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Endothelial-specific FoxO1 depletion prevents obesity-related disorders by increasing vascular metabolism and growth

eLife, 2018-12, Vol.7

2018

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Repeated transient mRNA bursts precede increases in transcriptional and mitochondrial proteins during training in human skeletal muscle

The Journal of physiology, 2010-12, Vol.588 (23), p.4795-4810

2010

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Strength athletes and mitochondria: it's about ‘time’

The Journal of physiology, 2023-07, Vol.601 (14), p.2753-2754

2023

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A HIF-1 signature dominates the attenuation in the human skeletal muscle transcriptional response to high-intensity interval training

Journal of applied physiology (1985), 2022-06, Vol.132 (6), p.1448-1459

2022

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Mitochondrial adaptations to exercise in human skeletal muscle: a possible role for cristae density as a determinant of muscle fitness

The Journal of physiology, 2017-05, Vol.595 (9), p.2773-2774

Perry, Christopher G. R.

2017

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Modelling in vivo creatine/phosphocreatine in vitro reveals divergent adaptations in human muscle mitochondrial respiratory control by ADP after acute and chronic exercise

The Journal of physiology, 2016-06, Vol.594 (11), p.3127-3140

2016

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Loss of dystrophin expression in skeletal muscle is associated with senescence of macrophages and endothelial cells

American Journal of Physiology: Cell Physiology, 2021-07, Vol.321 (1), p.C94-C103

2021

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High-intensity aerobic interval training increases fat and carbohydrate metabolic capacities in human skeletal muscle

Applied physiology, nutrition, and metabolism, 2008-12, Vol.33 (6), p.1112

2008

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Altered Skeletal Muscle Microtubule‐Mitochondrial Voltage Gated‐Dependent Anion Channel (VDAC) 2 Binding is Related to Bioenergetic Impairments after Paclitaxel but not Vinblastine Chemotherapies

The FASEB journal, 2019-04, Vol.33 (S1), p.868.7-868.7

2019

Volltextzugriff (PDF)

Mitochondrial Dysfunction and Disorganized Microtubules in Duchenne Muscular Dystrophy are Not Related to Altered α Tubulin‐Voltage Dependent Anion Channel (VDAC) 2 Interactions

The FASEB journal, 2019-04, Vol.33 (S1), p.868.27-868.27

2019

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Alterations in mitochondrial functions and morphology in muscle and non‐muscle tissues in type 1 diabetes: implications for metabolic health

Experimental physiology, 2020-04, Vol.105 (4), p.565-570

2020

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Muscle mitochondrial function is impaired in adults with type 1 diabetes

Journal of diabetes and its complications, 2024-08, Vol.38 (8), p.108798, Article 108798

2024

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Reduced ATGL-mediated lipolysis attenuates β-adrenergic-induced AMPK signaling, but not the induction of PKA-targeted genes, in adipocytes and adipose tissue

American Journal of Physiology: Cell Physiology, 2016-08, Vol.311 (2), p.C269-C276

2016

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Heterozygous SOD2 deletion impairs glucose-stimulated insulin secretion, but not insulin action, in high-fat-fed mice

Diabetes (New York, N.Y.), 2014-11, Vol.63 (11), p.3699-3710

2014

Volltextzugriff (PDF)

p107 Is a Crucial Regulator for Determining the Adipocyte Lineage Fate Choices of Stem Cells

Stem cells (Dayton, Ohio), 2014-05, Vol.32 (5), p.1323-1336

2014

Volltextzugriff (PDF)

Mitochondrial creatine kinase activity and phosphate shuttling are acutely regulated by exercise in human skeletal muscle

The Journal of physiology, 2012-11, Vol.590 (21), p.5475-5486

2012

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p107 Determines a Metabolic Checkpoint Required for Adipocyte Lineage Fates

Stem cells (Dayton, Ohio), 2017-05, Vol.35 (5), p.1378-1391

2017

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Mitohormesis during advanced stages of Duchenne muscular dystrophy reveals a redox-sensitive creatine pathway that can be enhanced by the mitochondrial-targeting peptide SBT-20

Redox biology, 2024-10, Vol.76, p.103319, Article 103319

2024

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High-intensity interval training increases SIRT1 activity in human skeletal muscle

Applied physiology, nutrition, and metabolism, 2010-06, Vol.35 (3), p.350

2010

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SIRT3 gene expression but not SIRT3 subcellular localization is altered in response to fasting and exercise in human skeletal muscle

Experimental physiology, 2016-08, Vol.101 (8), p.1101-1113

2016

Volltextzugriff (PDF)

H 1 /H 2 Histamine Receptor Blockade Alters the Glutathione Redox Status in Skeletal Muscle Following a Bout of Prolonged Exercise

The FASEB journal, 2019-04, Vol.33 (S1)

2019

Volltextzugriff (PDF)

H1/H2 Histamine Receptor Blockade Alters the Glutathione Redox Status in Skeletal Muscle Following a Bout of Prolonged Exercise

The FASEB journal, 2019-04, Vol.33 (S1), p.868.31-868.31

2019

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Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress

JCI insight, 2022-12, Vol.7 (24)

2022

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The impact of a 48-h fast on SIRT1 and GCN5 in human skeletal muscle

Applied physiology, nutrition, and metabolism, 2016-09, Vol.41 (9), p.953

2016

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Reduced ATGL-mediated lipolysis attenuates [Beta]-adrenergic-induced AMPK signaling, but not the induction of PKA-targeted genes, in adipocytes and adipose tissue

American Journal of Physiology: Cell Physiology, 2016-08, Vol.311 (2), p.C269

2016

Volltextzugriff (PDF)

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Endothelial-specific FoxO1 depletion prevents obesity-related disorders by increasing vascular metabolism and growth

Repeated transient mRNA bursts precede increases in transcriptional and mitochondrial proteins during training in human skeletal muscle

Strength athletes and mitochondria: it's about ‘time’

A HIF-1 signature dominates the attenuation in the human skeletal muscle transcriptional response to high-intensity interval training

Mitochondrial adaptations to exercise in human skeletal muscle: a possible role for cristae density as a determinant of muscle fitness

Modelling in vivo creatine/phosphocreatine in vitro reveals divergent adaptations in human muscle mitochondrial respiratory control by ADP after acute and chronic exercise

Loss of dystrophin expression in skeletal muscle is associated with senescence of macrophages and endothelial cells

High-intensity aerobic interval training increases fat and carbohydrate metabolic capacities in human skeletal muscle

Altered Skeletal Muscle Microtubule‐Mitochondrial Voltage Gated‐Dependent Anion Channel (VDAC) 2 Binding is Related to Bioenergetic Impairments after Paclitaxel but not Vinblastine Chemotherapies

Mitochondrial Dysfunction and Disorganized Microtubules in Duchenne Muscular Dystrophy are Not Related to Altered α Tubulin‐Voltage Dependent Anion Channel (VDAC) 2 Interactions

Alterations in mitochondrial functions and morphology in muscle and non‐muscle tissues in type 1 diabetes: implications for metabolic health

Muscle mitochondrial function is impaired in adults with type 1 diabetes

Reduced ATGL-mediated lipolysis attenuates β-adrenergic-induced AMPK signaling, but not the induction of PKA-targeted genes, in adipocytes and adipose tissue

Heterozygous SOD2 deletion impairs glucose-stimulated insulin secretion, but not insulin action, in high-fat-fed mice

p107 Is a Crucial Regulator for Determining the Adipocyte Lineage Fate Choices of Stem Cells

Mitochondrial creatine kinase activity and phosphate shuttling are acutely regulated by exercise in human skeletal muscle

p107 Determines a Metabolic Checkpoint Required for Adipocyte Lineage Fates

Mitohormesis during advanced stages of Duchenne muscular dystrophy reveals a redox-sensitive creatine pathway that can be enhanced by the mitochondrial-targeting peptide SBT-20

High-intensity interval training increases SIRT1 activity in human skeletal muscle

SIRT3 gene expression but not SIRT3 subcellular localization is altered in response to fasting and exercise in human skeletal muscle

H 1 /H 2 Histamine Receptor Blockade Alters the Glutathione Redox Status in Skeletal Muscle Following a Bout of Prolonged Exercise

H1/H2 Histamine Receptor Blockade Alters the Glutathione Redox Status in Skeletal Muscle Following a Bout of Prolonged Exercise

Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress

The impact of a 48-h fast on SIRT1 and GCN5 in human skeletal muscle

Reduced ATGL-mediated lipolysis attenuates [Beta]-adrenergic-induced AMPK signaling, but not the induction of PKA-targeted genes, in adipocytes and adipose tissue

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