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Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

PLoS biology, 2008-12, Vol.6 (12), p.2853-e301

2008

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A human-like senescence-associated secretory phenotype is conserved in mouse cells dependent on physiological oxygen

PloS one, 2010-02, Vol.5 (2), p.e9188-e9188

2010

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p38MAPK is a novel DNA damage response-independent regulator of the senescence-associated secretory phenotype

The EMBO journal, 2011-04, Vol.30 (8), p.1536-1548

2011

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MicroRNAs miR-146a/b negatively modulate the senescence-associated inflammatory mediators IL-6 and IL-8

Aging (Albany, NY.), 2009-04, Vol.1 (4), p.402-411

2009

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Gcn4p and novel upstream activating sequences regulate targets of the unfolded protein response

PLoS biology, 2004-08, Vol.2 (8), p.E246-E246

2004

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Tumor suppressor and aging biomarker p16(INK4a) induces cellular senescence without the associated inflammatory secretory phenotype

The Journal of biological chemistry, 2011-10, Vol.286 (42), p.36396

2011

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Of Rats and Men

Cell, 2013-08, Vol.154 (3), p.481-483

2013

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MTOR regulates the pro-tumorigenic senescence-associated secretory phenotype by promoting IL1A translation

Nature cell biology, 2015-08, Vol.17 (8), p.1049-1061

2015

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Persistent DNA damage signalling triggers senescence-associated inflammatory cytokine secretion

Nature cell biology, 2009-08, Vol.11 (8), p.973-979

2009

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Putting Your Best Egg Forward

Developmental cell, 2018-01, Vol.44 (1), p.5-7

2018

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DNA-Dependent Kinase (p350) as a Candidate Gene for the Murine SCID Defect

Science (American Association for the Advancement of Science), 1995-02, Vol.267 (5201), p.1178-1183

1995

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Functional and Genomic Analyses Reveal an Essential Coordination between the Unfolded Protein Response and ER-Associated Degradation

Cell, 2000-04, Vol.101 (3), p.249-258

2000

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Author Correction: MTOR regulates the pro-tumorigenic senescence-associated secretory phenotype by promoting IL1A translation

Nature cell biology, 2021-05, Vol.23 (5), p.564-565

2021

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Features of mammalian microRNA promoters emerge from polymerase II chromatin immunoprecipitation data

PloS one, 2009-04, Vol.4 (4), p.e5279-e5279

2009

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MicroRNAs: an important player in maintaining a balance between inflammation and tumor suppression

Cell cycle (Georgetown, Tex.), 2009-06, Vol.8 (12), p.1822-1822

2009

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Defining success in graduate school

Molecular biology of the cell, 2014-07, Vol.25 (13), p.1942-1944

2014

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Overexpression of transcription factor Sp1 leads to gene expression perturbations and cell cycle inhibition

PloS one, 2009-09, Vol.4 (9), p.e7035

2009

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GROα regulates human embryonic stem cell self-renewal or adoption of a neuronal fate

Differentiation (London), 2011-04, Vol.81 (4), p.222-232

2011

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The thorny path linking cellular senescence to organismal aging

Mechanisms of ageing and development, 2005-10, Vol.126 (10), p.1040-1045

2005

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Tumor Suppressor and Aging Biomarker p16INK4a Induces Cellular Senescence without the Associated Inflammatory Secretory Phenotype

The Journal of biological chemistry, 2011-10, Vol.286 (42), p.36396-36403

2011

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Persistent DNA damage signalling triggers senescence-associated inflammatory cytokine secretion

Nature cell biology, 2009-10, Vol.11 (10), p.1272-1272

2009

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Transcriptome-wide prediction of miRNA targets in human and mouse using FASTH

PloS one, 2009-05, Vol.4 (5), p.e5745-e5745

2009

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“The Molecular Basis of Aging”: The Boehringer Ingelheim Fonds 95th International Titisee Conference

Mechanisms of ageing and development, 2007-07, Vol.128 (7), p.469-475

2007

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Genome-scale approaches for discovering novel nonconventional splicing substrates of the Ire1 nuclease

Genome biology, 2004-12, Vol.6 (1), p.R3-R3, Article R3

2004

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Analysis of SEC9 suppression reveals a relationship of SNARE function to cell physiology

PloS one, 2009-05, Vol.4 (5), p.e5449-e5449

2009

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Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

A human-like senescence-associated secretory phenotype is conserved in mouse cells dependent on physiological oxygen

p38MAPK is a novel DNA damage response-independent regulator of the senescence-associated secretory phenotype

MicroRNAs miR-146a/b negatively modulate the senescence-associated inflammatory mediators IL-6 and IL-8

Gcn4p and novel upstream activating sequences regulate targets of the unfolded protein response

Tumor suppressor and aging biomarker p16(INK4a) induces cellular senescence without the associated inflammatory secretory phenotype

Of Rats and Men

MTOR regulates the pro-tumorigenic senescence-associated secretory phenotype by promoting IL1A translation

Persistent DNA damage signalling triggers senescence-associated inflammatory cytokine secretion

Putting Your Best Egg Forward

DNA-Dependent Kinase (p350) as a Candidate Gene for the Murine SCID Defect

Functional and Genomic Analyses Reveal an Essential Coordination between the Unfolded Protein Response and ER-Associated Degradation

Author Correction: MTOR regulates the pro-tumorigenic senescence-associated secretory phenotype by promoting IL1A translation

Features of mammalian microRNA promoters emerge from polymerase II chromatin immunoprecipitation data

MicroRNAs: an important player in maintaining a balance between inflammation and tumor suppression

Defining success in graduate school

Overexpression of transcription factor Sp1 leads to gene expression perturbations and cell cycle inhibition

GROα regulates human embryonic stem cell self-renewal or adoption of a neuronal fate

The thorny path linking cellular senescence to organismal aging

Tumor Suppressor and Aging Biomarker p16INK4a Induces Cellular Senescence without the Associated Inflammatory Secretory Phenotype

Persistent DNA damage signalling triggers senescence-associated inflammatory cytokine secretion

Transcriptome-wide prediction of miRNA targets in human and mouse using FASTH

“The Molecular Basis of Aging”: The Boehringer Ingelheim Fonds 95th International Titisee Conference

Genome-scale approaches for discovering novel nonconventional splicing substrates of the Ire1 nuclease

Analysis of SEC9 suppression reveals a relationship of SNARE function to cell physiology

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