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From CFTR biology toward combinatorial pharmacotherapy: expanded classification of cystic fibrosis mutations

Molecular biology of the cell, 2016-02, Vol.27 (3), p.424-433

2016

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Cystic Fibrosis Transmembrane Conductance Regulator Folding Mutations Reveal Differences in Corrector Efficacy Linked to Increases in Immature Cystic Fibrosis Transmembrane Conductance Regulator Expression

Frontiers in physiology, 2021-10, Vol.12, p.695767-695767

2021

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Small heat shock proteins target mutant cystic fibrosis transmembrane conductance regulator for degradation via a small ubiquitin-like modifier-dependent pathway

Molecular biology of the cell, 2013-01, Vol.24 (2), p.74-84

2013

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Phosphorylation-dependent 14-3-3 protein interactions regulate CFTR biogenesis

Molecular biology of the cell, 2012-03, Vol.23 (6), p.996-1009

2012

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Co-Translational Folding of the First Transmembrane Domain of ABC-Transporter CFTR is Supported by Assembly with the First Cytosolic Domain

Journal of molecular biology, 2021-06, Vol.433 (13), p.166955-166955, Article 166955

2021

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14-3-3 Isoforms Are Induced by Aldosterone and Participate in Its Regulation of Epithelial Sodium Channels

The Journal of biological chemistry, 2006-06, Vol.281 (24), p.16323-16332

2006

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The Epithelial Sodium Channel (ENaC) Traffics to Apical Membrane in Lipid Rafts in Mouse Cortical Collecting Duct Cells

The Journal of biological chemistry, 2007-12, Vol.282 (52), p.37402-37411

2007

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Regulation of CFTR Trafficking by Its R Domain

The Journal of biological chemistry, 2008-10, Vol.283 (42), p.28401-28412

2008

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Identification and membrane localization of electrogenic sodium bicarbonate cotransporters in Calu-3 cells

Biochimica et biophysica acta, 2006-07, Vol.1762 (7), p.704-710

2006

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AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking

Molecular biology of the cell, 2010-06, Vol.21 (12), p.2024-2033

2010

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An Obligatory Heterodimer of 14-3-3β and 14-3-3ϵ Is Required for Aldosterone Regulation of the Epithelial Sodium Channel

The Journal of biological chemistry, 2008-10, Vol.283 (41), p.27418-27425

2008

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Cysteine String Protein Interacts with and Modulates the Maturation of the Cystic Fibrosis Transmembrane Conductance Regulator

The Journal of biological chemistry, 2002-08, Vol.277 (32), p.28948-28958

2002

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Pharmacological rescue of the mutant cystic fibrosis transmembrane conductance regulator (CFTR) detected by use of a novel fluorescence platform

Molecular medicine (Cambridge, Mass.), 2012-04, Vol.18 (4), p.685-696

2012

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Airway Surface Liquid Volume Regulates ENaC by Altering the Serine Protease-Protease Inhibitor Balance: A MECHANISM FOR SODIUM HYPERABSORPTION IN CYSTIC FIBROSIS

The Journal of biological chemistry, 2006-09, Vol.281 (38), p.27942-27949

2006

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Clathrin-mediated endocytosis of the epithelial sodium channel. Role of epsin

The Journal of biological chemistry, 2006-05, Vol.281 (20), p.14129-14135

2006

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CFTR Folding Consortium: methods available for studies of CFTR folding and correction

Methods in molecular biology (Clifton, N.J.), 2011, Vol.742, p.335

2011

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Respiratory Carcinoma Cell Lines . MUC Genes and Glycoconjugates

American journal of respiratory cell and molecular biology, 1999-03, Vol.20 (3), p.500-510

1999

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Regulation of the Amiloride-sensitive Epithelial Sodium Channel by Syntaxin 1A

The Journal of biological chemistry, 1999-10, Vol.274 (43), p.30345-30348

1999

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Syntaxin 1A inhibits regulated CFTR trafficking in Xenopus oocytes

American Journal of Physiology: Cell Physiology, 1999-07, Vol.277 (1), p.C174-C180

1999

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Role of snare proteins in CFTR and ENaC trafficking

Pflügers Archiv, 2001-01, Vol.443 Suppl 1, p.S65-S69

2001

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Airway Surface Liquid Volume Regulates ENaC by Altering the Serine Protease-Protease Inhibitor Balance

The Journal of biological chemistry, 2006-09, Vol.281 (38), p.27942-27949

2006

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Clathrin-mediated Endocytosis of the Epithelial Sodium Channel

The Journal of biological chemistry, 2006-05, Vol.281 (20), p.14129-14135

2006

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The Rab‐GAP, AS160, participates in the regulation of apical membrane epithelial sodium channel (ENaC) density and recycling

The FASEB journal, 2010-04, Vol.24 (S1), p.1024.1-1024.1

2010

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Regulation of CFTR Trafficking by Its R DomainS

The Journal of biological chemistry, 2008-10, Vol.283 (42), p.28401-28412

2008

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An Obligatory Heterodimer of 14-3-3β and 14-3-3ε Is Required for Aldosterone Regulation of the Epithelial Sodium ChannelS

The Journal of biological chemistry, 2008-10, Vol.283 (41), p.27418-27425

2008

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From CFTR biology toward combinatorial pharmacotherapy: expanded classification of cystic fibrosis mutations

Cystic Fibrosis Transmembrane Conductance Regulator Folding Mutations Reveal Differences in Corrector Efficacy Linked to Increases in Immature Cystic Fibrosis Transmembrane Conductance Regulator Expression

Small heat shock proteins target mutant cystic fibrosis transmembrane conductance regulator for degradation via a small ubiquitin-like modifier-dependent pathway

Phosphorylation-dependent 14-3-3 protein interactions regulate CFTR biogenesis

Co-Translational Folding of the First Transmembrane Domain of ABC-Transporter CFTR is Supported by Assembly with the First Cytosolic Domain

14-3-3 Isoforms Are Induced by Aldosterone and Participate in Its Regulation of Epithelial Sodium Channels

The Epithelial Sodium Channel (ENaC) Traffics to Apical Membrane in Lipid Rafts in Mouse Cortical Collecting Duct Cells

Regulation of CFTR Trafficking by Its R Domain

Identification and membrane localization of electrogenic sodium bicarbonate cotransporters in Calu-3 cells

AS160 modulates aldosterone-stimulated epithelial sodium channel forward trafficking

An Obligatory Heterodimer of 14-3-3β and 14-3-3ϵ Is Required for Aldosterone Regulation of the Epithelial Sodium Channel

Cysteine String Protein Interacts with and Modulates the Maturation of the Cystic Fibrosis Transmembrane Conductance Regulator

Pharmacological rescue of the mutant cystic fibrosis transmembrane conductance regulator (CFTR) detected by use of a novel fluorescence platform

Airway Surface Liquid Volume Regulates ENaC by Altering the Serine Protease-Protease Inhibitor Balance: A MECHANISM FOR SODIUM HYPERABSORPTION IN CYSTIC FIBROSIS

Clathrin-mediated endocytosis of the epithelial sodium channel. Role of epsin

CFTR Folding Consortium: methods available for studies of CFTR folding and correction

Respiratory Carcinoma Cell Lines . MUC Genes and Glycoconjugates

Regulation of the Amiloride-sensitive Epithelial Sodium Channel by Syntaxin 1A

Syntaxin 1A inhibits regulated CFTR trafficking in Xenopus oocytes

Role of snare proteins in CFTR and ENaC trafficking

Airway Surface Liquid Volume Regulates ENaC by Altering the Serine Protease-Protease Inhibitor Balance

Clathrin-mediated Endocytosis of the Epithelial Sodium Channel

The Rab‐GAP, AS160, participates in the regulation of apical membrane epithelial sodium channel (ENaC) density and recycling

Regulation of CFTR Trafficking by Its R DomainS

An Obligatory Heterodimer of 14-3-3β and 14-3-3ε Is Required for Aldosterone Regulation of the Epithelial Sodium ChannelS

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