processing in a family of paralogous EPB41 (protein 4.1) genes facilitate expression of diverse tissue-specific isoforms

Genomics (San Diego, Calif.), 2004-10, Vol.84 (4), p.637-646

2004

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Molecular and Functional Characterization of Protein 4.1B, a Novel Member of the Protein 4.1 Family with High Level, Focal Expression in Brain

The Journal of biological chemistry, 2000-02, Vol.275 (5), p.3247-3255

2000

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The clonal and mutational evolution spectrum of primary triple―negative breast cancers

Nature (London), 2012-06, Vol.486 (7403), p.395-399

2012

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Distinct distribution of specific members of protein 4.1 gene family in the mouse nephron

Kidney international, 2003-04, Vol.63 (4), p.1321-1337

2003

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223 IMMUNE CELL PHENOTYPING IN BARRETT'S ESOPHAGUS IN PATIENTS PRIOR AND AT TIME OF PROGRESSION

Gastroenterology (New York, N.Y. 1943), 2023-05, Vol.164 (6), p.S-41-S-41

2023

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Apposition of Fibroblasts With Metaplastic Gastric Cells Promotes Dysplastic Transition

Gastroenterology (New York, N.Y. 1943), 2023-08, Vol.165 (2), p.374-390

2023

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Su1246 USING MASSIVE MULTIPLEX IMMUNOFLUORESENCE (CODEX) TO DEFINE THE INTRAEPITHELIAL IMMUNE CELL MICROENVIRONMENT IN BARRETT'S ESOPHAGUS IN PATIENTS THAT HAVE PROGRESSED TO CANCER

Gastroenterology (New York, N.Y. 1943), 2024-05, Vol.166 (5), p.S-707-S-707

2024

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DNA hypomethylation within specific transposable element families associates with tissue-specific enhancer landscape

Nature genetics, 2013-07, Vol.45 (7), p.836-841

2013

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Loss of PPARγ activity characterizes early protumorigenic stromal reprogramming and dictates the therapeutic window of opportunity

Proceedings of the National Academy of Sciences - PNAS, 2023-10, Vol.120 (42), p.e2303774120-e2303774120

2023

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Carcinoma-associated fibroblasts: orchestrating the composition of malignancy

An Oral Small Molecule VEGFR2 Inhibitor, Apatinib, in Patients with Recurrent or Refractory Cervical Cancer: A Real World Study

Clinical Outcome of Carotid Body Paraganglioma Management: A Review of 10-Year Experience

Stromal directives can control cancer

Rare somatic cells from human breast tissue exhibit extensive lineage plasticity

Functional DNA methylation differences between tissues, cell types, and across individuals discovered using the M&M algorithm

Basal Subtype and MAPK/ERK Kinase (MEK)-Phosphoinositide 3-Kinase Feedback Signaling Determine Susceptibility of Breast Cancer Cells to MEK Inhibition

Intermediate DNA methylation is a conserved signature of genome regulation

Epigenetic and transcriptional determinants of the human breast

Coordinate Transcriptional and Translational Repression of p53 by TGF-β1 Impairs the Stress Response

Regulatory network decoded from epigenomes of surface ectoderm-derived cell types

Stromal directives can control cancer: Could reprogramming the pretumor microenvironment transform cancer care?

Insights into the Function of the Unstructured N-Terminal Domain of Proteins 4.1R and 4.1G in Erythropoiesis

Similarities and differences in the structure and function of 4.1G and 4.1R135, two protein 4.1 paralogues expressed in erythroid cells

Identification of a third Protein 4.1 tumor suppressor, Protein 4.1R, in meningioma pathogenesis

Integrative analysis of 111 reference human epigenomes

Differential domain evolution and complex RNA processing in a family of paralogous EPB41 (protein 4.1) genes facilitate expression of diverse tissue-specific isoforms

Molecular and Functional Characterization of Protein 4.1B, a Novel Member of the Protein 4.1 Family with High Level, Focal Expression in Brain

The clonal and mutational evolution spectrum of primary triple―negative breast cancers

Distinct distribution of specific members of protein 4.1 gene family in the mouse nephron

223 IMMUNE CELL PHENOTYPING IN BARRETT'S ESOPHAGUS IN PATIENTS PRIOR AND AT TIME OF PROGRESSION

Apposition of Fibroblasts With Metaplastic Gastric Cells Promotes Dysplastic Transition

Su1246 USING MASSIVE MULTIPLEX IMMUNOFLUORESENCE (CODEX) TO DEFINE THE INTRAEPITHELIAL IMMUNE CELL MICROENVIRONMENT IN BARRETT'S ESOPHAGUS IN PATIENTS THAT HAVE PROGRESSED TO CANCER

DNA hypomethylation within specific transposable element families associates with tissue-specific enhancer landscape

Loss of PPARγ activity characterizes early protumorigenic stromal reprogramming and dictates the therapeutic window of opportunity

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