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Sirtuins in Renal Health and Disease

Journal of the American Society of Nephrology, 2018-07, Vol.29 (7), p.1799-1809

2018

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Immunity, endothelial injury and complement-induced coagulopathy in COVID-19

Nature reviews. Nephrology, 2021-01, Vol.17 (1), p.46-64

2021

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Manipulating Sirtuin 3 pathway ameliorates renal damage in experimental diabetes

Scientific reports, 2020-05, Vol.10 (1), p.8418-8418, Article 8418

2020

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Human mesenchymal stromal cells transplanted into mice stimulate renal tubular cells and enhance mitochondrial function

Nature communications, 2017-10, Vol.8 (1), p.983-17, Article 983

2017

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Mitochondrial Sirtuin 3 and Renal Diseases

Nephron (2015), 2016-01, Vol.134 (1), p.14-19

2016

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COVID-19: the renaissance of science in the face of adversity

Frontiers in Science, 2024-05, Vol.2

2024

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Sirt3 deficiency promotes endothelial dysfunction and aggravates renal injury

PloS one, 2023-10, Vol.18 (10), p.e0291909-e0291909

2023

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A new BEACON of hope for the treatment of inflammation? The endogenous metabolite itaconate as an alternative activator of the KEAP1-Nrf2 system

Kidney international, 2018-10, Vol.94 (4), p.646-649

2018

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SARS-CoV-2 spike protein induces lung endothelial cell dysfunction and thrombo-inflammation depending on the C3a/C3a receptor signalling

Scientific reports, 2023-07, Vol.13 (1), p.11392-11392, Article 11392

2023

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SARS-CoV-2 Spike Protein 1 Activates Microvascular Endothelial Cells and Complement System Leading to Platelet Aggregation

Frontiers in immunology, 2022-03, Vol.13, p.827146

2022

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Post-translational modifications by SIRT3 de-2-hydroxyisobutyrylase activity regulate glycolysis and enable nephrogenesis

Scientific reports, 2021-12, Vol.11 (1), p.23580-23580, Article 23580

2021

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COVID-19 and lombardy: TESTing the impact of the first wave of the pandemic

EBioMedicine, 2020-11, Vol.61, p.103069-103069, Article 103069

2020

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Long-term adaptive response in COVID-19 vaccine recipients and the effect of a booster dose

Frontiers in immunology, 2023-02, Vol.14, p.1123158-1123158

2023

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Sexual dimorphic response to rituximab treatment: A longitudinal observational study in a large cohort of patients with primary membranous nephropathy and persistent nephrotic syndrome

Frontiers in pharmacology, 2022-09, Vol.13, p.958136-14

2022

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Bi-specific autoantigen-T cell engagers as targeted immunotherapy for autoreactive B cell depletion in autoimmune diseases

Frontiers in immunology, 2024-02, Vol.15, p.1335998-1335998

2024

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Should COVID-19 Concern Nephrologists? Why and to What Extent? The Emerging Impasse of Angiotensin Blockade

Nephron (2015), 2020-05, Vol.144 (5), p.213-221

2020

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BRAF Signaling Pathway Inhibition, Podocyte Injury, and Nephrotic Syndrome

American journal of kidney diseases, 2017-07, Vol.70 (1), p.145-150

2017

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Sirtuin3 Dysfunction Is the Key Determinant of Skeletal Muscle Insulin Resistance by Angiotensin II

PloS one, 2015-05, Vol.10 (5), p.e0127172-e0127172

2015

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C3a receptor blockade protects podocytes from injury in diabetic nephropathy

JCI insight, 2020-03, Vol.5 (5)

2020

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Shiga Toxin Promotes Podocyte Injury in Experimental Hemolytic Uremic Syndrome via Activation of the Alternative Pathway of Complement

Journal of the American Society of Nephrology, 2014-08, Vol.25 (8), p.1786-1798

2014

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Low Nephron Number Induced by Maternal Protein Restriction Is Prevented by Nicotinamide Riboside Supplementation Depending on Sirtuin 3 Activation

Cells (Basel, Switzerland), 2022-10, Vol.11 (20), p.3316

2022

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SARS-CoV-2 and the spike protein in endotheliopathy

Trends in microbiology (Regular ed.), 2024-01, Vol.32 (1), p.53-67

2024

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The iNADequacy of renal cell metabolism: modulating NAD+ biosynthetic pathways to forestall kidney diseases

Kidney international, 2019-08, Vol.96 (2), p.264-267

2019

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Shiga Toxin 2 Triggers C3a-Dependent Glomerular and Tubular Injury through Mitochondrial Dysfunction in Hemolytic Uremic Syndrome

Cells (Basel, Switzerland), 2022-05, Vol.11 (11), p.1755

2022

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Podocyte-actin dynamics in health and disease

Nature reviews. Nephrology, 2016-11, Vol.12 (11), p.692-710

2016

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Sirtuins in Renal Health and Disease

Immunity, endothelial injury and complement-induced coagulopathy in COVID-19

Manipulating Sirtuin 3 pathway ameliorates renal damage in experimental diabetes

Human mesenchymal stromal cells transplanted into mice stimulate renal tubular cells and enhance mitochondrial function

Mitochondrial Sirtuin 3 and Renal Diseases

COVID-19: the renaissance of science in the face of adversity

Sirt3 deficiency promotes endothelial dysfunction and aggravates renal injury

A new BEACON of hope for the treatment of inflammation? The endogenous metabolite itaconate as an alternative activator of the KEAP1-Nrf2 system

SARS-CoV-2 spike protein induces lung endothelial cell dysfunction and thrombo-inflammation depending on the C3a/C3a receptor signalling

SARS-CoV-2 Spike Protein 1 Activates Microvascular Endothelial Cells and Complement System Leading to Platelet Aggregation

Post-translational modifications by SIRT3 de-2-hydroxyisobutyrylase activity regulate glycolysis and enable nephrogenesis

COVID-19 and lombardy: TESTing the impact of the first wave of the pandemic

Long-term adaptive response in COVID-19 vaccine recipients and the effect of a booster dose

Sexual dimorphic response to rituximab treatment: A longitudinal observational study in a large cohort of patients with primary membranous nephropathy and persistent nephrotic syndrome

Bi-specific autoantigen-T cell engagers as targeted immunotherapy for autoreactive B cell depletion in autoimmune diseases

Should COVID-19 Concern Nephrologists? Why and to What Extent? The Emerging Impasse of Angiotensin Blockade

BRAF Signaling Pathway Inhibition, Podocyte Injury, and Nephrotic Syndrome

Sirtuin3 Dysfunction Is the Key Determinant of Skeletal Muscle Insulin Resistance by Angiotensin II

C3a receptor blockade protects podocytes from injury in diabetic nephropathy

Shiga Toxin Promotes Podocyte Injury in Experimental Hemolytic Uremic Syndrome via Activation of the Alternative Pathway of Complement

Low Nephron Number Induced by Maternal Protein Restriction Is Prevented by Nicotinamide Riboside Supplementation Depending on Sirtuin 3 Activation

SARS-CoV-2 and the spike protein in endotheliopathy

The iNADequacy of renal cell metabolism: modulating NAD+ biosynthetic pathways to forestall kidney diseases

Shiga Toxin 2 Triggers C3a-Dependent Glomerular and Tubular Injury through Mitochondrial Dysfunction in Hemolytic Uremic Syndrome

Podocyte-actin dynamics in health and disease

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