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Severe Sepsis and Therapy with Activated Protein C

The New England journal of medicine, 2005-09, Vol.353 (13), p.1398-1400

Parrillo, Joseph E

2005

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A lipid A analog, E5531, blocks the endotoxin response in human volunteers with experimental endotoxemia

Critical care medicine, 2000-08, Vol.28 (8), p.2713-2720

2000

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Pulmonary artery occlusion pressure and central venous pressure fail to predict ventricular filling volume, cardiac performance, or the response to volume infusion in normal subjects

Critical care medicine, 2004-03, Vol.32 (3), p.691-699

2004

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Clinical review: Myocardial depression in sepsis and septic shock

Critical care (London, England), 2002-12, Vol.6 (6), p.500-508

2002

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Translating research to clinical practice : A 1-year experience with implementing early goal-directed therapy for septic shock in the emergency department

Chest, 2006-02, Vol.129 (2), p.225-232

2006

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The Duration of Hypotension before the Initiation of Antibiotic Treatment Is a Critical Determinant of Survival in a Murine Model of Escherichia coli Septic Shock: Association with Serum Lactate and Inflammatory Cytokine Levels

The Journal of infectious diseases, 2006-01, Vol.193 (2), p.251-258

2006

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Mechanisms of Disease: Pathogenetic Mechanisms of Septic Shock

The New England journal of medicine, 1993-05, Vol.328 (20), p.1471

1993

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Pathogenetic Mechanisms of Septic Shock

The New England journal of medicine, 1993-05, Vol.328 (20), p.1471-1477

Parrillo, Joseph E

1993

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Cardiogenic shock

Annals of internal medicine, 1999-07, Vol.131 (1), p.47

1999

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Coronary endothelial dysfunction after heart transplantation predicts allograft vasculopathy and cardiac death

Circulation (New York, N.Y.), 2001-12, Vol.104 (25), p.3091-3096

2001

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Tumor necrosis factor alpha and interleukin 1 beta are responsible for in vitro myocardial cell depression induced by human septic shock serum

The Journal of experimental medicine, 1996-03, Vol.183 (3), p.949-958

1996

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Increased Microvascular Reactivity and Improved Mortality in Septic Mice Lacking Inducible Nitric Oxide Synthase

Circulation research, 2000-04, Vol.86 (7), p.774-778

2000

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Severe Sepsis and Therapy with Activated Protein C

The New England journal of medicine, 2005-11, Vol.353 (21), p.2311-2311

2005

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Role of nitric oxide and cGMP in human septic serum-induced depression of cardiac myocyte contractility

American journal of physiology. Regulatory, integrative and comparative physiology, 1999-01, Vol.276 (1), p.265-R276

1999

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Human Serum from Patients with Septic Shock Activates Transcription Factors STAT1, IRF1, and NF-κB and Induces Apoptosis in Human Cardiac Myocytes

The Journal of biological chemistry, 2005-12, Vol.280 (52), p.42619-42626

2005

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MYOCARDIAL DYSFUNCTION IN SEPTIC SHOCK

Critical care clinics, 2000-04, Vol.16 (2), p.251-287

2000

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Utility of history, physical examination, electrocardiogram, and chest radiograph for differentiating normal from decreased systolic function in patients with heart failure

The American journal of medicine, 2002-04, Vol.112 (6), p.437-445

2002

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Inflammatory cardiomyopathy (myocarditis): which patients should be treated with anti-inflammatory therapy?

Circulation (New York, N.Y.), 2001-07, Vol.104 (1), p.4-6

Parrillo, J E

2001

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Mediator modulation therapy of severe sepsis and septic shock: does it work?

Critical care medicine, 2004-01, Vol.32 (1), p.282-286

2004

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Characterization of a hyperdynamic murine model of resuscitated sepsis using echocardiography

American journal of respiratory and critical care medicine, 2001-09, Vol.164 (5), p.891-895

2001

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Protective effect of a novel, potent inhibitor of poly(adenosine 5′-diphosphate-ribose) synthetase in a porcine model of severe bacterial sepsis

Critical care medicine, 2002-05, Vol.30 (5), p.974-980

2002

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Cloning and characterization of the human heparanase-1 (HPR1) gene promoter: role of GA-binding protein and Sp1 in regulating HPR1 basal promoter activity

The Journal of biological chemistry, 2002-03, Vol.277 (11), p.8989-8998

2002

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Complement

Critical care medicine, 2005-12, Vol.33 (12 Suppl), p.S482-S484

2005

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Myocardial dysfunction in septic shock: Part I. Clinical manifestation of cardiovascular dysfunction

Journal of cardiothoracic and vascular anesthesia, 2001-06, Vol.15 (3), p.364-376

2001

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Myocardial dysfunction in septic shock: Part II. Role of cytokines and nitric oxide

Journal of cardiothoracic and vascular anesthesia, 2001-08, Vol.15 (4), p.485-511

2001

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Severe Sepsis and Therapy with Activated Protein C

A lipid A analog, E5531, blocks the endotoxin response in human volunteers with experimental endotoxemia

Pulmonary artery occlusion pressure and central venous pressure fail to predict ventricular filling volume, cardiac performance, or the response to volume infusion in normal subjects

Clinical review: Myocardial depression in sepsis and septic shock

Translating research to clinical practice : A 1-year experience with implementing early goal-directed therapy for septic shock in the emergency department

The Duration of Hypotension before the Initiation of Antibiotic Treatment Is a Critical Determinant of Survival in a Murine Model of Escherichia coli Septic Shock: Association with Serum Lactate and Inflammatory Cytokine Levels

Mechanisms of Disease: Pathogenetic Mechanisms of Septic Shock

Pathogenetic Mechanisms of Septic Shock

Cardiogenic shock

Coronary endothelial dysfunction after heart transplantation predicts allograft vasculopathy and cardiac death

Tumor necrosis factor alpha and interleukin 1 beta are responsible for in vitro myocardial cell depression induced by human septic shock serum

Increased Microvascular Reactivity and Improved Mortality in Septic Mice Lacking Inducible Nitric Oxide Synthase

Severe Sepsis and Therapy with Activated Protein C

Role of nitric oxide and cGMP in human septic serum-induced depression of cardiac myocyte contractility

Human Serum from Patients with Septic Shock Activates Transcription Factors STAT1, IRF1, and NF-κB and Induces Apoptosis in Human Cardiac Myocytes

MYOCARDIAL DYSFUNCTION IN SEPTIC SHOCK

Utility of history, physical examination, electrocardiogram, and chest radiograph for differentiating normal from decreased systolic function in patients with heart failure

Inflammatory cardiomyopathy (myocarditis): which patients should be treated with anti-inflammatory therapy?

Mediator modulation therapy of severe sepsis and septic shock: does it work?

Characterization of a hyperdynamic murine model of resuscitated sepsis using echocardiography

Protective effect of a novel, potent inhibitor of poly(adenosine 5′-diphosphate-ribose) synthetase in a porcine model of severe bacterial sepsis

Cloning and characterization of the human heparanase-1 (HPR1) gene promoter: role of GA-binding protein and Sp1 in regulating HPR1 basal promoter activity

Complement

Myocardial dysfunction in septic shock: Part I. Clinical manifestation of cardiovascular dysfunction

Myocardial dysfunction in septic shock: Part II. Role of cytokines and nitric oxide

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