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Hypoxia and Neurodegeneration

Annals of the New York Academy of Sciences, 2009-10, Vol.1177 (1), p.169-177

2009

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Physiological roles for amyloid β peptides

The Journal of physiology, 2006-08, Vol.575 (1), p.5-10

2006

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Robotic multiwell planar patch-clamp for native and primary mammalian cells

Nature protocols, 2009-02, Vol.4 (2), p.244-255

2009

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α-Synuclein modulation of Ca²⁺ signaling in human neuroblastoma (SH-SY5Y) cells

Journal of neurochemistry, 2009-12, Vol.111 (5), p.1192-1201

2009

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Hypoxia Suppresses Glutamate Transport in Astrocytes

The Journal of neuroscience, 2007-04, Vol.27 (15), p.3946-3955

2007

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Carbon monoxide protects against oxidant‐induced apoptosis via inhibition of Kv2.1

The FASEB journal, 2011-05, Vol.25 (5), p.1519-1530

2011

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The production of amyloid beta peptide is a critical requirement for the viability of central neurons

The Journal of neuroscience, 2003-07, Vol.23 (13), p.5531-5535

2003

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A central role for ROS in the functional remodelling of L-type Ca2+ channels by hypoxia

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2005-12, Vol.360 (1464), p.2247

2005

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Amyloid β peptide as a physiological modulator of neuronal ‘A’-type K + current

Neurobiology of aging, 2006-11, Vol.27 (11), p.1673-1683

2006

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Hypoxic Remodeling of Ca2+ Stores in Type I Cortical Astrocytes

The Journal of biological chemistry, 2003-02, Vol.278 (7), p.4875-4881

2003

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Hypoxia and Alzheimer's disease

Essays in biochemistry, 2007-01, Vol.43, p.153

2007

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Differential involvement of TNFα in hypoxic suppression of astrocyte glutamate transporters

Glia, 2008-07, Vol.56 (9), p.998-1004

2008

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Modulation of ‘A’-type K+ current by rodent and human forms of amyloid β protein

Neuroreport, 2008-05, Vol.19 (8), p.839-843

2008

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Peroxynitrite mediates disruption of Ca2+ homeostasis by carbon monoxide via Ca2+ ATPase degradation

Antioxidants & redox signaling, 2012-09, Vol.17 (5), p.744

2012

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Calcium channel subtypes – another layer of complexity to an already intricate story

The Journal of physiology, 2007-02, Vol.578 (3), p.629-629

Pearson, Hugh A.

2007

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Cellular consequences of the expression of Alzheimer's disease-causing presenilin 1 mutations in human neuroblastoma (SH-SY5Y) cells

Brain research, 2012-03, Vol.1443, p.75-88

2012

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Hypoxia suppresses astrocyte glutamate transport independently of amyloid formation

Biochemical and biophysical research communications, 2007-12, Vol.364 (1), p.100-104

2007

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Hypoxic depolarization of cerebellar granule neurons by specific inhibition of TASK-1

Stroke (1970), 2002-09, Vol.33 (9), p.2324-2328

2002

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Gap junction-mediated spontaneous Ca2+ waves in differentiated cholinergic SN56 cells

Biochemical and biophysical research communications, 2010-07, Vol.397 (3), p.564-568

2010

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Gap junction-mediated spontaneous Ca(2+) waves in differentiated cholinergic SN56 cells

Biochemical and biophysical research communications, 2010-07, Vol.397 (3), p.564-568

2010

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Differential effects of unaggregated and aggregated amyloid β protein (1–40) on K+ channel currents in primary cultures of rat cerebellar granule and cortical neurones

Journal of neurochemistry, 2001-11, Vol.79 (3), p.699-712

2001

Volltextzugriff (PDF)

Hypoxia stimulates Ca2+ release from intracellular stores in astrocytes via cyclic ADP ribose-mediated activation of ryanodine receptors

Cell calcium (Edinburgh), 2006-01, Vol.39 (1), p.95-100

2006

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Chronic hypoxia inhibits Na+/Ca2+ exchanger expression in cortical astrocytes

Neuroreport, 2006-04, Vol.17 (6), p.649-652

2006

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Hypoxic regulation of Ca2+ signalling in astrocytes and endothelial cells

Advances in experimental medicine and biology, 2006, Vol.580, p.185

2006

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Presenilin-1 mutations alter K+ currents in the human neuroblastoma cell line, SH-SY5Y

Neuroreport, 2002-08, Vol.13 (12), p.1553-1556

2002

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Hypoxia and Neurodegeneration

Physiological roles for amyloid β peptides

Robotic multiwell planar patch-clamp for native and primary mammalian cells

α-Synuclein modulation of Ca²⁺ signaling in human neuroblastoma (SH-SY5Y) cells

Hypoxia Suppresses Glutamate Transport in Astrocytes

Carbon monoxide protects against oxidant‐induced apoptosis via inhibition of Kv2.1

The production of amyloid beta peptide is a critical requirement for the viability of central neurons

A central role for ROS in the functional remodelling of L-type Ca2+ channels by hypoxia

Amyloid β peptide as a physiological modulator of neuronal ‘A’-type K + current

Hypoxic Remodeling of Ca2+ Stores in Type I Cortical Astrocytes

Hypoxia and Alzheimer's disease

Differential involvement of TNFα in hypoxic suppression of astrocyte glutamate transporters

Modulation of ‘A’-type K+ current by rodent and human forms of amyloid β protein

Peroxynitrite mediates disruption of Ca2+ homeostasis by carbon monoxide via Ca2+ ATPase degradation

Calcium channel subtypes – another layer of complexity to an already intricate story

Cellular consequences of the expression of Alzheimer's disease-causing presenilin 1 mutations in human neuroblastoma (SH-SY5Y) cells

Hypoxia suppresses astrocyte glutamate transport independently of amyloid formation

Hypoxic depolarization of cerebellar granule neurons by specific inhibition of TASK-1

Gap junction-mediated spontaneous Ca2+ waves in differentiated cholinergic SN56 cells

Gap junction-mediated spontaneous Ca(2+) waves in differentiated cholinergic SN56 cells

Differential effects of unaggregated and aggregated amyloid β protein (1–40) on K+ channel currents in primary cultures of rat cerebellar granule and cortical neurones

Hypoxia stimulates Ca2+ release from intracellular stores in astrocytes via cyclic ADP ribose-mediated activation of ryanodine receptors

Chronic hypoxia inhibits Na+/Ca2+ exchanger expression in cortical astrocytes

Hypoxic regulation of Ca2+ signalling in astrocytes and endothelial cells

Presenilin-1 mutations alter K+ currents in the human neuroblastoma cell line, SH-SY5Y

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