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Activation of PKA leads to mesenchymal-to-epithelial transition and loss of tumor-initiating ability

Science (American Association for the Advancement of Science), 2016-03, Vol.351 (6277), p.1042-1042

2016

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Integrin-β4 identifies cancer stem cell-enriched populations of partially mesenchymal carcinoma cells

Proceedings of the National Academy of Sciences - PNAS, 2017-03, Vol.114 (12), p.E2337-E2346

2017

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The systemic response to surgery triggers the outgrowth of distant immune-controlled tumors in mouse models of dormancy

Science translational medicine, 2018-04, Vol.10 (436)

2018

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Endothelial E-selectin inhibition improves acute myeloid leukaemia therapy by disrupting vascular niche-mediated chemoresistance

Nature communications, 2020-04, Vol.11 (1), p.2042-2042, Article 2042

2020

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Targeting the Epithelial-to-Mesenchymal Transition: The Case for Differentiation-Based Therapy

Cold Spring Harbor Symposia on Quantitative Biology, 2016, Vol.81, p.11-19

2016

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miR-139-5p is a regulator of metastatic pathways in breast cancer

RNA (Cambridge), 2013-12, Vol.19 (12), p.1767-1780

2013

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Quantifying the Epithelial-to-Mesenchymal Transition (EMT) from Bench to Bedside

Cancers, 2022-02, Vol.14 (5), p.1138

2022

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Lineage plasticity enables low-ER luminal tumors to evolve and gain basal-like traits

Breast cancer research : BCR, 2023-03, Vol.25 (1), p.23-23, Article 23

2023

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Emerging Biological Principles of Metastasis

Cell, 2017-02, Vol.168 (4), p.670-691

2017

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Tackling the cancer stem cells - what challenges do they pose?

Nature reviews. Drug discovery, 2014-07, Vol.13 (7), p.497-512

2014

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Phenotypic heterogeneity driven by plasticity of the intermediate EMT state governs disease progression and metastasis in breast cancer

Science advances, 2022-08, Vol.8 (31), p.eabj8002-eabj8002

2022

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Quantifying epithelial-mesenchymal heterogeneity and EMT scoring in tumor samples via tyramide signal amplification (TSA)

Methods in cell biology, 2022, Vol.171, p.149-161

2022

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Mutations in Multiple Domains of c-Myb Disrupt Interaction with CBP/p300 and Abrogate Myeloid Transforming Ability

Molecular cancer research, 2009-09, Vol.7 (9), p.1477-1486

2009

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Limiting Self-Renewal of the Basal Compartment by PKA Activation Induces Differentiation and Alters the Evolution of Mammary Tumors

Developmental cell, 2020-12, Vol.55 (5), p.544-557.e6

2020

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Interaction of c-Myb with p300 is required for the induction of acute myeloid leukemia (AML) by human AML oncogenes

Blood, 2014-04, Vol.123 (17), p.2682-2690

2014

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Distinct cytosine modification profiles define epithelial-to-mesenchymal cell-state transitions

Epigenomics, 2022-05, Vol.14 (9), p.519-535

2022

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Alteration of DNA methyltransferases by eribulin elicits broad DNA methylation changes with potential therapeutic implications for triple-negative breast cancer

Epigenomics, 2024-03, Vol.16 (5), p.293-308

2024

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Integrated genome-wide chromatin occupancy and expression analyses identify key myeloid pro-differentiation transcription factors repressed by Myb

Nucleic acids research, 2011-06, Vol.39 (11), p.4664-4679

2011

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Pharmacological induction of chromatin remodeling drives chemosensitization in triple-negative breast cancer

Cell reports. Medicine, 2024-04, Vol.5 (4), p.101504-101504, Article 101504

2024

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A recessive screen for genes regulating hematopoietic stem cells

Blood, 2010-12, Vol.116 (26), p.5849-5858

2010

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Abstract 6430: Redefining therapy regimens for triple negative breast cancer - Exploiting the epigenetic effects of eribulin action

Cancer research (Chicago, Ill.), 2020-08, Vol.80 (16_Supplement), p.6430-6430

2020

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Abstract P4-07-19: Quantifying epithelial-mesenchymal tumor heterogeneity for prediction of patient prognosis based on EMT state

Cancer research (Chicago, Ill.), 2022-02, Vol.82 (4_Supplement), p.P4-P4-07-19

2022

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Abstract P3-06-01: Eribulin alters the chromatin landscape to induce MET, attenuating metastatic progression and sensitizing breast tumors to subsequent chemotherapy

Cancer research (Chicago, Ill.), 2022-02, Vol.82 (4_Supplement), p.P3-P3-06-01

2022

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Abstract 2115: Multicomponent epigenetic profiling of intermediate epithelial-to-mesenchymal states in triple negative breast cancer identifies distinct regulatory signatures

Cancer research (Chicago, Ill.), 2021-07, Vol.81 (13_Supplement), p.2115-2115

2021

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Abstract 1417: Curtailing epithelial-mesenchymal plasticity for improved response to breast cancer therapy

Cancer research (Chicago, Ill.), 2021-07, Vol.81 (13_Supplement), p.1417-1417

2021

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Activation of PKA leads to mesenchymal-to-epithelial transition and loss of tumor-initiating ability

Integrin-β4 identifies cancer stem cell-enriched populations of partially mesenchymal carcinoma cells

The systemic response to surgery triggers the outgrowth of distant immune-controlled tumors in mouse models of dormancy

Endothelial E-selectin inhibition improves acute myeloid leukaemia therapy by disrupting vascular niche-mediated chemoresistance

Targeting the Epithelial-to-Mesenchymal Transition: The Case for Differentiation-Based Therapy

miR-139-5p is a regulator of metastatic pathways in breast cancer

Quantifying the Epithelial-to-Mesenchymal Transition (EMT) from Bench to Bedside

Lineage plasticity enables low-ER luminal tumors to evolve and gain basal-like traits

Emerging Biological Principles of Metastasis

Tackling the cancer stem cells - what challenges do they pose?

Phenotypic heterogeneity driven by plasticity of the intermediate EMT state governs disease progression and metastasis in breast cancer

Quantifying epithelial-mesenchymal heterogeneity and EMT scoring in tumor samples via tyramide signal amplification (TSA)

Mutations in Multiple Domains of c-Myb Disrupt Interaction with CBP/p300 and Abrogate Myeloid Transforming Ability

Limiting Self-Renewal of the Basal Compartment by PKA Activation Induces Differentiation and Alters the Evolution of Mammary Tumors

Interaction of c-Myb with p300 is required for the induction of acute myeloid leukemia (AML) by human AML oncogenes

Distinct cytosine modification profiles define epithelial-to-mesenchymal cell-state transitions

Alteration of DNA methyltransferases by eribulin elicits broad DNA methylation changes with potential therapeutic implications for triple-negative breast cancer

Integrated genome-wide chromatin occupancy and expression analyses identify key myeloid pro-differentiation transcription factors repressed by Myb

Pharmacological induction of chromatin remodeling drives chemosensitization in triple-negative breast cancer

A recessive screen for genes regulating hematopoietic stem cells

Abstract 6430: Redefining therapy regimens for triple negative breast cancer - Exploiting the epigenetic effects of eribulin action

Abstract P4-07-19: Quantifying epithelial-mesenchymal tumor heterogeneity for prediction of patient prognosis based on EMT state

Abstract P3-06-01: Eribulin alters the chromatin landscape to induce MET, attenuating metastatic progression and sensitizing breast tumors to subsequent chemotherapy

Abstract 2115: Multicomponent epigenetic profiling of intermediate epithelial-to-mesenchymal states in triple negative breast cancer identifies distinct regulatory signatures

Abstract 1417: Curtailing epithelial-mesenchymal plasticity for improved response to breast cancer therapy

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