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Ethanol potentiates tumor necrosis factor‐α cytotoxicity in hepatoma cells and primary rat hepatocytes by promoting induction of the mitochondrial permeability transition

Hepatology (Baltimore, Md.), 2000-05, Vol.31 (5), p.1141-1152

2000

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Cytochrome c-Dependent Activation of Caspase-3 by Tumor Necrosis Factor Requires Induction of the Mitochondrial Permeability Transition

The American journal of pathology, 2000-06, Vol.156 (6), p.2111-2121

2000

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Ethanol, oxidative stress, and cytokine-induced liver cell injury

Alcohol, 2002-05, Vol.27 (1), p.63-68

2002

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The Overexpression of Bax Produces Cell Death upon Induction of the Mitochondrial Permeability Transition

The Journal of biological chemistry, 1998-03, Vol.273 (13), p.7770-7775

1998

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Functional Consequences of the Sustained or Transient Activation by Bax of the Mitochondrial Permeability Transition Pore

The Journal of biological chemistry, 1999-10, Vol.274 (44), p.31734-31739

1999

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Protoporphyrin IX, an endogenous ligand of the peripheral benzodiazepine receptor, potentiates induction of the mitochondrial permeability transition and the killing of cultured hepatocytes by rotenone

The Journal of biological chemistry, 1994-12, Vol.269 (49), p.31041-31046

1994

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Ca2+ depletion prevents anoxic death of hepatocytes by inhibiting mitochondrial permeability transition

American Journal of Physiology: Cell Physiology, 1995-03, Vol.268 (3), p.C676-C685

1995

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Cyclosporin and carnitine prevent the anoxic death of cultured hepatocytes by inhibiting the mitochondrial permeability transition

The Journal of biological chemistry, 1993-07, Vol.268 (19), p.13791-13798

1993

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Tumor Necrosis Factor Induces Phosphorylation and Translocation of BAD through a Phosphatidylinositide-3-OH Kinase-dependent Pathway

The Journal of biological chemistry, 1999-07, Vol.274 (27), p.19411-19416

1999

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The Cytotoxicity of Tumor Necrosis Factor Depends on Induction of the Mitochondrial Permeability Transition

The Journal of biological chemistry, 1996-11, Vol.271 (47), p.29792-29798

1996

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Potentiation by Chronic Ethanol Treatment of the Mitochondrial Permeability Transition

Biochemical and biophysical research communications, 1999-11, Vol.265 (2), p.405-409

1999

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ATP synthase activity is required for fructose to protect cultured hepatocytes from the toxicity of cyanide

American Journal of Physiology: Cell Physiology, 1993-03, Vol.264 (3), p.C709-C714

1993

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Mitochondrial Binding of Hexokinase II Inhibits Bax-induced Cytochrome c Release and Apoptosis

The Journal of biological chemistry, 2002-03, Vol.277 (9), p.7610-7618

2002

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Hexokinase II: the integration of energy metabolism and control of apoptosis

Current medicinal chemistry, 2003-08, Vol.10 (16), p.1535-1551

2003

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Alcohol and mitochondria: A dysfunctional relationship

Gastroenterology (New York, N.Y. 1943), 2002-06, Vol.122 (7), p.2049-2063

2002

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Cellular Signaling Mechanisms in Alcohol-Induced Liver Damage

Seminars in liver disease, 2004-08, Vol.24 (3), p.257-272

2004

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Increased sensitivity to oxidative injury in chinese hamster ovary cells stably transfected with rat liver S-adenosylmethionine synthetase cDNA

Biochemical journal, 1996-11, Vol.319 ( Pt 3) (3), p.767-773

1996

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Protection by cyclosporin A of cultured hepatocytes from the toxic consequences of the loss of mitochondrial energization produced by 1-methyl-4-phenylpyridinium

Biochemical pharmacology, 1992-08, Vol.44 (4), p.833-835

1992

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Cytochrome c Release upon Fas Receptor Activation Depends on Translocation of Full-length Bid and the Induction of the Mitochondrial Permeability Transition

The Journal of biological chemistry, 2002-03, Vol.277 (12), p.10073-10082

2002

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TNF-alpha-induced cell death in ethanol-exposed cells depends on p38 MAPK signaling but is independent of Bid and caspase-8

American journal of physiology: Gastrointestinal and liver physiology, 2003-09, Vol.285 (3), p.G503-G516

2003

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Elevated PTEN Levels Account for the Increased Sensitivity of Ethanol-exposed Cells to Tumor Necrosis Factor-induced Cytotoxicity

The Journal of biological chemistry, 2005-03, Vol.280 (10), p.9416-9424

2005

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Dexamethasone Induces Resistance to the Lethal Consequences of Electron Transport Inhibition in Cultured Hepatocytes

Archives of biochemistry and biophysics, 1995-04, Vol.318 (1), p.175-181

1995

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Functional consequences of the sustained or transient activation by Bax of the mitochondrial permeability transition pore

The Journal of biological chemistry, 2000-03, Vol.275 (11), p.8262-8262

2000

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Ca2+ depletion prevents anoxic death of hepatocytes by inhibiting mitochondrial permeability transition

American Journal of Physiology: Cell Physiology, 1995, Vol.37 (3), p.C676-C685

1995

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Cyclosporin and carnitine prevent the anowic death of cultured hepatocytes by inhibiting the mitochondrial permeability transition

The Journal of biological chemistry, 1993, Vol.268 (19), p.13791-13798

1993

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Ethanol potentiates tumor necrosis factor‐α cytotoxicity in hepatoma cells and primary rat hepatocytes by promoting induction of the mitochondrial permeability transition

Cytochrome c-Dependent Activation of Caspase-3 by Tumor Necrosis Factor Requires Induction of the Mitochondrial Permeability Transition

Ethanol, oxidative stress, and cytokine-induced liver cell injury

The Overexpression of Bax Produces Cell Death upon Induction of the Mitochondrial Permeability Transition

Functional Consequences of the Sustained or Transient Activation by Bax of the Mitochondrial Permeability Transition Pore

Protoporphyrin IX, an endogenous ligand of the peripheral benzodiazepine receptor, potentiates induction of the mitochondrial permeability transition and the killing of cultured hepatocytes by rotenone

Ca2+ depletion prevents anoxic death of hepatocytes by inhibiting mitochondrial permeability transition

Cyclosporin and carnitine prevent the anoxic death of cultured hepatocytes by inhibiting the mitochondrial permeability transition

Tumor Necrosis Factor Induces Phosphorylation and Translocation of BAD through a Phosphatidylinositide-3-OH Kinase-dependent Pathway

The Cytotoxicity of Tumor Necrosis Factor Depends on Induction of the Mitochondrial Permeability Transition

Potentiation by Chronic Ethanol Treatment of the Mitochondrial Permeability Transition

ATP synthase activity is required for fructose to protect cultured hepatocytes from the toxicity of cyanide

Mitochondrial Binding of Hexokinase II Inhibits Bax-induced Cytochrome c Release and Apoptosis

Hexokinase II: the integration of energy metabolism and control of apoptosis

Alcohol and mitochondria: A dysfunctional relationship

Cellular Signaling Mechanisms in Alcohol-Induced Liver Damage

Increased sensitivity to oxidative injury in chinese hamster ovary cells stably transfected with rat liver S-adenosylmethionine synthetase cDNA

Protection by cyclosporin A of cultured hepatocytes from the toxic consequences of the loss of mitochondrial energization produced by 1-methyl-4-phenylpyridinium

Cytochrome c Release upon Fas Receptor Activation Depends on Translocation of Full-length Bid and the Induction of the Mitochondrial Permeability Transition

TNF-alpha-induced cell death in ethanol-exposed cells depends on p38 MAPK signaling but is independent of Bid and caspase-8

Elevated PTEN Levels Account for the Increased Sensitivity of Ethanol-exposed Cells to Tumor Necrosis Factor-induced Cytotoxicity

Dexamethasone Induces Resistance to the Lethal Consequences of Electron Transport Inhibition in Cultured Hepatocytes

Functional consequences of the sustained or transient activation by Bax of the mitochondrial permeability transition pore

Ca2+ depletion prevents anoxic death of hepatocytes by inhibiting mitochondrial permeability transition

Cyclosporin and carnitine prevent the anowic death of cultured hepatocytes by inhibiting the mitochondrial permeability transition

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