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APP Receptor? To Be or Not To Be

Trends in pharmacological sciences (Regular ed.), 2016-05, Vol.37 (5), p.390-411

2016

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A Function for EHD Family Proteins in Unidirectional Retrograde Dendritic Transport of BACE1 and Alzheimer’s Disease Aβ Production

Cell reports (Cambridge), 2013-12, Vol.5 (6), p.1552-1563

2013

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Correction: Predominant expression of Alzheimer's disease-associated BIN1 in mature oligodendrocytes and localization to white matter tracts

Molecular neurodegeneration, 2023-10, Vol.18 (1), p.72-72, Article 72

2023

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Novel GαS-protein signaling associated with membrane-tethered amyloid precursor protein intracellular domain

The Journal of neuroscience, 2012-02, Vol.32 (5), p.1714-1729

2012

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Transgenic neuronal overexpression reveals that stringently regulated p23 expression is critical for coordinated movement in mice

Molecular neurodegeneration, 2011-12, Vol.6 (1), p.87-87, Article 87

2011

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Mutation Analysis of the Presenilin 1 N-terminal Domain Reveals a Broad Spectrum of γ-Secretase Activity toward Amyloid Precursor Protein and Other Substrates

The Journal of biological chemistry, 2010-12, Vol.285 (49), p.38042-38052

2010

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Loss of presenilin function is associated with a selective gain of APP function

eLife, 2016-05, Vol.5

2016

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Identification of the role of presenilins beyond Alzheimer’s disease

Pharmacological research, 2004-10, Vol.50 (4), p.411-418

2004

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APP-Mediated Signaling Prevents Memory Decline in Alzheimer’s Disease Mouse Model

Cell reports (Cambridge), 2019-04, Vol.27 (5), p.1345-1355.e6

2019

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Lack of BACE1 S-palmitoylation reduces amyloid burden and mitigates memory deficits in transgenic mouse models of Alzheimer’s disease

Proceedings of the National Academy of Sciences - PNAS, 2017-11, Vol.114 (45), p.E9665-E9674

2017

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Modeling Presenilin-Dependent Familial Alzheimer's Disease: Emphasis on Presenilin Substrate-Mediated Signaling and Synaptic Function

International journal of alzheimer's disease, 2010, Vol.2010, p.1-11

2010

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Spatial segregation of gamma-secretase and substrates in distinct membrane domains

The Journal of biological chemistry, 2005-07, Vol.280 (27), p.25892-25900

2005

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Steady-state increase of cAMP-response element binding protein, Rac, and PAK signaling in presenilin-deficient neurons

Journal of neurochemistry, 2008-03, Vol.104 (6), p.1637-1648

2008

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D-Serine Is an Endogenous Ligand for the Glycine Site of the N-Methyl-D-Aspartate Receptor

Proceedings of the National Academy of Sciences - PNAS, 2000-04, Vol.97 (9), p.4926-4931

2000

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Stringently regulated p23 expression is critical for coordinated movement in mice: implications for Alzheimer’s disease

Molecular neurodegeneration, 2012-02, Vol.7 (Suppl 1), p.L2-L2

2012

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Predominant expression of Alzheimer's disease-associated BIN1 in mature oligodendrocytes and localization to white matter tracts

Molecular neurodegeneration, 2016-08, Vol.11 (1), p.59-59, Article 59

2016

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Regulation of dynamic BACE1 trafficking in neurons

Molecular neurodegeneration, 2013-09, Vol.8 (S1), p.O8-O8, Article O8

2013

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Evidence That CD147 Modulation of β-Amyloid (Aβ) Levels Is Mediated by Extracellular Degradation of Secreted Aβ

The Journal of biological chemistry, 2008-07, Vol.283 (28), p.19489-19498

2008

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Reduced Alzheimer's Disease β-Amyloid Deposition in Transgenic Mice Expressing S -Palmitoylation-Deficient APH1aL and Nicastrin

The Journal of neuroscience, 2010-12, Vol.30 (48), p.16160-16169

2010

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O4-06-05: Presenilin-dependent changes in synaptic components and associated signaling cascades

Alzheimer's & dementia, 2008-07, Vol.4 (4), p.T197-T197

2008

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Localization and regional distribution of p23/TMP21 in the brain

Neurobiology of disease, 2008-10, Vol.32 (1), p.37-49

2008

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Mechanisms of Disease: new therapeutic strategies for Alzheimer's disease-targeting APP processing in lipid rafts

Nature Clinical Practice Neurology, 2007-07, Vol.3 (7), p.374-382

2007

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Synaptic Transmission and Hippocampal Long-Term Potentiation in Transgenic Mice Expressing FAD-Linked Presenilin 1

Neurobiology of disease, 1999-02, Vol.6 (1), p.56-62

1999

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Presenilin Attenuates Receptor-Mediated Signaling and Synaptic Function

The Journal of neuroscience, 2005-02, Vol.25 (6), p.1540-1549

2005

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Enhanced Synaptic Potentiation in Transgenic Mice Expressing presenilin 1 Familial Alzheimer's Disease Mutation Is Normalized with a Benzodiazepine

Neurobiology of disease, 2000-02, Vol.7 (1), p.54-63

2000

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APP Receptor? To Be or Not To Be

A Function for EHD Family Proteins in Unidirectional Retrograde Dendritic Transport of BACE1 and Alzheimer’s Disease Aβ Production

Correction: Predominant expression of Alzheimer's disease-associated BIN1 in mature oligodendrocytes and localization to white matter tracts

Novel GαS-protein signaling associated with membrane-tethered amyloid precursor protein intracellular domain

Transgenic neuronal overexpression reveals that stringently regulated p23 expression is critical for coordinated movement in mice

Mutation Analysis of the Presenilin 1 N-terminal Domain Reveals a Broad Spectrum of γ-Secretase Activity toward Amyloid Precursor Protein and Other Substrates

Loss of presenilin function is associated with a selective gain of APP function

Identification of the role of presenilins beyond Alzheimer’s disease

APP-Mediated Signaling Prevents Memory Decline in Alzheimer’s Disease Mouse Model

Lack of BACE1 S-palmitoylation reduces amyloid burden and mitigates memory deficits in transgenic mouse models of Alzheimer’s disease

Modeling Presenilin-Dependent Familial Alzheimer's Disease: Emphasis on Presenilin Substrate-Mediated Signaling and Synaptic Function

Spatial segregation of gamma-secretase and substrates in distinct membrane domains

Steady-state increase of cAMP-response element binding protein, Rac, and PAK signaling in presenilin-deficient neurons

D-Serine Is an Endogenous Ligand for the Glycine Site of the N-Methyl-D-Aspartate Receptor

Stringently regulated p23 expression is critical for coordinated movement in mice: implications for Alzheimer’s disease

Predominant expression of Alzheimer's disease-associated BIN1 in mature oligodendrocytes and localization to white matter tracts

Regulation of dynamic BACE1 trafficking in neurons

Evidence That CD147 Modulation of β-Amyloid (Aβ) Levels Is Mediated by Extracellular Degradation of Secreted Aβ

Reduced Alzheimer's Disease β-Amyloid Deposition in Transgenic Mice Expressing S -Palmitoylation-Deficient APH1aL and Nicastrin

O4-06-05: Presenilin-dependent changes in synaptic components and associated signaling cascades

Localization and regional distribution of p23/TMP21 in the brain

Mechanisms of Disease: new therapeutic strategies for Alzheimer's disease-targeting APP processing in lipid rafts

Synaptic Transmission and Hippocampal Long-Term Potentiation in Transgenic Mice Expressing FAD-Linked Presenilin 1

Presenilin Attenuates Receptor-Mediated Signaling and Synaptic Function

Enhanced Synaptic Potentiation in Transgenic Mice Expressing presenilin 1 Familial Alzheimer's Disease Mutation Is Normalized with a Benzodiazepine

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