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IVIVE: Facilitating the Use of In Vitro Toxicity Data in Risk Assessment and Decision Making

Toxics (Basel), 2022-05, Vol.10 (5), p.232

2022

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COVID-19 Seroprevalence and Active Infection in an Asymptomatic Population

Frontiers in medicine, 2021-09, Vol.8, p.749732-749732

2021

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Host Cell Transcriptional Tuning with CRISPR/dCas9 to Mitigate the Effects of Toxin Exposure

ACS synthetic biology, 2022-11, Vol.11 (11), p.3657-3668

2022

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Considerations for Development of Exposure Limits for Chemicals Encountered During Aircraft Operation

Military medicine, 2020-01, Vol.185 (Supplement_1), p.390-395

2020

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Development and evaluation of a high throughput inhalation model for organic chemicals

Journal of exposure science & environmental epidemiology, 2020-09, Vol.30 (5), p.866-877

2020

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Manganese binding to antioxidant peptides involved in extreme radiation resistance in Deinococcus radiodurans

Journal of inorganic biochemistry, 2016-11, Vol.164, p.49-58

2016

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Correction: Development and evaluation of a high throughput inhalation model for organic chemicals

Journal of exposure science & environmental epidemiology, 2020-09, Vol.30 (5), p.898-898

2020

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Riding (High) into the danger zone: a review of potential differences in chemical exposures in fighter pilots resulting from high altitude and G-forces

Expert opinion on drug metabolism & toxicology, 2017-09, Vol.13 (9), p.925-934

2017

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Sulindac metabolites induce proteosomal and lysosomal degradation of the epidermal growth factor receptor

Cancer prevention research (Philadelphia, Pa.), 2010-04, Vol.3 (4), p.560-572

2010

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W1742 Downregulation of EGFR Family Members is Not Sufficient for the Apoptotic Effects of Sulindac Metabolites

Gastroenterology (New York, N.Y. 1943), 2010, Vol.138 (5), p.S-731-S-731

2010

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S1653 Sulindac Metabolites (SMS) Inhibit Epidermal Growth Factor Receptor (EGFR) Signaling and Induce Lysosomal/Proteasomal Degradation of EGFR

Gastroenterology (New York, N.Y. 1943), 2008, Vol.134 (4), p.A-242-A-243

2008

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Sulindac metabolites inhibit epidermal growth factor receptor activation and expression

Journal of carcinogenesis, 2005-09, Vol.4 (1), p.16-16

2005

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Science and Technology Solutions for Scalable SARS-CoV-2 Testing to Inform Return to Full Capacity Strategy in United States Air Force Workforce Personnel

The Medical journal (Fort Sam Houston, Tex. : Print), 2021-01 (PB 8-21-01/02/03), p.37

2021

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In Silico Identification of Protein Targets for Chemical Neurotoxins Using ToxCast In Vitro Data and Read-Across within the QSAR Toolbox

Total Exposure Health, 2020, p.103-119

1, 2020

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Predicting neurological targets for chemical neurotoxins using ToxCast in vitro data and read-across within QSAR Toolbox

Toxicology letters, 2017-10, Vol.280, p.S281-S281

2017

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The complement inhibitors Crry and factor H are critical for preventing autologous complement activation on renal tubular epithelial cells

The Journal of immunology (1950), 2010-09, Vol.185 (5), p.3086-3094

2010

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Effects of the non-steriodal anti-inflammatory drug (NSAID) sulindac on epidermal growth factor receptor (EGFR) expression and signaling in colorectal cancer

Pangburn, Heather Ann

2007

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IVIVE: Facilitating the Use of In Vitro Toxicity Data in Risk Assessment and Decision Making

COVID-19 Seroprevalence and Active Infection in an Asymptomatic Population

Host Cell Transcriptional Tuning with CRISPR/dCas9 to Mitigate the Effects of Toxin Exposure

Considerations for Development of Exposure Limits for Chemicals Encountered During Aircraft Operation

Development and evaluation of a high throughput inhalation model for organic chemicals

Manganese binding to antioxidant peptides involved in extreme radiation resistance in Deinococcus radiodurans

Correction: Development and evaluation of a high throughput inhalation model for organic chemicals

Riding (High) into the danger zone: a review of potential differences in chemical exposures in fighter pilots resulting from high altitude and G-forces

Sulindac metabolites induce proteosomal and lysosomal degradation of the epidermal growth factor receptor

W1742 Downregulation of EGFR Family Members is Not Sufficient for the Apoptotic Effects of Sulindac Metabolites

S1653 Sulindac Metabolites (SMS) Inhibit Epidermal Growth Factor Receptor (EGFR) Signaling and Induce Lysosomal/Proteasomal Degradation of EGFR

Sulindac metabolites inhibit epidermal growth factor receptor activation and expression

Science and Technology Solutions for Scalable SARS-CoV-2 Testing to Inform Return to Full Capacity Strategy in United States Air Force Workforce Personnel

In Silico Identification of Protein Targets for Chemical Neurotoxins Using ToxCast In Vitro Data and Read-Across within the QSAR Toolbox

Predicting neurological targets for chemical neurotoxins using ToxCast in vitro data and read-across within QSAR Toolbox

The complement inhibitors Crry and factor H are critical for preventing autologous complement activation on renal tubular epithelial cells

Effects of the non-steriodal anti-inflammatory drug (NSAID) sulindac on epidermal growth factor receptor (EGFR) expression and signaling in colorectal cancer

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