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Acute Neurological Illness in a Kidney Transplant Recipient Following Infection With Enterovirus‐D68: An Emerging Infection?

American journal of transplantation, 2015-12, Vol.15 (12), p.3224-3228

2015

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Rethinking cellular targets for lead neurotoxicity

Neurotoxicology (Park Forest South), 2015-05, Vol.48, p.249-249

Poretz, R.D.

2015

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Oligodendroglia in Developmental Neurotoxicity

Neurotoxicology, 2003-03, Vol.24 (2), p.161-178

2003

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Protein kinase C activation is required for the lead-induced inhibition of proliferation and differentiation of cultured oligodendroglial progenitor cells

Brain research, 2002-03, Vol.929 (1), p.87-95

2002

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Lead Exposure Delays the Differentiation of Oligodendroglial Progenitors in Vitro

Toxicology and applied pharmacology, 2001-08, Vol.174 (3), p.235-244

2001

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Lead Exposure Affects Levels of Galactolipid Metabolic Enzymes in the Developing Rat Brain

Toxicology and applied pharmacology, 2001-04, Vol.172 (2), p.98-107

2001

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EVOLUTION OF OUTPATIENT PARENTERAL ANTIBIOTIC THERAPY

Infectious disease clinics of North America, 1998-12, Vol.12 (4), p.827-834

Poretz, Donald M.

1998

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Sites of Photodamage Induced by Photodynamic Therapy with a Chlorin e6 Triacetoxymethyl Ester (CAME)

Photochemistry and photobiology, 2000-01, Vol.71 (1), p.94-96

2000

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Antimutagenicity, cytotoxicity and composition of chlorophyllin copper complex

Cancer letters, 1997-12, Vol.120 (2), p.141-147

1997

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Editorial Response: Direct Involvement of Physicians Is Vital to Outpatient Parenteral Antimicrobial Therapy for Central Nervous System Infections

Clinical infectious diseases, 1999-12, Vol.29 (6), p.1400-1401, Article 1400

Poretz, D M

1999

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Use of the Selective Oral Neuraminidase Inhibitor Oseltamivir to Prevent Influenza

The New England journal of medicine, 1999-10, Vol.341 (18), p.1336-1343

1999

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Lead Alters the Developmental Profile of the Galactolipid Metabolic Enzymes in Cultured Oligodendrocyte Lineage Cells

Neurotoxicology (Park Forest South), 2001-08, Vol.22 (4), p.429-437

2001

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Outpatient management of infectious diseases

International journal of clinical practice. Supplement, 2000-12 (115), p.23

Poretz, D M

2000

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Lead causes human fibroblasts to mis-sort arylsulfatase A

Toxicology (Amsterdam), 2001-06, Vol.163 (2), p.107-114

2001

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Reduction of Kaposiʼs sarcoma lesions following treatment of AIDS with ritonovir

AIDS (London), 1997-08, Vol.11 (10), p.1300-1301

1997

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Hybrid myeloma cells which secrete heterodimetric IgG: A model to study the N‐linked glycan

Immunology and cell biology, 2017-12, Vol.69 (3), p.151-157

2017

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The interaction of lead exposure and arylsulfatase A genotype affects sulfatide catabolism in human fibroblasts

Neurotoxicology (Park Forest South), 2000-06, Vol.21 (3), p.379-387

2000

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Association of Alcoholism with the N-Glycosylation Polymorphism of Pseudodeficient Human Arylsulfatase A

Alcoholism, clinical and experimental research, 1996-04, Vol.20 (2), p.228-233

1996

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Ritonavir and saquinavir combination therapy for the treatment of HIV infection

AIDS (London), 1999-02, Vol.13 (2), p.213-224

1999

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The R496H mutation of arylsulfatase A does not cause metachromatic leukodystrophy

Human mutation, 1998, Vol.12 (4), p.238-239

1998

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Outcomes of a trial of HIV-1 immunogen in patients with HIV infection

JAMA : the journal of the American Medical Association, 2001-05, Vol.285 (17), p.2192

Poretz, D M

2001

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Structural Characterization of Variant Forms of Arylsulfatase A that Associate with Alcoholism

Alcoholism, clinical and experimental research, 1996-04, Vol.20 (2), p.234-239

1996

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An acidic modification of the cytoplasmic domain contributes to the charge heterogeneity of the MHC class I antigens

Immunogenetics (New York), 1998-04, Vol.47 (5), p.381-389

1998

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Outpatient parenteral antibiotic therapy

International journal of antimicrobial agents, 1995, Vol.5 (1), p.9-12

Poretz, Donald M.

1995

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Acute Neurological Illness in a Kidney Transplant Recipient Following Infection With Enterovirus‐D68: An Emerging Infection?

Rethinking cellular targets for lead neurotoxicity

Oligodendroglia in Developmental Neurotoxicity

Protein kinase C activation is required for the lead-induced inhibition of proliferation and differentiation of cultured oligodendroglial progenitor cells

Lead Exposure Delays the Differentiation of Oligodendroglial Progenitors in Vitro

Lead Exposure Affects Levels of Galactolipid Metabolic Enzymes in the Developing Rat Brain

EVOLUTION OF OUTPATIENT PARENTERAL ANTIBIOTIC THERAPY

Sites of Photodamage Induced by Photodynamic Therapy with a Chlorin e6 Triacetoxymethyl Ester (CAME)

Switch to a raltegravir-based regimen versus continuation of a lopinavir-ritonavir-based regimen in stable HIV-infected patients with suppressed viraemia (SWITCHMRK 1 and 2): two multicentre, double-blind, randomised controlled trials

Antimutagenicity, cytotoxicity and composition of chlorophyllin copper complex

Editorial Response: Direct Involvement of Physicians Is Vital to Outpatient Parenteral Antimicrobial Therapy for Central Nervous System Infections

Use of the Selective Oral Neuraminidase Inhibitor Oseltamivir to Prevent Influenza

Lead Alters the Developmental Profile of the Galactolipid Metabolic Enzymes in Cultured Oligodendrocyte Lineage Cells

Outpatient management of infectious diseases

Lead causes human fibroblasts to mis-sort arylsulfatase A

Reduction of Kaposiʼs sarcoma lesions following treatment of AIDS with ritonovir

Hybrid myeloma cells which secrete heterodimetric IgG: A model to study the N‐linked glycan

The interaction of lead exposure and arylsulfatase A genotype affects sulfatide catabolism in human fibroblasts

Association of Alcoholism with the N-Glycosylation Polymorphism of Pseudodeficient Human Arylsulfatase A

Ritonavir and saquinavir combination therapy for the treatment of HIV infection

The R496H mutation of arylsulfatase A does not cause metachromatic leukodystrophy

Outcomes of a trial of HIV-1 immunogen in patients with HIV infection

Structural Characterization of Variant Forms of Arylsulfatase A that Associate with Alcoholism

An acidic modification of the cytoplasmic domain contributes to the charge heterogeneity of the MHC class I antigens

Outpatient parenteral antibiotic therapy

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