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Immobilization of the Gas Signaling Molecule H2S by Radioisotopes: Detection, Quantification, and In Vivo Imaging

Angewandte Chemie (International ed.), 2016-08, Vol.55 (32), p.9365-9370

International ed. in English, 2016

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Acute renal failure in zebrafish: a novel system to study a complex disease

American journal of physiology. Renal physiology, 2005-05, Vol.288 (5), p.F923-F929

2005

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Erythropoietin decreases renal fibrosis in mice with ureteral obstruction : Role of inhibiting TGF-β-induced epithelial-to-mesenchymal transition

Journal of the American Society of Nephrology, 2007-05, Vol.18 (5), p.1497-1507

2007

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Experimental evidence that preexisting chronic kidney disease is a risk factor for acute kidney injury

Kidney Research and Clinical Practice, 2014, 33(2), , pp.71-72

Moo Park, Kwon

2014

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Persistent low level of osterix accelerates interleukin-6 production and impairs regeneration after tissue injury

PloS one, 2013-07, Vol.8 (7), p.e69859-e69859

2013

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Loss of SAV1 in Kidney Proximal Tubule Induces Maladaptive Repair after Ischemia and Reperfusion Injury

International journal of molecular sciences, 2024-05, Vol.25 (9), p.4610

2024

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Reactive oxygen species/oxidative stress contributes to progression of kidney fibrosis following transient ischemic injury in mice

American journal of physiology. Renal physiology, 2009-08, Vol.297 (2), p.F461-F470

2009

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Increased obesity resistance and insulin sensitivity in mice lacking the isocitrate dehydrogenase 2 gene

Free radical biology & medicine, 2016-10, Vol.99, p.179-188

2016

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Methionine Sulfoxide Reductase A Deficiency Exacerbates Cisplatin-Induced Nephrotoxicity via Increased Mitochondrial Damage and Renal Cell Death

Antioxidants & redox signaling, 2017-10, Vol.27 (11), p.727

2017

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Oxidative stress following acute kidney injury causes disruption of lung cell cilia and their release into the bronchoaveolar lavage fluid and lung injury, which are exacerbated by Idh2 deletion

Redox biology, 2021-10, Vol.46, p.102077-102077, Article 102077

2021

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Deficiency of primary cilia in kidney epithelial cells induces epithelial to mesenchymal transition

Biochemical and biophysical research communications, 2018-02, Vol.496 (2), p.450-454

2018

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Reactive oxygen species generated by renal ischemia and reperfusion trigger protection against subsequent renal ischemia and reperfusion injury in mice

American journal of physiology. Renal physiology, 2010-01, Vol.298 (1), p.F158-F166

2010

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IDH2 deficiency increases the liver susceptibility to ischemia-reperfusion injury via increased mitochondrial oxidative injury

Redox biology, 2018-04, Vol.14, p.142-153

2018

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Isocitrate dehydrogenase 2 deficiency aggravates prolonged high-fat diet intake-induced hypertension

Redox biology, 2020-07, Vol.34, p.101548-101548, Article 101548

2020

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Omi/HtrA2 protease mediates cisplatin-induced cell death in renal cells

American journal of physiology. Renal physiology, 2005-02, Vol.288 (2), p.F371-F379

2005

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Short‐term control of diet affects cisplatin‐induced acute kidney injury through modulation of mitochondrial dynamics and mitochondrial GSH

Physiological reports, 2022-06, Vol.10 (12), p.e15348-n/a

2022

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Restoration of tubular epithelial cells during repair of the postischemic kidney occurs independently of bone marrow-derived stem cells

The Journal of clinical investigation, 2005-07, Vol.115 (7), p.1743-1755

2005

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Recruitment and subsequent proliferation of bone marrow-derived cells in the postischemic kidney are important to the progression of fibrosis

American journal of physiology. Renal physiology, 2014-06, Vol.306 (12), p.F1451-F1461

2014

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Unilateral nephrectomy elongates primary cilia in the remaining kidney via reactive oxygen species

Scientific reports, 2016-02, Vol.6 (1), p.22281-22281, Article 22281

2016

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Fragmentation of kidney epithelial cell primary cilia occurs by cisplatin and these cilia fragments are excreted into the urine

Redox biology, 2019-01, Vol.20, p.38-45

2019

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Reactive oxygen species differently regulate renal tubular epithelial and interstitial cell proliferation after ischemia and reperfusion injury

American journal of physiology. Renal physiology, 2010-05, Vol.298 (5), p.F1118-F1129

2010

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Testosterone Is Responsible for Enhanced Susceptibility of Males to Ischemic Renal Injury

The Journal of biological chemistry, 2004-12, Vol.279 (50), p.52282-52292

2004

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Protective role of methionine sulfoxide reductase A against ischemia/reperfusion injury in mouse kidney and its involvement in the regulation of trans-sulfuration pathway

Antioxidants & redox signaling, 2013-06, Vol.18 (17), p.2241

2013

Volltextzugriff (PDF)

Hydrogen sulfide, a gaseous signaling molecule, elongates primary cilia on kidney tubular epithelial cells by activating extracellular signal-regulated kinase

The Korean Journal of Physiology & Pharmacology, 2021, 25(6), , pp.593-601

2021

Volltextzugriff (PDF)

Macrophages promote Fibrinogenesis during kidney injury

Frontiers in medicine, 2023-06, Vol.10, p.1206362-1206362

2023

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Immobilization of the Gas Signaling Molecule H2S by Radioisotopes: Detection, Quantification, and In Vivo Imaging

Acute renal failure in zebrafish: a novel system to study a complex disease

Erythropoietin decreases renal fibrosis in mice with ureteral obstruction : Role of inhibiting TGF-β-induced epithelial-to-mesenchymal transition

Experimental evidence that preexisting chronic kidney disease is a risk factor for acute kidney injury

Persistent low level of osterix accelerates interleukin-6 production and impairs regeneration after tissue injury

Loss of SAV1 in Kidney Proximal Tubule Induces Maladaptive Repair after Ischemia and Reperfusion Injury

Reactive oxygen species/oxidative stress contributes to progression of kidney fibrosis following transient ischemic injury in mice

Increased obesity resistance and insulin sensitivity in mice lacking the isocitrate dehydrogenase 2 gene

Methionine Sulfoxide Reductase A Deficiency Exacerbates Cisplatin-Induced Nephrotoxicity via Increased Mitochondrial Damage and Renal Cell Death

Oxidative stress following acute kidney injury causes disruption of lung cell cilia and their release into the bronchoaveolar lavage fluid and lung injury, which are exacerbated by Idh2 deletion

Deficiency of primary cilia in kidney epithelial cells induces epithelial to mesenchymal transition

Reactive oxygen species generated by renal ischemia and reperfusion trigger protection against subsequent renal ischemia and reperfusion injury in mice

IDH2 deficiency increases the liver susceptibility to ischemia-reperfusion injury via increased mitochondrial oxidative injury

Isocitrate dehydrogenase 2 deficiency aggravates prolonged high-fat diet intake-induced hypertension

Omi/HtrA2 protease mediates cisplatin-induced cell death in renal cells

Short‐term control of diet affects cisplatin‐induced acute kidney injury through modulation of mitochondrial dynamics and mitochondrial GSH

Restoration of tubular epithelial cells during repair of the postischemic kidney occurs independently of bone marrow-derived stem cells

Recruitment and subsequent proliferation of bone marrow-derived cells in the postischemic kidney are important to the progression of fibrosis

Unilateral nephrectomy elongates primary cilia in the remaining kidney via reactive oxygen species

Fragmentation of kidney epithelial cell primary cilia occurs by cisplatin and these cilia fragments are excreted into the urine

Reactive oxygen species differently regulate renal tubular epithelial and interstitial cell proliferation after ischemia and reperfusion injury

Testosterone Is Responsible for Enhanced Susceptibility of Males to Ischemic Renal Injury

Protective role of methionine sulfoxide reductase A against ischemia/reperfusion injury in mouse kidney and its involvement in the regulation of trans-sulfuration pathway

Hydrogen sulfide, a gaseous signaling molecule, elongates primary cilia on kidney tubular epithelial cells by activating extracellular signal-regulated kinase

Macrophages promote Fibrinogenesis during kidney injury

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