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Let-7 and miR-200 microRNAs: Guardians against pluripotency and cancer progression

Cell cycle (Georgetown, Tex.), 2009-03, Vol.8 (6), p.843-852

Peter, Marcus E.

2009

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Cancer kill code extension

Molecular therapy. Nucleic acids, 2023-09, Vol.33, p.773-773

Peter, Marcus E.

2023

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The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

Genes & development, 2008-04, Vol.22 (7), p.894-907

2008

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The role of let-7 in cell differentiation and cancer

Endocrine-related cancer, 2010-03, Vol.17 (1), p.F19-F36

2010

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Adipocytes promote ovarian cancer metastasis and provide energy for rapid tumor growth

Nature medicine, 2011-11, Vol.17 (11), p.1498-1503

2011

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SPOROS: A pipeline to analyze DISE/6mer seed toxicity

PLoS computational biology, 2022-03, Vol.18 (3), p.e1010022-e1010022

2022

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Cell type-restricted activity of hnRNPM promotes breast cancer metastasis via regulating alternative splicing

Genes & development, 2014-06, Vol.28 (11), p.1191-1203

2014

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Genomics of Ovarian Cancer Progression Reveals Diverse Metastatic Trajectories Including Intraepithelial Metastasis to the Fallopian Tube

Cancer discovery, 2016-12, Vol.6 (12), p.1342-1351

2016

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Cancer-Associated IDH1 Promotes Growth and Resistance to Targeted Therapies in the Absence of Mutation

Cell reports (Cambridge), 2017-05, Vol.19 (9), p.1858-1873

2017

Volltextzugriff (PDF)

Coordinated epigenetic repression of the miR‐200 family and miR‐205 in invasive bladder cancer

International journal of cancer, 2011-03, Vol.128 (6), p.1327-1334

2011

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Let-7 modulates acquired resistance of ovarian cancer to Taxanes via IMP-1-mediated stabilization of multidrug resistance 1

International journal of cancer, 2012-04, Vol.130 (8), p.1787-1797

2012

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Let-7 Expression Defines Two Differentiation Stages of Cancer

Proceedings of the National Academy of Sciences - PNAS, 2007-07, Vol.104 (27), p.11400-11405

2007

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6mer seed toxicity in tumor suppressive microRNAs

Nature communications, 2018-10, Vol.9 (1), p.4504-16, Article 4504

2018

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CD95/Fas Increases Stemness in Cancer Cells by Inducing a STAT1-Dependent Type I Interferon Response

Cell reports (Cambridge), 2017-03, Vol.18 (10), p.2373-2386

2017

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CD95 is part of a let-7/p53/miR-34 regulatory network

PloS one, 2012-11, Vol.7 (11), p.e49636-e49636

2012

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DISE/6mer seed toxicity-a powerful anti-cancer mechanism with implications for other diseases

Journal of experimental & clinical cancer research, 2021-12, Vol.40 (1), p.389-12, Article 389

2021

Volltextzugriff (PDF)

CD95 and CD95L promote and protect cancer stem cells

Nature communications, 2014-11, Vol.5 (1), p.5238-5238, Article 5238

2014

Volltextzugriff (PDF)

Identification of the toxic 6mer seed consensus for human cancer cells

Scientific reports, 2022-03, Vol.12 (1), p.5130-5130, Article 5130

2022

Volltextzugriff (PDF)

Loss of E-Cadherin Promotes Ovarian Cancer Metastasis via α5-Integrin, which Is a Therapeutic Target

Cancer research (Chicago, Ill.), 2008-04, Vol.68 (7), p.2329-2339

2008

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Death induced by CD95 or CD95 ligand elimination

Cell reports (Cambridge), 2014-04, Vol.7 (1), p.208-222

2014

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Death Induced by Survival gene Elimination (DISE) correlates with neurotoxicity in Alzheimer's disease and aging

Nature communications, 2024-01, Vol.15 (1), p.264-264, Article 264

2024

Volltextzugriff (PDF)

The flip side of FLIP

Biochemical journal, 2004-09, Vol.382 (Pt 2), p.e1-e3

Peter, Marcus E

2004

Volltextzugriff (PDF)

Programmed cell death: Apoptosis meets necrosis

Nature (London), 2011-03, Vol.471 (7338), p.310-312

Peter, Marcus E

2011

Volltextzugriff (PDF)

The length of uninterrupted CAG repeats in stem regions of repeat disease associated hairpins determines the amount of short CAG oligonucleotides that are toxic to cells through RNA interference

Cell death & disease, 2022-12, Vol.13 (12), p.1078-12, Article 1078

2022

Volltextzugriff (PDF)

miRConnect: identifying effector genes of miRNAs and miRNA families in cancer cells

PloS one, 2011-10, Vol.6 (10), p.e26521-e26521

2011

Volltextzugriff (PDF)

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Let-7 and miR-200 microRNAs: Guardians against pluripotency and cancer progression

Cancer kill code extension

The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

The role of let-7 in cell differentiation and cancer

Adipocytes promote ovarian cancer metastasis and provide energy for rapid tumor growth

SPOROS: A pipeline to analyze DISE/6mer seed toxicity

Cell type-restricted activity of hnRNPM promotes breast cancer metastasis via regulating alternative splicing

Genomics of Ovarian Cancer Progression Reveals Diverse Metastatic Trajectories Including Intraepithelial Metastasis to the Fallopian Tube

Cancer-Associated IDH1 Promotes Growth and Resistance to Targeted Therapies in the Absence of Mutation

Coordinated epigenetic repression of the miR‐200 family and miR‐205 in invasive bladder cancer

Let-7 modulates acquired resistance of ovarian cancer to Taxanes via IMP-1-mediated stabilization of multidrug resistance 1

Let-7 Expression Defines Two Differentiation Stages of Cancer

6mer seed toxicity in tumor suppressive microRNAs

CD95/Fas Increases Stemness in Cancer Cells by Inducing a STAT1-Dependent Type I Interferon Response

CD95 is part of a let-7/p53/miR-34 regulatory network

DISE/6mer seed toxicity-a powerful anti-cancer mechanism with implications for other diseases

CD95 and CD95L promote and protect cancer stem cells

Identification of the toxic 6mer seed consensus for human cancer cells

Loss of E-Cadherin Promotes Ovarian Cancer Metastasis via α5-Integrin, which Is a Therapeutic Target

Death induced by CD95 or CD95 ligand elimination

Death Induced by Survival gene Elimination (DISE) correlates with neurotoxicity in Alzheimer's disease and aging

The flip side of FLIP

Programmed cell death: Apoptosis meets necrosis

The length of uninterrupted CAG repeats in stem regions of repeat disease associated hairpins determines the amount of short CAG oligonucleotides that are toxic to cells through RNA interference

miRConnect: identifying effector genes of miRNAs and miRNA families in cancer cells

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