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Brugada Syndrome: Clinical, Genetic, Molecular, Cellular, and Ionic Aspects

Current problems in cardiology, 2016-01, Vol.41 (1), p.7-57

2016

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Mechanisms Underlying Epicardial Radiofrequency Ablation to Suppress Arrhythmogenesis in Experimental Models of Brugada Syndrome

JACC. Clinical electrophysiology, 2017-04, Vol.3 (4), p.353-363

2017

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Mechanisms underlying the development of the electrocardiographic and arrhythmic manifestations of early repolarization syndrome

Journal of molecular and cellular cardiology, 2014-03, Vol.68, p.20-28

2014

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Repolarization defects can recapitulate arrhythmic and electrographic abnormalities in Brugada syndrome

Heart rhythm, 2022-03, Vol.19 (3), p.405-406

Patocskai, Bence

2022

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Mechanisms underlying the antiarrhythmic effect of ARumenamide-787 in experimental models of the J wave syndromes and hypothermia

PloS one, 2023-05, Vol.18 (5), p.e0281977-e0281977

2023

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Cellular Mechanism Underlying Hypothermia-Induced Ventricular Tachycardia/Ventricular Fibrillation in the Setting of Early Repolarization and the Protective Effect of Quinidine, Cilostazol, and Milrinone

Circulation. Arrhythmia and electrophysiology, 2014-02, Vol.7 (1), p.134-142

2014

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Acacetin suppresses the electrocardiographic and arrhythmic manifestations of the J wave syndromes

PloS one, 2020-11, Vol.15 (11), p.e0242747

2020

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Cellular and ionic mechanisms underlying the effects of cilostazol, milrinone, and isoproterenol to suppress arrhythmogenesis in an experimental model of early repolarization syndrome

Heart rhythm, 2016-06, Vol.13 (6), p.1326-1334

2016

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Hyperthermia Influences the Effects of Sodium Channel Blocking Drugs in Human-Induced Pluripotent Stem Cell-Derived Cardiomyocytes

PloS one, 2016-11, Vol.11 (11), p.e0166143

2016

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Ajmaline-Induced Slowing of Conduction in the Right Ventricular Outflow Tract Cannot Account for ST Elevation in Patients With Type I Brugada ECG

Circulation. Arrhythmia and electrophysiology, 2017-10, Vol.10 (10)

2017

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Ajmaline-Induced Slowing of Conduction in the RVOT Cannot Account for ST Elevation in Patients with Type I Brugada ECG

Circulation. Arrhythmia and electrophysiology, 2017-10, Vol.10 (10)

2017

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Epicardial Substrate as a Target for Radiofrequency Ablation in an Experimental Model of Early Repolarization Syndrome

Circulation. Arrhythmia and electrophysiology, 2018-09, Vol.11 (9), p.e006511-e006511

2018

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PO-01-148 COMBINATION OF FLECAINIDE AND IBUTILIDE FOR EFFECTIVE AND SAFE MANAGEMENT OF ATRIAL FIBRILLATION

Heart rhythm, 2023-05, Vol.20 (5), p.S144-S145

2023

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Novel therapeutic strategies for the management of ventricular arrhythmias associated with the Brugada syndrome

Expert opinion on orphan drugs, 2015-06, Vol.3 (6), p.633-651

2015

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Effect of Flecainide and Ibutilide Alone and in Combination to Terminate and Prevent Recurrence of Atrial Fibrillation

Circulation. Arrhythmia and electrophysiology, 2024-01, Vol.17 (1), p.e012454

2024

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Estradiol protection against toxic effects of catecholamine on electrical properties in human-induced pluripotent stem cell derived cardiomyocytes

International journal of cardiology, 2018-03, Vol.254, p.195-202

2018

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Cellular Mechanisms Underlying the Effects of Cilostazol, Milrinone, Isoproterenol and Ajmaline on Electrographic and Arrhythmic Manifestations of Early Repolarization Syndrome, and Comparative Analysis of the Cardiac Electrophysiological Effects of the Optical Isomers of Mexiletine

Patocskai, Bence

2017

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ProQuest

Fractionated Epicardial Electrograms

JACC. Clinical electrophysiology, 2021-02, Vol.7 (2), p.258-270

2021

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Fractionated Epicardial Electrograms: Implication for Mechanism of the Brugada Pattern

JACC. Clinical electrophysiology, 2021-02, Vol.7 (2), p.258-270

2021

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Different electrophysiological effects of the levo- and dextro-rotatory isomers of mexiletine in isolated rabbit cardiac muscle

Canadian journal of physiology and pharmacology, 2017-07, Vol.95 (7), p.830

2017

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Cellular Mechanism Underlying Hypothermia-Induced VT/VF in the Setting of Early Repolarization and the Protective Effect of Quinidine, Cilostazol and Milrinone

Circulation. Arrhythmia and electrophysiology, 2014-01, Vol.7 (1), p.134-142

2014

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Abstract 14547: Pharmacologic Approach to Treatment of the J Wave Syndromes

Circulation (New York, N.Y.), 2019-11, Vol.140 (Suppl_1 Suppl 1), p.A14547-A14547

2019

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Abstract 12543: Acacetin is a Promising New Pharmacologic Agent for Suppression of Life-threatening Arrhythmias Associated With the Brugada and Early Repolarization Syndromes

Circulation (New York, N.Y.), 2019-11, Vol.140 (Suppl_1 Suppl 1), p.A12543-A12543

2019

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Genetic, Ionic, and Cellular Mechanisms Underlying the J Wave Syndromes

Cardiac Electrophysiology: From Cell to Bedside, 2018, p.483-493

Seventh Edition, 2018

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51 - Genetic, Ionic, and Cellular Mechanisms Underlying the J Wave Syndromes

Cardiac Electrophysiology, 2018, p.483-493

Seventh Edition, 2018

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Brugada Syndrome: Clinical, Genetic, Molecular, Cellular, and Ionic Aspects

Mechanisms Underlying Epicardial Radiofrequency Ablation to Suppress Arrhythmogenesis in Experimental Models of Brugada Syndrome

Mechanisms underlying the development of the electrocardiographic and arrhythmic manifestations of early repolarization syndrome

Repolarization defects can recapitulate arrhythmic and electrographic abnormalities in Brugada syndrome

Mechanisms underlying the antiarrhythmic effect of ARumenamide-787 in experimental models of the J wave syndromes and hypothermia

Cellular Mechanism Underlying Hypothermia-Induced Ventricular Tachycardia/Ventricular Fibrillation in the Setting of Early Repolarization and the Protective Effect of Quinidine, Cilostazol, and Milrinone

Acacetin suppresses the electrocardiographic and arrhythmic manifestations of the J wave syndromes

Cellular and ionic mechanisms underlying the effects of cilostazol, milrinone, and isoproterenol to suppress arrhythmogenesis in an experimental model of early repolarization syndrome

Hyperthermia Influences the Effects of Sodium Channel Blocking Drugs in Human-Induced Pluripotent Stem Cell-Derived Cardiomyocytes

Ajmaline-Induced Slowing of Conduction in the Right Ventricular Outflow Tract Cannot Account for ST Elevation in Patients With Type I Brugada ECG

Ajmaline-Induced Slowing of Conduction in the RVOT Cannot Account for ST Elevation in Patients with Type I Brugada ECG

Epicardial Substrate as a Target for Radiofrequency Ablation in an Experimental Model of Early Repolarization Syndrome

PO-01-148 COMBINATION OF FLECAINIDE AND IBUTILIDE FOR EFFECTIVE AND SAFE MANAGEMENT OF ATRIAL FIBRILLATION

Novel therapeutic strategies for the management of ventricular arrhythmias associated with the Brugada syndrome

Effect of Flecainide and Ibutilide Alone and in Combination to Terminate and Prevent Recurrence of Atrial Fibrillation

Estradiol protection against toxic effects of catecholamine on electrical properties in human-induced pluripotent stem cell derived cardiomyocytes

Cellular Mechanisms Underlying the Effects of Cilostazol, Milrinone, Isoproterenol and Ajmaline on Electrographic and Arrhythmic Manifestations of Early Repolarization Syndrome, and Comparative Analysis of the Cardiac Electrophysiological Effects of the Optical Isomers of Mexiletine

Fractionated Epicardial Electrograms

Fractionated Epicardial Electrograms: Implication for Mechanism of the Brugada Pattern

Different electrophysiological effects of the levo- and dextro-rotatory isomers of mexiletine in isolated rabbit cardiac muscle

Cellular Mechanism Underlying Hypothermia-Induced VT/VF in the Setting of Early Repolarization and the Protective Effect of Quinidine, Cilostazol and Milrinone

Abstract 14547: Pharmacologic Approach to Treatment of the J Wave Syndromes

Abstract 12543: Acacetin is a Promising New Pharmacologic Agent for Suppression of Life-threatening Arrhythmias Associated With the Brugada and Early Repolarization Syndromes

Genetic, Ionic, and Cellular Mechanisms Underlying the J Wave Syndromes

51 - Genetic, Ionic, and Cellular Mechanisms Underlying the J Wave Syndromes

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