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The epigenetic landscape related to reactive oxygen species formation in the cardiovascular system

British journal of pharmacology, 2017-06, Vol.174 (12), p.1533-1554

2017

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The NADPH oxidase subunit NOX4 is a new target gene of the hypoxia-inducible factor-1

Molecular biology of the cell, 2010-06, Vol.21 (12), p.2087-2096

2010

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Inflammation and Fibrosis in Progeria: Organ-Specific Responses in an HGPS Mouse Model

International journal of molecular sciences, 2024-08, Vol.25 (17), p.9323

2024

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NOX2 and NOX4 mediate proliferative response in endothelial cells

Antioxidants & redox signaling, 2006-09, Vol.8 (9-10), p.1473-1484

2006

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NOX4 mediates activation of FoxO3a and matrix metalloproteinase-2 expression by urotensin-II

Molecular biology of the cell, 2011-11, Vol.22 (22), p.4424-4434

2011

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NADPH oxidases and HIF1 promote cardiac dysfunction and pulmonary hypertension in response to glucocorticoid excess

Redox biology, 2020-07, Vol.34, p.101536-101536, Article 101536

2020

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Immunoproteasome subunit ß5i/LMP7-deficiency in atherosclerosis

Scientific reports, 2017-10, Vol.7 (1), p.13342-10, Article 13342

2017

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Phosphodiesterase 2 Mediates Redox-Sensitive Endothelial Cell Proliferation and Angiogenesis by Thrombin via Rac1 and NADPH Oxidase 2

Circulation research, 2009-05, Vol.104 (10), p.1169-1177

2009

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NADPH-Oxidasen und HIF: zentrale Elemente der Redoxhomöostase

Biospektrum, 2021-03, Vol.27 (2), p.142-145

2021

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Regulation of NADPH Oxidases by G Protein-Coupled Receptors

Antioxidants & redox signaling, 2019-01, Vol.30 (1), p.74-94

2019

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NADPH-Oxidasen und HIF: zentrale Elemente der Redoxhomöostase

Biospektrum, 2021-03, Vol.27 (2), p.142-145

2021

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Receptor activation of NADPH oxidases

Antioxidants & redox signaling, 2010-08, Vol.13 (4), p.467-487

2010

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Reactive oxygen species, nutrition, hypoxia and diseases: Problems solved?

Redox biology, 2015-12, Vol.6, p.372-385

2015

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Reactive oxygen species, nutrition, hypoxia and diseases: Problems solved?

Redox biology, 2015-12, Vol.6, p.372-385

2015

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Abstract B51: Adaptation to oncogene-induced metabolic stress by MondoA (MLXIP) drives common acute lymphoblastic leukemia (cALL) malignancy

Cancer research (Chicago, Ill.), 2020-07, Vol.80 (14_Supplement), p.B51-B51

2020

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Cross Talk Between p22phox and ATF4 in the Endothelial Unfolded Protein Response

Antioxidants & redox signaling, 2019-01, Vol.30 (1), p.40-55

2019

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Stabilization of p22phox by Hypoxia Promotes Pulmonary Hypertension

Antioxidants & redox signaling, 2019-01, Vol.30 (1), p.56-73

2019

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The HIF1 target gene NOX2 promotes angiogenesis through urotensin-II

Journal of cell science, 2012-02, Vol.125 (Pt 4), p.956-964

2012

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Adaptation to Metabolic Stress By Mondoα in Common B-Cell Acute Lymphoblastic Leukemia

Blood, 2018-11, Vol.132 (Supplement 1), p.3888-3888

2018

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NOX5 variants are functionally active in endothelial cells

Free radical biology & medicine, 2007-02, Vol.42 (4), p.446-459

2007

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Differential transcriptional regulation of hypoxia-inducible factor-1α by arsenite under normoxia and hypoxia: involvement of Nrf2

Journal of molecular medicine (Berlin, Germany), 2016-10, Vol.94 (10), p.1153-1166

2016

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Volumetric optoacoustic tomography enables non-invasive in vivo characterization of impaired heart function in hypoxic conditions

Scientific reports, 2019-06, Vol.9 (1), p.8369, Article 8369

2019

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MondoA drives malignancy in B-ALL through enhanced adaptation to metabolic stress

Blood, 2022-02, Vol.139 (8), p.1184-1197

2022

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Prolyl-hydroxylase inhibition induces SDF-1 associated with increased CXCR4+/CD11b+ subpopulations and cardiac repair

Journal of molecular medicine (Berlin, Germany), 2017-08, Vol.95 (8), p.825-837

2017

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Differential transcriptional regulation of hypoxia-inducible factor-1[alpha] by arsenite under normoxia and hypoxia: involvement of Nrf2

Journal of molecular medicine (Berlin, Germany), 2016-10, Vol.94 (10), p.1153

2016

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The epigenetic landscape related to reactive oxygen species formation in the cardiovascular system

The NADPH oxidase subunit NOX4 is a new target gene of the hypoxia-inducible factor-1

Inflammation and Fibrosis in Progeria: Organ-Specific Responses in an HGPS Mouse Model

NOX2 and NOX4 mediate proliferative response in endothelial cells

NOX4 mediates activation of FoxO3a and matrix metalloproteinase-2 expression by urotensin-II

NADPH oxidases and HIF1 promote cardiac dysfunction and pulmonary hypertension in response to glucocorticoid excess

Immunoproteasome subunit ß5i/LMP7-deficiency in atherosclerosis

Phosphodiesterase 2 Mediates Redox-Sensitive Endothelial Cell Proliferation and Angiogenesis by Thrombin via Rac1 and NADPH Oxidase 2

NADPH-Oxidasen und HIF: zentrale Elemente der Redoxhomöostase

Regulation of NADPH Oxidases by G Protein-Coupled Receptors

NADPH-Oxidasen und HIF: zentrale Elemente der Redoxhomöostase

Receptor activation of NADPH oxidases

Reactive oxygen species, nutrition, hypoxia and diseases: Problems solved?

Reactive oxygen species, nutrition, hypoxia and diseases: Problems solved?

Abstract B51: Adaptation to oncogene-induced metabolic stress by MondoA (MLXIP) drives common acute lymphoblastic leukemia (cALL) malignancy

Cross Talk Between p22phox and ATF4 in the Endothelial Unfolded Protein Response

Stabilization of p22phox by Hypoxia Promotes Pulmonary Hypertension

The HIF1 target gene NOX2 promotes angiogenesis through urotensin-II

Adaptation to Metabolic Stress By Mondoα in Common B-Cell Acute Lymphoblastic Leukemia

NOX5 variants are functionally active in endothelial cells

Differential transcriptional regulation of hypoxia-inducible factor-1α by arsenite under normoxia and hypoxia: involvement of Nrf2

Volumetric optoacoustic tomography enables non-invasive in vivo characterization of impaired heart function in hypoxic conditions

MondoA drives malignancy in B-ALL through enhanced adaptation to metabolic stress

Prolyl-hydroxylase inhibition induces SDF-1 associated with increased CXCR4+/CD11b+ subpopulations and cardiac repair

Differential transcriptional regulation of hypoxia-inducible factor-1[alpha] by arsenite under normoxia and hypoxia: involvement of Nrf2

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