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Evolution of Barrett's esophagus through space and time at single-crypt and whole-biopsy levels

Nature communications, 2018-02, Vol.9 (1), p.794-12, Article 794

2018

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Temporal and Spatial Evolution of Somatic Chromosomal Alterations: A Case-Cohort Study of Barrett's Esophagus

Cancer prevention research (Philadelphia, Pa.), 2014-01, Vol.7 (1), p.114-127

2014

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Somatic whole genome dynamics of precancer in Barrett's esophagus reveals features associated with disease progression

Nature communications, 2022-04, Vol.13 (1), p.2300-2300, Article 2300

2022

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NSAIDs modulate clonal evolution in Barrett's esophagus

PLoS genetics, 2013-06, Vol.9 (6), p.e1003553

2013

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Chromosomal Instability and Copy Number Alterations in Barrett's Esophagus and Esophageal Adenocarcinoma

Clinical cancer research, 2009-05, Vol.15 (10), p.3305-3314

2009

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Within‐patient phylogenetic reconstruction reveals early events in Barrett’s Esophagus

Evolutionary applications, 2021-02, Vol.14 (2), p.399-415

2021

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Warburg and Crabtree effects in premalignant Barrett's esophagus cell lines with active mitochondria

PloS one, 2013-02, Vol.8 (2), p.e56884

2013

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NSAID use and somatic exomic mutations in Barrett's esophagus

Genome medicine, 2018-02, Vol.10 (1), p.17-17, Article 17

2018

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Focus on Barrett's esophagus and esophageal adenocarcinoma

Cancer cell, 2004-07, Vol.6 (1), p.11-16

2004

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The combination of genetic instability and clonal expansion predicts progression to esophageal adenocarcinoma

Cancer research (Chicago, Ill.), 2004-10, Vol.64 (20), p.7629-7633

2004

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Correction to: NSAID use and somatic exomic mutations in Barrett's esophagus

Genome medicine, 2019-03, Vol.11 (1), p.14-14, Article 14

2019

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p16 mutation spectrum in the premalignant condition Barrett's esophagus

PloS one, 2008-11, Vol.3 (11), p.e3809-e3809

2008

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Genetic Insights in Barrett’s Esophagus and Esophageal Adenocarcinoma

Gastroenterology (New York, N.Y. 1943), 2015-10, Vol.149 (5), p.1142-1152.e3

2015

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Cell Proliferation, Cell Cycle Abnormalities, and Cancer Outcome in Patients with Barrett's Esophagus: A Long-term Prospective Study

Clinical cancer research, 2008-11, Vol.14 (21), p.6988-6995

2008

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Extrachromosomal DNA in the cancerous transformation of Barrett's oesophagus

Nature (London), 2023-04, Vol.616 (7958), p.798-805

2023

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Significance of Crypt Atypia in Barrett’s Esophagus: A Clinical, Molecular, and Outcome Study

Clinical gastroenterology and hepatology, 2024-04, Vol.22 (4), p.741-748.e2

2024

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Studying Cancer Evolution in Barrett’s Esophagus and Esophageal Adenocarcinoma

Advances in experimental medicine and biology, 2016-01, Vol.908, p.213-236

2016

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Method for physiologic phenotype characterization at the single-cell level in non-interacting and interacting cells

Journal of biomedical optics, 2012-03, Vol.17 (3), p.037008-1-037008-24

2012

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Genetic clonal diversity predicts progression to esophageal adenocarcinoma

Nature genetics, 2006-04, Vol.38 (4), p.468-473

2006

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Neosquamous Epithelium Does Not Typically Arise from Barrett's Epithelium

Clinical cancer research, 2006-03, Vol.12 (6), p.1701-1706

2006

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Assessment of Esophageal Adenocarcinoma Risk Using Somatic Chromosome Alterations in Longitudinal Samples in Barrett's Esophagus

Cancer prevention research (Philadelphia, Pa.), 2015-09, Vol.8 (9), p.845-856

2015

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Direct inference of SNP heterozygosity rates and resolution of LOH detection

PLoS computational biology, 2007-11, Vol.3 (11), p.e244-e244

2007

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Mutagen Sensitivity and Neoplastic Progression in Patients with Barrett's Esophagus: A Prospective Analysis

Cancer epidemiology, biomarkers & prevention, 2006-10, Vol.15 (10), p.1935-1940

2006

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Selectively advantageous mutations and hitchhikers in neoplasms: P16 lesions are selected in Barrett's esophagus

Cancer research (Chicago, Ill.), 2004-05, Vol.64 (10), p.3414-3427

2004

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QUANTIFICATION OF MULTIPLE TUMOR CLONES USING GENE ARRAY AND SEQUENCING DATA

The annals of applied statistics, 2017-06, Vol.11 (2), p.967-991

2017

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Evolution of Barrett's esophagus through space and time at single-crypt and whole-biopsy levels

Temporal and Spatial Evolution of Somatic Chromosomal Alterations: A Case-Cohort Study of Barrett's Esophagus

Somatic whole genome dynamics of precancer in Barrett's esophagus reveals features associated with disease progression

NSAIDs modulate clonal evolution in Barrett's esophagus

Chromosomal Instability and Copy Number Alterations in Barrett's Esophagus and Esophageal Adenocarcinoma

Within‐patient phylogenetic reconstruction reveals early events in Barrett’s Esophagus

Warburg and Crabtree effects in premalignant Barrett's esophagus cell lines with active mitochondria

NSAID use and somatic exomic mutations in Barrett's esophagus

Focus on Barrett's esophagus and esophageal adenocarcinoma

The combination of genetic instability and clonal expansion predicts progression to esophageal adenocarcinoma

Correction to: NSAID use and somatic exomic mutations in Barrett's esophagus

p16 mutation spectrum in the premalignant condition Barrett's esophagus

Genetic Insights in Barrett’s Esophagus and Esophageal Adenocarcinoma

Cell Proliferation, Cell Cycle Abnormalities, and Cancer Outcome in Patients with Barrett's Esophagus: A Long-term Prospective Study

Extrachromosomal DNA in the cancerous transformation of Barrett's oesophagus

Significance of Crypt Atypia in Barrett’s Esophagus: A Clinical, Molecular, and Outcome Study

Studying Cancer Evolution in Barrett’s Esophagus and Esophageal Adenocarcinoma

Method for physiologic phenotype characterization at the single-cell level in non-interacting and interacting cells

Genetic clonal diversity predicts progression to esophageal adenocarcinoma

Neosquamous Epithelium Does Not Typically Arise from Barrett's Epithelium

Assessment of Esophageal Adenocarcinoma Risk Using Somatic Chromosome Alterations in Longitudinal Samples in Barrett's Esophagus

Direct inference of SNP heterozygosity rates and resolution of LOH detection

Mutagen Sensitivity and Neoplastic Progression in Patients with Barrett's Esophagus: A Prospective Analysis

Selectively advantageous mutations and hitchhikers in neoplasms: P16 lesions are selected in Barrett's esophagus

QUANTIFICATION OF MULTIPLE TUMOR CLONES USING GENE ARRAY AND SEQUENCING DATA

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