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Cytokines and Chemokines at the Crossroads of Neuroinflammation, Neurodegeneration, and Neuropathic Pain

Mediators of Inflammation, 2013-01, Vol.2013 (2013), p.646-665-159

2013

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Variable manifestations, diverse seroreactivity and post-treatment persistence in non-human primates exposed to Borrelia burgdorferi by tick feeding

PloS one, 2017-12, Vol.12 (12), p.e0189071

2017

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Analysis of an ordered, comprehensive STM mutant library in infectious Borrelia burgdorferi: insights into the genes required for mouse infectivity

PloS one, 2012-10, Vol.7 (10), p.e47532

2012

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Persistence of Borrelia burgdorferi in rhesus macaques following antibiotic treatment of disseminated infection

PloS one, 2012-01, Vol.7 (1), p.e29914-e29914

2012

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Receptor tyrosine kinases play a significant role in human oligodendrocyte inflammation and cell death associated with the Lyme disease bacterium Borrelia burgdorferi

Journal of neuroinflammation, 2017-05, Vol.14 (1), p.110-110, Article 110

2017

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A possible role for inflammation in mediating apoptosis of oligodendrocytes as induced by the Lyme disease spirochete Borrelia burgdorferi

Journal of neuroinflammation, 2012-04, Vol.9 (1), p.72-72, Article 72

2012

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Repeated Tick Infestations Impair Borrelia burgdorferi Transmission in a Non-Human Primate Model of Tick Feeding

Pathogens (Basel), 2023-01, Vol.12 (1), p.132

2023

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Human microglia and astrocytes constitutively express the neurokinin-1 receptor and functionally respond to substance P

Journal of neuroinflammation, 2017-12, Vol.14 (1), p.245-245, Article 245

2017

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Central role of the Holliday junction helicase RuvAB in vlsE recombination and infectivity of Borrelia burgdorferi

PLoS pathogens, 2009-12, Vol.5 (12), p.e1000679-1000679

2009

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Microglia are mediators of Borrelia burgdorferi-induced apoptosis in SH-SY5Y neuronal cells

PLoS pathogens, 2009-11, Vol.5 (11), p.e1000659

2009

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Interleukin-10 Alters Effector Functions of Multiple Genes Induced by Borrelia burgdorferi in Macrophages To Regulate Lyme Disease Inflammation

Infection and Immunity, 2011-12, Vol.79 (12), p.4876-4892

2011

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Live Borrelia burgdorferi Spirochetes Elicit Inflammatory Mediators from Human Monocytes via the Toll-Like Receptor Signaling Pathway

Infection and Immunity, 2009-03, Vol.77 (3), p.1238-1245

2009

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Different patterns of expression and of IL-10 modulation of inflammatory mediators from macrophages of Lyme disease-resistant and -susceptible mice

PloS one, 2012-09, Vol.7 (9), p.e43860-e43860

2012

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Serodiagnosis of Lyme Disease by Kinetic Enzyme-Linked Immunosorbent Assay Using Recombinant VlsE1 or Peptide Antigens of Borrelia burgdorferi Compared with 2-Tiered Testing Using Whole-Cell Lysates

The Journal of infectious diseases, 2003-04, Vol.187 (8), p.1187-1199

2003

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Toll-Like Receptors: Insights into Their Possible Role in the Pathogenesis of Lyme Neuroborreliosis

Infection and Immunity, 2008-10, Vol.76 (10), p.4385-4395

2008

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Review: apoptotic mechanisms in bacterial infections of the central nervous system

Frontiers in immunology, 2012-01, Vol.3, p.306-306

2012

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Dynamic Longitudinal Antibody Responses during Borrelia burgdorferi Infection and Antibiotic Treatment of Rhesus Macaques

Clinical and Vaccine Immunology, 2012-08, Vol.19 (8), p.1218-1226

2012

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Multi-platform Approach for Microbial Biomarker Identification Using Borrelia burgdorferi as a Model

Frontiers in cellular and infection microbiology, 2019-06, Vol.9, p.179-179

2019

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Possible role of glial cells in the onset and progression of Lyme neuroborreliosis

Journal of neuroinflammation, 2009-08, Vol.6 (1), p.23-23, Article 23

2009

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Intracellular TLR7 is activated in human oligodendrocytes in response to Borrelia burgdorferi exposure

Neuroscience letters, 2018-04, Vol.671, p.38-42

2018

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Failure of Immune Response Evasion by Linear Plasmid 28-1-Deficient Borrelia burgdorferi Is Attributable to Persistent Expression of an Outer Surface Protein

Infection and Immunity, 2008-09, Vol.76 (9), p.3984-3991

2008

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Effect of Borrelia burgdorferi Genotype on the Sensitivity of C6 and 2-Tier Testing in North American Patients with Culture-Confirmed Lyme Disease

Clinical infectious diseases, 2008-10, Vol.47 (7), p.910-914

2008

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Interleukin-10 Anti-Inflammatory Response to Borrelia burgdorferi, the Agent of Lyme Disease: a Possible Role for Suppressors of Cytokine Signaling 1 and 3

Infection and Immunity, 2006-10, Vol.74 (10), p.5780-5789

2006

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The MEK/ERK pathway is the primary conduit for Borrelia burgdorferi-induced inflammation and P53-mediated apoptosis in oligodendrocytes

Apoptosis (London), 2014, Vol.19 (1), p.76-89

2014

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Mediators of Neuroinflammation

Mediators of Inflammation, 2013-01, Vol.2013 (2013), p.939-940-083

2013

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Cytokines and Chemokines at the Crossroads of Neuroinflammation, Neurodegeneration, and Neuropathic Pain

Variable manifestations, diverse seroreactivity and post-treatment persistence in non-human primates exposed to Borrelia burgdorferi by tick feeding

Analysis of an ordered, comprehensive STM mutant library in infectious Borrelia burgdorferi: insights into the genes required for mouse infectivity

Persistence of Borrelia burgdorferi in rhesus macaques following antibiotic treatment of disseminated infection

Receptor tyrosine kinases play a significant role in human oligodendrocyte inflammation and cell death associated with the Lyme disease bacterium Borrelia burgdorferi

A possible role for inflammation in mediating apoptosis of oligodendrocytes as induced by the Lyme disease spirochete Borrelia burgdorferi

Repeated Tick Infestations Impair Borrelia burgdorferi Transmission in a Non-Human Primate Model of Tick Feeding

Human microglia and astrocytes constitutively express the neurokinin-1 receptor and functionally respond to substance P

Central role of the Holliday junction helicase RuvAB in vlsE recombination and infectivity of Borrelia burgdorferi

Microglia are mediators of Borrelia burgdorferi-induced apoptosis in SH-SY5Y neuronal cells

Interleukin-10 Alters Effector Functions of Multiple Genes Induced by Borrelia burgdorferi in Macrophages To Regulate Lyme Disease Inflammation

Live Borrelia burgdorferi Spirochetes Elicit Inflammatory Mediators from Human Monocytes via the Toll-Like Receptor Signaling Pathway

Different patterns of expression and of IL-10 modulation of inflammatory mediators from macrophages of Lyme disease-resistant and -susceptible mice

Serodiagnosis of Lyme Disease by Kinetic Enzyme-Linked Immunosorbent Assay Using Recombinant VlsE1 or Peptide Antigens of Borrelia burgdorferi Compared with 2-Tiered Testing Using Whole-Cell Lysates

Toll-Like Receptors: Insights into Their Possible Role in the Pathogenesis of Lyme Neuroborreliosis

Review: apoptotic mechanisms in bacterial infections of the central nervous system

Dynamic Longitudinal Antibody Responses during Borrelia burgdorferi Infection and Antibiotic Treatment of Rhesus Macaques

Multi-platform Approach for Microbial Biomarker Identification Using Borrelia burgdorferi as a Model

Possible role of glial cells in the onset and progression of Lyme neuroborreliosis

Intracellular TLR7 is activated in human oligodendrocytes in response to Borrelia burgdorferi exposure

Failure of Immune Response Evasion by Linear Plasmid 28-1-Deficient Borrelia burgdorferi Is Attributable to Persistent Expression of an Outer Surface Protein

Effect of Borrelia burgdorferi Genotype on the Sensitivity of C6 and 2-Tier Testing in North American Patients with Culture-Confirmed Lyme Disease

Interleukin-10 Anti-Inflammatory Response to Borrelia burgdorferi, the Agent of Lyme Disease: a Possible Role for Suppressors of Cytokine Signaling 1 and 3

The MEK/ERK pathway is the primary conduit for Borrelia burgdorferi-induced inflammation and P53-mediated apoptosis in oligodendrocytes

Mediators of Neuroinflammation

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