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The ClC-K2 Chloride Channel Is Critical for Salt Handling in the Distal Nephron

Journal of the American Society of Nephrology, 2017-01, Vol.28 (1), p.209-217

2017

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Piezo1-dependent stretch-activated channels are inhibited by Polycystin-2 in renal tubular epithelial cells

EMBO reports, 2013-12, Vol.14 (12), p.1143-1148

2013

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Renal phenotype in mice lacking the Kir5.1 (Kcnj16) K+channel subunit contrasts with that observed in SeSAME/EAST syndrome

Proceedings of the National Academy of Sciences - PNAS, 2011-06, Vol.108 (25), p.10361-10366

2011

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Kir4.1/Kir5.1 channel forms the major K+ channel in the basolateral membrane of mouse renal collecting duct principal cells

American journal of physiology. Renal physiology, 2008-06, Vol.294 (6), p.F1398-F1407

2008

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A calcium-permeable non-selective cation channel in the thick ascending limb apical membrane of the mouse kidney

Biochimica et biophysica acta, 2012-05, Vol.1818 (5), p.1135-1141

2012

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A Na+- and Cl- -activated K+ channel in the thick ascending limb of mouse kidney

The Journal of general physiology, 2006-02, Vol.127 (2), p.205-215

2006

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An inward rectifier K+ channel at the basolateral membrane of the mouse distal convoluted tubule: similarities with Kir4-Kir5.1 heteromeric channels

The Journal of physiology, 2002-01, Vol.538 (2), p.391-404

2002

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Inhibitors of vacuolar H+-ATPase impair the preferential accumulation of daunomycin in lysosomes and reverse the resistance to anthracyclines in drug-resistant renal epithelial cells

Biochemical journal, 2003-02, Vol.370 (Pt 1), p.185-193

2003

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The apical 70-pS K + channel in the thick ascending limb of Henle’s loop is a large-conductance Na + -and Cl – -activated, K Na 1.1-like, channel

American journal of physiology. Renal physiology, 2024-03, Vol.326 (3), p.F485-F496

2024

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Defective bicarbonate reabsorption in Kir4.2 potassium channel deficient mice impairs acid-base balance and ammonia excretion

Kidney international, 2020-02, Vol.97 (2), p.304-315

2020

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Properties of an inwardly rectifying K(+) channel in the basolateral membrane of mouse TAL

American journal of physiology. Renal physiology, 2002-05, Vol.282 (5), p.F866-F876

2002

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Dual regulation of the native ClC-K2 chloride channel in the distal nephron by voltage and pH

The Journal of general physiology, 2016-09, Vol.148 (3), p.213-226

2016

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Defective bicarbonate reabsorption in Kir4.2 potassium channel deficient mice impairs acid-base balance and ammonia excretion: Kir4.2 and renal ammoniagenesis

Kidney international, 2020-02, Vol.97 (2), p.304-315

2020

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Analysis of CLCNKB mutations at dimer‐interface, calcium‐binding site, and pore reveals a variety of functional alterations in ClC‐Kb channel leading to Bartter syndrome

Human mutation, 2020-04, Vol.41 (4), p.774-785

2020

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Neutrophil Elastase Degrades Cystic Fibrosis Transmembrane Conductance Regulator via Calpains and Disables Channel Function In Vitro and In Vivo

American journal of respiratory and critical care medicine, 2013-01, Vol.187 (2), p.170-179

2013

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Phosphatidylinositol (4,5)-bisphosphate dynamically regulates the K2P background K+ channel TASK-2

Scientific reports, 2017-03, Vol.7 (1), p.45407, Article 45407

2017

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Characterization of the mouse ClC-K1/Barttin chloride channel

Biochimica et biophysica acta, 2013-11, Vol.1828 (11), p.2399-2409

2013

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Inhibition of the Calcium Release-activated Calcium (CRAC) Current in Jurkat T Cells by the HIV-1 Envelope Protein gp160

The Journal of biological chemistry, 2002-02, Vol.277 (8), p.6044-6050

2002

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Extracellular ATP raises cytosolic calcium and activates basolateral chloride conductance in Necturus proximal tubule

The Journal of physiology, 1998-07, Vol.510 (2), p.535-548

1998

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Similar chloride channels in the connecting tubule and cortical collecting duct of the mouse kidney

American journal of physiology. Renal physiology, 2006-06, Vol.290 (6), p.F1421-F1429

2006

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Exploration of the Basolateral Chloride Channels in the Renal Tubule using

Nephron Physiology, 2005-02, Vol.99 (2), p.p64-p68

2005

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Hyperaldosteronemia and activation of the epithelial sodium channel are not required for sodium retention in puromycin-induced nephrosis

Journal of the American Society of Nephrology, 2005-12, Vol.16 (12), p.3642-3650

2005

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Phosphatidylinositol (4,5)-bisphosphate dynamically regulates the K 2P background K + channel TASK-2

Scientific reports, 2017-03, Vol.7, p.45407

2017

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A chloride channel at the basolateral membrane of the distal-convoluted tubule: a candidate ClC-K channel

The Journal of general physiology, 2003-04, Vol.121 (4), p.287-300

2003

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Neutrophil Elastase Degrades Cystic Fibrosis Transmembrane Conductance Regulator via Calpains and Disables Channel Function In Vitro and In Vivo

American journal of respiratory and critical care medicine, 2013-01, Vol.187 (2), p.170-179

2013

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The ClC-K2 Chloride Channel Is Critical for Salt Handling in the Distal Nephron

Piezo1-dependent stretch-activated channels are inhibited by Polycystin-2 in renal tubular epithelial cells

Renal phenotype in mice lacking the Kir5.1 (Kcnj16) K+channel subunit contrasts with that observed in SeSAME/EAST syndrome

Kir4.1/Kir5.1 channel forms the major K+ channel in the basolateral membrane of mouse renal collecting duct principal cells

A calcium-permeable non-selective cation channel in the thick ascending limb apical membrane of the mouse kidney

A Na+- and Cl- -activated K+ channel in the thick ascending limb of mouse kidney

An inward rectifier K+ channel at the basolateral membrane of the mouse distal convoluted tubule: similarities with Kir4-Kir5.1 heteromeric channels

Inhibitors of vacuolar H+-ATPase impair the preferential accumulation of daunomycin in lysosomes and reverse the resistance to anthracyclines in drug-resistant renal epithelial cells

The apical 70-pS K + channel in the thick ascending limb of Henle’s loop is a large-conductance Na + -and Cl – -activated, K Na 1.1-like, channel

Defective bicarbonate reabsorption in Kir4.2 potassium channel deficient mice impairs acid-base balance and ammonia excretion

Properties of an inwardly rectifying K(+) channel in the basolateral membrane of mouse TAL

Dual regulation of the native ClC-K2 chloride channel in the distal nephron by voltage and pH

Defective bicarbonate reabsorption in Kir4.2 potassium channel deficient mice impairs acid-base balance and ammonia excretion: Kir4.2 and renal ammoniagenesis

Analysis of CLCNKB mutations at dimer‐interface, calcium‐binding site, and pore reveals a variety of functional alterations in ClC‐Kb channel leading to Bartter syndrome

Neutrophil Elastase Degrades Cystic Fibrosis Transmembrane Conductance Regulator via Calpains and Disables Channel Function In Vitro and In Vivo

Phosphatidylinositol (4,5)-bisphosphate dynamically regulates the K2P background K+ channel TASK-2

Characterization of the mouse ClC-K1/Barttin chloride channel

Inhibition of the Calcium Release-activated Calcium (CRAC) Current in Jurkat T Cells by the HIV-1 Envelope Protein gp160

Extracellular ATP raises cytosolic calcium and activates basolateral chloride conductance in Necturus proximal tubule

Similar chloride channels in the connecting tubule and cortical collecting duct of the mouse kidney

Exploration of the Basolateral Chloride Channels in the Renal Tubule using

Hyperaldosteronemia and activation of the epithelial sodium channel are not required for sodium retention in puromycin-induced nephrosis

Phosphatidylinositol (4,5)-bisphosphate dynamically regulates the K 2P background K + channel TASK-2

A chloride channel at the basolateral membrane of the distal-convoluted tubule: a candidate ClC-K channel

Neutrophil Elastase Degrades Cystic Fibrosis Transmembrane Conductance Regulator via Calpains and Disables Channel Function In Vitro and In Vivo

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