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Genome-wide CRISPR screens and their applications in infectious disease

Frontiers in genome editing, 2023-09, Vol.5, p.1243731-1243731

2023

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Gain-of-function SOS1 mutations cause a distinctive form of Noonan syndrome

Nature genetics, 2007-01, Vol.39 (1), p.75-79

2007

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IL32: The multifaceted and unconventional cytokine

Human immunology, 2021-09, Vol.82 (9), p.659-667

2021

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Sputum Proteomics Reveals a Shift in Vitamin D-binding Protein and Antimicrobial Protein Axis in Tuberculosis Patients

Scientific reports, 2019-01, Vol.9 (1), p.1036-1036, Article 1036

2019

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Association of clade-G SARS-CoV-2 viruses and age with increased mortality rates across 57 countries and India

Infection, genetics and evolution, 2021-06, Vol.90, p.104734-104734, Article 104734

2021

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An exome wide association study of pulmonary tuberculosis patients and their asymptomatic household contacts

Infection, genetics and evolution, 2019-07, Vol.71, p.76-81

2019

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Genes and Genetics of Tuberculosis

International journal of pulmonary & respiratory sciences, 2017-05, Vol.1 (4)

Pandit, Bhaswati

2017

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CXCL10 is overexpressed in active tuberculosis patients compared to M. tuberculosis-exposed household contacts

Tuberculosis (Edinburgh, Scotland), 2018-03, Vol.109, p.8-16

2018

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SIGLECs and their contribution to tuberculosis

Nucleus (Calcutta), 2019-08, Vol.62 (2), p.119-125

2019

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Correction to: SIGLECs and their contribution to tuberculosis

Nucleus (Calcutta), 2019-08, Vol.62 (2), p.187-187

2019

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Deregulated Tyrosine–Phenylalanine Metabolism in Pulmonary Tuberculosis Patients

Journal of proteome research, 2015-04, Vol.14 (4), p.1947-1956

2015

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A detailed Hapmap of the Sitosterolemia locus spanning 69 kb; differences between Caucasians and African-Americans

BMC medical genetics, 2006-02, Vol.7 (1), p.13-13, Article 13

2006

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Analysis of variants in mitochondrial genome and their putative pathogenicity in tuberculosis patients from Mizoram, North east India

Mitochondrion, 2020-09, Vol.54, p.21-25

2020

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The rat STSL locus: characterization, chromosomal assignment, and genetic variations in sitosterolemic hypertensive rats

BMC cardiovascular disorders, 2003-06, Vol.3 (1), p.4-4, Article 4

2003

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Dissecting host factors that regulate the early stages of tuberculosis infection

Tuberculosis (Edinburgh, Scotland), 2016-09, Vol.100, p.102-113

2016

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Roles for Transcription Factors Sp1, NF-κB, IRF3, and IRF7 in Expression of the Human IFNL4 Gene

Viral immunology, 2016-01, Vol.29 (1), p.49-63

2016

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Correction: Corrigendum: Gain-of-function SOS1 mutations cause a distinctive form of Noonan syndrome

Nature genetics, 2007-02, Vol.39 (2), p.276

2007

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Loss of apolipoprotein E exacerbates the neonatal lethality of the Smith-Lemli-Opitz syndrome mouse

Molecular genetics and metabolism, 2007-05, Vol.91 (1), p.7-14

2007

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Induction of apoptosis by benzamide and its inhibition by aurin tricarboxylic acid (ATA) in Chinese hamster V79 cells

Mutation research, 2004-10, Vol.554 (1), p.121-129

2004

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Detection of Telomerase Activity in Chinese Hamster V79 Cells and Its Inhibition by 7-Deaza-deoxy Guanosine Triphosphate and (TTAGGG)4in Vitro

Biochemical and biophysical research communications, 1998-10, Vol.251 (2), p.620-624

1998

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Detection of telomerase activity in Chinese hamster V79 cells and its inhibition by 7-deaza-deoxy guanosine triphosphate and (TTAGGG)4 in vitro

Biochemical and biophysical research communications, 1998-10, Vol.251 (2), p.620-624

1998

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Erratum: Corrigendum: Gain-of-function SOS1 mutations cause a distinctive form of Noonan syndrome

Nature genetics, 2007-02, Vol.39 (2), p.276-276

2007

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Corrigendum: Gain-of-function SOS1 mutations cause a distinctive form of Noonan syndrome

Nature genetics, 2007-02, Vol.39 (2), p.276-276

2007

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Identification of Two Differentially Expressed Mitochondrial Genes in a Methotrexate-Resistant Chinese Hamster Cell Strain Derived from V79 Cells Using RNA Fingerprinting by Arbitrary Primed Polymerase Chain Reaction

Radiation research, 2003-07, Vol.160 (1), p.77-85

2003

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Genome-wide CRISPR screens and their applications in infectious disease

Gain-of-function SOS1 mutations cause a distinctive form of Noonan syndrome

IL32: The multifaceted and unconventional cytokine

Sputum Proteomics Reveals a Shift in Vitamin D-binding Protein and Antimicrobial Protein Axis in Tuberculosis Patients

Association of clade-G SARS-CoV-2 viruses and age with increased mortality rates across 57 countries and India

An exome wide association study of pulmonary tuberculosis patients and their asymptomatic household contacts

Genes and Genetics of Tuberculosis

CXCL10 is overexpressed in active tuberculosis patients compared to M. tuberculosis-exposed household contacts

SIGLECs and their contribution to tuberculosis

Correction to: SIGLECs and their contribution to tuberculosis

Deregulated Tyrosine–Phenylalanine Metabolism in Pulmonary Tuberculosis Patients

Gain-of-function RAF1 mutations cause Noonan and LEOPARD syndromes with hypertrophic cardiomyopathy

A detailed Hapmap of the Sitosterolemia locus spanning 69 kb; differences between Caucasians and African-Americans

Analysis of variants in mitochondrial genome and their putative pathogenicity in tuberculosis patients from Mizoram, North east India

The rat STSL locus: characterization, chromosomal assignment, and genetic variations in sitosterolemic hypertensive rats

Dissecting host factors that regulate the early stages of tuberculosis infection

Roles for Transcription Factors Sp1, NF-κB, IRF3, and IRF7 in Expression of the Human IFNL4 Gene

Correction: Corrigendum: Gain-of-function SOS1 mutations cause a distinctive form of Noonan syndrome

Loss of apolipoprotein E exacerbates the neonatal lethality of the Smith-Lemli-Opitz syndrome mouse

Induction of apoptosis by benzamide and its inhibition by aurin tricarboxylic acid (ATA) in Chinese hamster V79 cells

Detection of Telomerase Activity in Chinese Hamster V79 Cells and Its Inhibition by 7-Deaza-deoxy Guanosine Triphosphate and (TTAGGG)4in Vitro

Detection of telomerase activity in Chinese hamster V79 cells and its inhibition by 7-deaza-deoxy guanosine triphosphate and (TTAGGG)4 in vitro

Erratum: Corrigendum: Gain-of-function SOS1 mutations cause a distinctive form of Noonan syndrome

Corrigendum: Gain-of-function SOS1 mutations cause a distinctive form of Noonan syndrome

Identification of Two Differentially Expressed Mitochondrial Genes in a Methotrexate-Resistant Chinese Hamster Cell Strain Derived from V79 Cells Using RNA Fingerprinting by Arbitrary Primed Polymerase Chain Reaction

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