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μ‐Opioid receptors in primary sensory neurons are essential for opioid analgesic effect on acute and inflammatory pain and opioid‐induced hyperalgesia

The Journal of physiology, 2019-03, Vol.597 (6), p.1661-1675

2019

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Increased α2δ‐1–NMDA receptor coupling potentiates glutamatergic input to spinal dorsal horn neurons in chemotherapy‐induced neuropathic pain

Journal of neurochemistry, 2019-01, Vol.148 (2), p.252-274

2019

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α2δ‐1 protein drives opioid‐induced conditioned reward and synaptic NMDA receptor hyperactivity in the nucleus accumbens

Journal of neurochemistry, 2023-01, Vol.164 (2), p.143-157

2023

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Presynaptic NMDA receptors control nociceptive transmission at the spinal cord level in neuropathic pain

Cellular and molecular life sciences : CMLS, 2019-05, Vol.76 (10), p.1889-1899

2019

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Regulation of sympathetic vasomotor activity by the hypothalamic paraventricular nucleus in normotensive and hypertensive states

American journal of physiology. Heart and circulatory physiology, 2018-11, Vol.315 (5), p.H1200-H1214

2018

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Endogenous transient receptor potential ankyrin 1 and vanilloid 1 activity potentiates glutamatergic input to spinal lamina I neurons in inflammatory pain

Journal of neurochemistry, 2019-05, Vol.149 (3), p.381-398

2019

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NMDA Receptors at Primary Afferent-Excitatory Neuron Synapses Differentially Sustain Chemotherapy- and Nerve Trauma-Induced Chronic Pain

The Journal of neuroscience, 2023-05, Vol.43 (21), p.3933-3948

2023

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Regulating nociceptive transmission by VGluT2‐expressing spinal dorsal horn neurons

Journal of neurochemistry, 2018-11, Vol.147 (4), p.526-540

2018

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The α2δ-1-NMDA Receptor Complex Is Critically Involved in Neuropathic Pain Development and Gabapentin Therapeutic Actions

Cell reports (Cambridge), 2018-02, Vol.22 (9), p.2307-2321

2018

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Endogenous AT1 receptor–protein kinase C activity in the hypothalamus augments glutamatergic input and sympathetic outflow in hypertension

The Journal of physiology, 2019-08, Vol.597 (16), p.4325-4340

2019

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δ-Opioid receptors in primary sensory neurons tonically restrain nociceptive input in chronic pain but do not enhance morphine analgesic tolerance

Neuropharmacology, 2022-10, Vol.217, p.109202-109202, Article 109202

2022

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AMPK activation attenuates inflammatory pain through inhibiting NF-κB activation and IL-1β expression

Journal of neuroinflammation, 2019-02, Vol.16 (1), p.34-34, Article 34

2019

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α2δ‐1 couples to NMDA receptors in the hypothalamus to sustain sympathetic vasomotor activity in hypertension

The Journal of physiology, 2018-09, Vol.596 (17), p.4269-4283

2018

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Mitogen‐activated protein kinase signaling mediates opioid‐induced presynaptic NMDA receptor activation and analgesic tolerance

Journal of neurochemistry, 2019-01, Vol.148 (2), p.275-290

2019

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Calcineurin regulates synaptic Ca2+‐permeable AMPA receptors in hypothalamic presympathetic neurons via α2δ‐1‐mediated GluA1/GluA2 assembly

The Journal of physiology, 2024-05, Vol.602 (10), p.2179-2197

2024

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Cannabinoids suppress inflammatory and neuropathic pain by targeting α3 glycine receptors

The Journal of experimental medicine, 2012-06, Vol.209 (6), p.1121-1134

2012

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Ghrelin receptors mediate ghrelin‐induced excitation of agouti‐related protein/neuropeptide Y but not pro‐opiomelanocortin neurons

Journal of neurochemistry, 2017-08, Vol.142 (4), p.512-520

2017

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Pannexin-1 Up-regulation in the Dorsal Root Ganglion Contributes to Neuropathic Pain Development

The Journal of biological chemistry, 2015-06, Vol.290 (23), p.14647-14655

2015

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Bortezomib induces neuropathic pain through protein kinase C-mediated activation of presynaptic NMDA receptors in the spinal cord

Neuropharmacology, 2017-09, Vol.123, p.477-487

2017

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N-Methyl-d-aspartate Receptor- and Calpain-mediated Proteolytic Cleavage of K+-Cl− Cotransporter-2 Impairs Spinal Chloride Homeostasis in Neuropathic Pain

The Journal of biological chemistry, 2012-09, Vol.287 (40), p.33853-33864

2012

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Identification of diverse modulators of central and peripheral circadian clocks by high-throughput chemical screening

Proceedings of the National Academy of Sciences - PNAS, 2012-01, Vol.109 (1), p.101-106

2012

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CaMKII Regulates Synaptic NMDA Receptor Activity of Hypothalamic Presympathetic Neurons and Sympathetic Outflow in Hypertension

The Journal of neuroscience, 2017-11, Vol.37 (44), p.10690-10699

2017

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α2δ-1 Is Essential for Sympathetic Output and NMDA Receptor Activity Potentiated by Angiotensin II in the Hypothalamus

The Journal of neuroscience, 2018-07, Vol.38 (28), p.6388-6398

2018

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α2δ‐1 protein promotes synaptic expression of Ca2+ permeable–AMPA receptors by inhibiting GluA1/GluA2 heteromeric assembly in the hypothalamus in hypertension

Journal of neurochemistry, 2022-04, Vol.161 (1), p.40-52

2022

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Nerve injury increases brain-derived neurotrophic factor levels to suppress BK channel activity in primary sensory neurons

Journal of neurochemistry, 2012-06, Vol.121 (6), p.944-953

2012

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μ‐Opioid receptors in primary sensory neurons are essential for opioid analgesic effect on acute and inflammatory pain and opioid‐induced hyperalgesia

Increased α2δ‐1–NMDA receptor coupling potentiates glutamatergic input to spinal dorsal horn neurons in chemotherapy‐induced neuropathic pain

α2δ‐1 protein drives opioid‐induced conditioned reward and synaptic NMDA receptor hyperactivity in the nucleus accumbens

Presynaptic NMDA receptors control nociceptive transmission at the spinal cord level in neuropathic pain

Regulation of sympathetic vasomotor activity by the hypothalamic paraventricular nucleus in normotensive and hypertensive states

Endogenous transient receptor potential ankyrin 1 and vanilloid 1 activity potentiates glutamatergic input to spinal lamina I neurons in inflammatory pain

NMDA Receptors at Primary Afferent-Excitatory Neuron Synapses Differentially Sustain Chemotherapy- and Nerve Trauma-Induced Chronic Pain

Regulating nociceptive transmission by VGluT2‐expressing spinal dorsal horn neurons

The α2δ-1-NMDA Receptor Complex Is Critically Involved in Neuropathic Pain Development and Gabapentin Therapeutic Actions

Endogenous AT1 receptor–protein kinase C activity in the hypothalamus augments glutamatergic input and sympathetic outflow in hypertension

δ-Opioid receptors in primary sensory neurons tonically restrain nociceptive input in chronic pain but do not enhance morphine analgesic tolerance

AMPK activation attenuates inflammatory pain through inhibiting NF-κB activation and IL-1β expression

α2δ‐1 couples to NMDA receptors in the hypothalamus to sustain sympathetic vasomotor activity in hypertension

Mitogen‐activated protein kinase signaling mediates opioid‐induced presynaptic NMDA receptor activation and analgesic tolerance

Calcineurin regulates synaptic Ca2+‐permeable AMPA receptors in hypothalamic presympathetic neurons via α2δ‐1‐mediated GluA1/GluA2 assembly

Cannabinoids suppress inflammatory and neuropathic pain by targeting α3 glycine receptors

Ghrelin receptors mediate ghrelin‐induced excitation of agouti‐related protein/neuropeptide Y but not pro‐opiomelanocortin neurons

Pannexin-1 Up-regulation in the Dorsal Root Ganglion Contributes to Neuropathic Pain Development

Bortezomib induces neuropathic pain through protein kinase C-mediated activation of presynaptic NMDA receptors in the spinal cord

N-Methyl-d-aspartate Receptor- and Calpain-mediated Proteolytic Cleavage of K+-Cl− Cotransporter-2 Impairs Spinal Chloride Homeostasis in Neuropathic Pain

Identification of diverse modulators of central and peripheral circadian clocks by high-throughput chemical screening

CaMKII Regulates Synaptic NMDA Receptor Activity of Hypothalamic Presympathetic Neurons and Sympathetic Outflow in Hypertension

α2δ-1 Is Essential for Sympathetic Output and NMDA Receptor Activity Potentiated by Angiotensin II in the Hypothalamus

α2δ‐1 protein promotes synaptic expression of Ca2+ permeable–AMPA receptors by inhibiting GluA1/GluA2 heteromeric assembly in the hypothalamus in hypertension

Nerve injury increases brain-derived neurotrophic factor levels to suppress BK channel activity in primary sensory neurons

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