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Biology of the cardiac myocyte in heart disease

Molecular biology of the cell, 2016-07, Vol.27 (14), p.2149-2160

2016

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Modulating fast skeletal muscle contraction protects skeletal muscle in animal models of Duchenne muscular dystrophy

The Journal of clinical investigation, 2023-05, Vol.133 (10), p.1-14

2023

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Cardiac Fibroblasts Mediate a Sexually Dimorphic Fibrotic Response to β-Adrenergic Stimulation

Journal of the American Heart Association, 2021-06, Vol.10 (11), p.e018876-e018876

2021

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Inhibition of Coxsackievirus-associated dystrophin cleavage prevents cardiomyopathy

The Journal of clinical investigation, 2013-12, Vol.123 (12), p.5146-5151

2013

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Increased Echogenicity and Radiodense Foci on Echocardiogram and MicroCT in Murine Myocarditis

PloS one, 2016-08, Vol.11 (8), p.e0159971-e0159971

2016

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Loss of Enigma Homolog Protein Results in Dilated Cardiomyopathy

Circulation research, 2010-08, Vol.107 (3), p.348-356

2010

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Sarcospan-dependent Akt activation is required for utrophin expression and muscle regeneration

The Journal of cell biology, 2012-06, Vol.197 (7), p.1009-1027

2012

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Postnatal Loss of Kindlin-2 Leads to Progressive Heart Failure

Circulation. Heart failure, 2016-08, Vol.9 (8)

2016

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The costamere bridges sarcomeres to the sarcolemma in striated muscle

Progress in pediatric cardiology, 2011-05, Vol.31 (2), p.83-88

2011

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Expression of Normally Repressed Myosin Heavy Chain 7b in the Mammalian Heart Induces Dilated Cardiomyopathy

Journal of the American Heart Association, 2019-08, Vol.8 (15), p.e013318-e013318

2019

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Myogenic Akt signaling upregulates the utrophin–glycoprotein complex and promotes sarcolemma stability in muscular dystrophy

Human molecular genetics, 2009-01, Vol.18 (2), p.318-327

2009

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Cypher and Enigma Homolog Protein Are Essential for Cardiac Development and Embryonic Survival

Journal of the American Heart Association, 2015-05, Vol.4 (5), p.n/a

2015

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miR-206 enforces a slow muscle phenotype

Journal of cell science, 2020-08, Vol.133 (15)

2020

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Expanding our scientific horizons: utilization of unique model organisms in biological research

The EMBO journal, 2017-08, Vol.36 (16), p.2311-2314

2017

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Nanospan, an alternatively spliced isoform of sarcospan, localizes to the sarcoplasmic reticulum in skeletal muscle and is absent in limb girdle muscular dystrophy 2F

Skeletal muscle, 2017-06, Vol.7 (1), p.11-17, Article 11

2017

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Kindlin-2 Is Essential for Preserving Integrity of the Developing Heart and Preventing Ventricular Rupture

Circulation (New York, N.Y.), 2019-03, Vol.139 (12), p.1554-1556

2019

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Obscurin is required for ankyrinB-dependent dystrophin localization and sarcolemma integrity

The Journal of cell biology, 2013-02, Vol.200 (4), p.523-536

2013

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Rates of myogenesis and myofiber numbers are reduced in late gestation IUGR fetal sheep

Journal of endocrinology, 2020-02, Vol.244 (2), p.339-352

2020

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Cardiomyocyte Expression of ZO-1 Is Essential for Normal Atrioventricular Conduction but Does Not Alter Ventricular Function

Circulation research, 2020-07, Vol.127 (2), p.284-297

2020

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Burmese pythons exhibit a transient adaptation to nutrient overload that prevents liver damage

The Journal of general physiology, 2022-04, Vol.154 (4)

2022

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Hypertrophic response of Duchenne and limb-girdle muscular dystrophies is associated with activation of Akt pathway

Experimental cell research, 2006-08, Vol.312 (13), p.2580-2591

2006

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Loss of FHL1 induces an age-dependent skeletal muscle myopathy associated with myofibrillar and intermyofibrillar disorganization in mice

Human molecular genetics, 2014-01, Vol.23 (1), p.209-225

2014

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Sarcospan reduces dystrophic pathology: stabilization of the utrophin-glycoprotein complex

The Journal of cell biology, 2008-11, Vol.183 (3), p.419-427

2008

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Selective deletion of long but not short Cypher isoforms leads to late-onset dilated cardiomyopathy

Human molecular genetics, 2011-05, Vol.20 (9), p.1751-1762

2011

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Disrupted mechanical stability of the dystrophin-glycoprotein complex causes severe muscular dystrophy in sarcospan transgenic mice

Journal of cell science, 2007-03, Vol.120 (Pt 6), p.996-1008

2007

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Biology of the cardiac myocyte in heart disease

Modulating fast skeletal muscle contraction protects skeletal muscle in animal models of Duchenne muscular dystrophy

Cardiac Fibroblasts Mediate a Sexually Dimorphic Fibrotic Response to β-Adrenergic Stimulation

Inhibition of Coxsackievirus-associated dystrophin cleavage prevents cardiomyopathy

Increased Echogenicity and Radiodense Foci on Echocardiogram and MicroCT in Murine Myocarditis

Loss of Enigma Homolog Protein Results in Dilated Cardiomyopathy

Sarcospan-dependent Akt activation is required for utrophin expression and muscle regeneration

Postnatal Loss of Kindlin-2 Leads to Progressive Heart Failure

The costamere bridges sarcomeres to the sarcolemma in striated muscle

Expression of Normally Repressed Myosin Heavy Chain 7b in the Mammalian Heart Induces Dilated Cardiomyopathy

Myogenic Akt signaling upregulates the utrophin–glycoprotein complex and promotes sarcolemma stability in muscular dystrophy

Cypher and Enigma Homolog Protein Are Essential for Cardiac Development and Embryonic Survival

miR-206 enforces a slow muscle phenotype

Expanding our scientific horizons: utilization of unique model organisms in biological research

Nanospan, an alternatively spliced isoform of sarcospan, localizes to the sarcoplasmic reticulum in skeletal muscle and is absent in limb girdle muscular dystrophy 2F

Kindlin-2 Is Essential for Preserving Integrity of the Developing Heart and Preventing Ventricular Rupture

Obscurin is required for ankyrinB-dependent dystrophin localization and sarcolemma integrity

Rates of myogenesis and myofiber numbers are reduced in late gestation IUGR fetal sheep

Cardiomyocyte Expression of ZO-1 Is Essential for Normal Atrioventricular Conduction but Does Not Alter Ventricular Function

Burmese pythons exhibit a transient adaptation to nutrient overload that prevents liver damage

Hypertrophic response of Duchenne and limb-girdle muscular dystrophies is associated with activation of Akt pathway

Loss of FHL1 induces an age-dependent skeletal muscle myopathy associated with myofibrillar and intermyofibrillar disorganization in mice

Sarcospan reduces dystrophic pathology: stabilization of the utrophin-glycoprotein complex

Selective deletion of long but not short Cypher isoforms leads to late-onset dilated cardiomyopathy

Disrupted mechanical stability of the dystrophin-glycoprotein complex causes severe muscular dystrophy in sarcospan transgenic mice

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