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Dynamic sorting of nuclear components into distinct nucleolar caps during transcriptional inhibition

Molecular biology of the cell, 2005-05, Vol.16 (5), p.2395-2413

2005

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siRNA therapeutics: big potential from small RNAs

Gene therapy, 2005-01, Vol.12 (1), p.5-11

2005

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Multiple RRMs Contribute to RNA Binding Specificity and Affinity for Polypyrimidine Tract Binding Protein

Biochemistry (Easton), 1997-09, Vol.36 (39), p.11881-11890

1997

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An RNA Recognition Motif (RRM) Is Required for the Localization of PTB-Associated Splicing Factor (PSF) to Subnuclear Speckles

Experimental cell research, 2001-02, Vol.263 (1), p.131-144

2001

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GH1 Splicing Is Regulated by Multiple Enhancers Whose Mutation Produces a Dominant-Negative GH Isoform That Can Be Degraded by Allele-Specific Small Interfering RNA (siRNA)

Endocrinology (Philadelphia), 2004-06, Vol.145 (6), p.2988-2996

2004

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Mutation of PTB binding sites causes misregulation of alternative 3' splice site selection in vivo

RNA (Cambridge), 1997-07, Vol.3 (7), p.764-778

1997

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Cloning and domain structure of the mammalian splicing factor U2AF

Nature (London), 1992-02, Vol.355 (6361), p.609-614

1992

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Regulation of alternative 3' splice site selection by constitutive splicing factors

RNA (Cambridge), 1995-05, Vol.1 (3), p.234-245

1995

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Direct evidence that polypyrimidine tract binding protein (PTB) is essential for internal initiation of translation of encephalomyocarditis virus RNA

RNA (Cambridge), 1995-11, Vol.1 (9), p.924-938

1995

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Isolated growth hormone deficiency type II caused by a point mutation that alters both splice site strength and splicing enhancer function

Clinical genetics, 2008-12, Vol.74 (6), p.539-545

2008

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Functional analysis of the polypyrimidine tract in pre-mRNA splicing

Nucleic acids research, 1997-02, Vol.25 (4), p.888-896

1997

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Disruption of exon definition produces a dominant-negative growth hormone isoform that causes somatotroph death and IGHD II

Human genetics, 2003-07, Vol.113 (2), p.140-148

2003

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Regulation of Alternative Splicing by SRrp86 and Its Interacting Proteins

Molecular and Cellular Biology, 2003-11, Vol.23 (21), p.7437-7447

2003

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Nuclear relocalization of the pre-mRNA splicing factor PSF during apoptosis involves hyperphosphorylation, masking of antigenic epitopes, and changes in protein interactions

Molecular biology of the cell, 2001-08, Vol.12 (8), p.2328-2340

2001

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Alternative Splicing in the Control of Gene Expression

Annual review of genetics, 1989-01, Vol.23 (1), p.527-577

1989

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Identification and Characterization of a Novel Serine-Arginine-Rich Splicing Regulatory Protein

Molecular and Cellular Biology, 2000-05, Vol.20 (9), p.3049-3057

2000

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Regulation of alternative splicing by SRrp86 through coactivation and repression of specific SR proteins

RNA (Cambridge), 2002-04, Vol.8 (4), p.526-533, Article S1355838202029928

2002

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A unique glutamic acid-lysine (EK) domain acts as a splicing inhibitor

The Journal of biological chemistry, 2002-10, Vol.277 (42), p.39485-39492

2002

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Scanning from an independently specified branch point defines the 3′ splice site of mammalian introns

Nature (London), 1989-11, Vol.342 (6247), p.243-247

1989

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The involvement of a spliceosome component in internal initiation of human rhinovirus RNA translation

Journal of general virology, 1993-09, Vol.74 (9), p.1775-1788

1993

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A new double-stranded RNA-binding protein that interacts with PKR

Nucleic acids research, 2000-03, Vol.28 (6), p.1407-1417

2000

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α-Tropomyosin mutually exclusive exon selection : competition between branchpoint/polypyrimidine tracts determines default exon choice

Genes & development, 1991-04, Vol.5 (4), p.642-655

1991

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Functional crosstalk between exon enhancers, polypyrimidine tracts and branchpoint sequences

The EMBO journal, 1997-12, Vol.16 (23), p.7174-7183

1997

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Identification of Psf, the Polypyrimidine Tract-Binding Protein-Associated Splicing Factor, as a Developmentally Regulated Neuronal Protein

Journal of neuroscience research, 1999-07, Vol.57 (1), p.62-73

1999

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Enhancer elements activate the weak 3′ splice site of α-tropomyosin exon 2

RNA (Cambridge), 1998-12, Vol.4 (12), p.1523-1536, Article S1355838298980360

1998

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Dynamic sorting of nuclear components into distinct nucleolar caps during transcriptional inhibition

siRNA therapeutics: big potential from small RNAs

Multiple RRMs Contribute to RNA Binding Specificity and Affinity for Polypyrimidine Tract Binding Protein

An RNA Recognition Motif (RRM) Is Required for the Localization of PTB-Associated Splicing Factor (PSF) to Subnuclear Speckles

GH1 Splicing Is Regulated by Multiple Enhancers Whose Mutation Produces a Dominant-Negative GH Isoform That Can Be Degraded by Allele-Specific Small Interfering RNA (siRNA)

Mutation of PTB binding sites causes misregulation of alternative 3' splice site selection in vivo

Cloning and domain structure of the mammalian splicing factor U2AF

Regulation of alternative 3' splice site selection by constitutive splicing factors

Direct evidence that polypyrimidine tract binding protein (PTB) is essential for internal initiation of translation of encephalomyocarditis virus RNA

Isolated growth hormone deficiency type II caused by a point mutation that alters both splice site strength and splicing enhancer function

Functional analysis of the polypyrimidine tract in pre-mRNA splicing

Disruption of exon definition produces a dominant-negative growth hormone isoform that causes somatotroph death and IGHD II

Regulation of Alternative Splicing by SRrp86 and Its Interacting Proteins

Nuclear relocalization of the pre-mRNA splicing factor PSF during apoptosis involves hyperphosphorylation, masking of antigenic epitopes, and changes in protein interactions

Alternative Splicing in the Control of Gene Expression

Identification and Characterization of a Novel Serine-Arginine-Rich Splicing Regulatory Protein

Regulation of alternative splicing by SRrp86 through coactivation and repression of specific SR proteins

A unique glutamic acid-lysine (EK) domain acts as a splicing inhibitor

Scanning from an independently specified branch point defines the 3′ splice site of mammalian introns

The involvement of a spliceosome component in internal initiation of human rhinovirus RNA translation

A new double-stranded RNA-binding protein that interacts with PKR

α-Tropomyosin mutually exclusive exon selection : competition between branchpoint/polypyrimidine tracts determines default exon choice

Functional crosstalk between exon enhancers, polypyrimidine tracts and branchpoint sequences

Identification of Psf, the Polypyrimidine Tract-Binding Protein-Associated Splicing Factor, as a Developmentally Regulated Neuronal Protein

Enhancer elements activate the weak 3′ splice site of α-tropomyosin exon 2

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