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Carbon quantum dots/block copolymer ensembles for metal-ion sensing and bioimaging

Journal of materials chemistry. B, Materials for biology and medicine, 2017-07, Vol.5 (27), p.5397-5402

2017

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Newly established tumourigenic primary human colon cancer cell lines are sensitive to TRAIL-induced apoptosis in vitro and in vivo

British journal of cancer, 2007-07, Vol.97 (1), p.73-84

2007

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A molecular signature for oncogenic BRAF in human colon cancer cells is revealed by microarray analysis

Current cancer drug targets, 2012-09, Vol.12 (7), p.873-898

2012

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902 BRAFV600E Inhibitor Resistance Mechanisms – Co-treatment With TRAIL and PI3K Inhibitors can Sensitise to Apoptosis Resistant Colon Cancer Cells

European journal of cancer (1990), 2012-07, Vol.48, p.S218-S219

2012

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Newly established tumourigenic primary human colon cancer cell lines are sensitive to TRAIL induced apoptosis in vitro and in vivo

British journal of cancer, 2007-12, Vol.97 (12), p.1716-1716

2007

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411 Epithelial plasticity during Epithelial-Mesenchymal Transition (EMT) is associated with alterations of histone H3 modifications

European journal of cancer supplements, 2010, Vol.8 (5), p.105-105

2010

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242 RAS and BRAF oncogenes sensitise colorectal tumours to TRAIL induced apoptosis: from cell and animal models to the clinic

European journal of cancer supplements, 2010, Vol.8 (5), p.64-64

2010

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804 Bioinformatic analysis of BRAFV600E vs RASG12V signatures in colon cancer cells reveals differential regulation of cellular pathways related to MSI or EMT

European journal of cancer supplements, 2010, Vol.8 (5), p.203-203

2010

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6126 Presence of K-RAS and BRAF oncogenic mutations sensitise colorectal tumours to TRAIL induced apoptosis: evidence from cell and animal models translated to the clinic

European journal of cancer supplements, 2009, Vol.7 (2), p.360-360

2009

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Transformation by Oncogenic RAS Sensitizes Human Colon Cells to TRAIL-induced Apoptosis by Up-regulating Death Receptor 4 and Death Receptor 5 through a MEK-dependent Pathway

The Journal of biological chemistry, 2005-06, Vol.280 (24), p.22856-22867

2005

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Carbon quantum dots/block copolymer ensembles for metal-ion sensing and bioimagingElectronic supplementary information (ESI) available: Hydrodynamic radii and normalized scattering intensity of CQDs/CSS-IEO-3 at different NaCl concentrations, additional TEM images for CQDs and CQDs/CSS-IEO-3, photoluminescence spectra of CQDs/CSS-IEO-3, titrated with several cations, and histograms of the photoluminescence intensity of CQDs, CQDs/CSS-IEO-3 and CQDs/CSS-IEO-3·BSA. See DOI: 10.1039/c7tb01352c

2017-07

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Microarray analysis and functional studies in a novel human colon cancer model of EMT: TAF12 regulates E-cadherin and Fra-1 regulates Vimentin expression

European journal of cancer supplements, 2008, Vol.6 (9), p.17-18

2008

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Cancer Genetics of Sporadic Colorectal Cancer: BRAF and PI3KCA Mutations, their Impact on Signaling and Novel Targeted Therapies

Anticancer research, 2006-03, Vol.26 (2A), p.1077-1084

2006

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High levels of phosphorylated c-Jun, Fra-1, Fra-2 and ATF-2 proteins correlate with malignant phenotypes in the multistage mouse skin carcinogenesis model

Oncogene, 2000-08, Vol.19 (35), p.4011-4021

2000

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Microarray analysis of the differential transformation mediated by Kirsten and Harvey Ras oncogenes in a human colorectal adenocarcinoma cell line

International journal of cancer, 2006-02, Vol.118 (3), p.616-627

2006

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Sensitization of (colon) cancer cells to death receptor related therapies: A report from the FP6-ONCODEATH research consortium

Cancer biology & therapy, 2012-05, Vol.13 (7), p.458-466

2012

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High Activity of Serum Response Factor in the Mesenchymal Transition of Epithelial Tumor Cells Is Regulated by RhoA Signaling

The Journal of biological chemistry, 2002-08, Vol.277 (33), p.29490-29495

2002

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Net, a new ets transcription factor that is activated by Ras

Genes & development, 1994-07, Vol.8 (13), p.1502-1513

1994

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Erratum: Newly established tumourigenic primary human colon cancer cell lines are sensitive to TRAIL induced apoptosis in vitro and in vivo

British journal of cancer, 2007-12, Vol.97 (12), p.1716-1716

2007

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Regulation of AP-1/DNA complex formation in vitro

Oncogene, 1991-02, Vol.6 (2), p.205-209

1991

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Transformation by the fos or jun oncogene does not increase AP-1 DNA-binding activity

Journal of Virology, 1993-09, Vol.67 (9), p.5487-5495

1993

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Molecular mechanisms involved in CD43-mediated apoptosis of TF-1 cells. Roles of transcription Daxx expression, and adhesion molecules

The Journal of biological chemistry, 2002-03, Vol.277 (10), p.7955-7961

2002

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Prognostic significance of matrix metalloproteinases 2 and 9 in bladder cancer

Anticancer research, 2000-05, Vol.20 (3B), p.2009

2000

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TAF7 (TAF sub(II)55) Plays a Role in the Transcription Activation by c-Jun

The Journal of biological chemistry, 2003-06, Vol.278 (24), p.21510-21516

2003

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Expression of ras and myc oncogenes in human hepatocellular carcinoma and non-neoplastic liver tissues

Anticancer research, 1989-05, Vol.9 (3), p.715

1989

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Carbon quantum dots/block copolymer ensembles for metal-ion sensing and bioimaging

Newly established tumourigenic primary human colon cancer cell lines are sensitive to TRAIL-induced apoptosis in vitro and in vivo

A molecular signature for oncogenic BRAF in human colon cancer cells is revealed by microarray analysis

902 BRAFV600E Inhibitor Resistance Mechanisms – Co-treatment With TRAIL and PI3K Inhibitors can Sensitise to Apoptosis Resistant Colon Cancer Cells

Newly established tumourigenic primary human colon cancer cell lines are sensitive to TRAIL induced apoptosis in vitro and in vivo

411 Epithelial plasticity during Epithelial-Mesenchymal Transition (EMT) is associated with alterations of histone H3 modifications

242 RAS and BRAF oncogenes sensitise colorectal tumours to TRAIL induced apoptosis: from cell and animal models to the clinic

804 Bioinformatic analysis of BRAFV600E vs RASG12V signatures in colon cancer cells reveals differential regulation of cellular pathways related to MSI or EMT

6126 Presence of K-RAS and BRAF oncogenic mutations sensitise colorectal tumours to TRAIL induced apoptosis: evidence from cell and animal models translated to the clinic

Transformation by Oncogenic RAS Sensitizes Human Colon Cells to TRAIL-induced Apoptosis by Up-regulating Death Receptor 4 and Death Receptor 5 through a MEK-dependent Pathway

Microarray analysis and functional studies in a novel human colon cancer model of EMT: TAF12 regulates E-cadherin and Fra-1 regulates Vimentin expression

Cancer Genetics of Sporadic Colorectal Cancer: BRAF and PI3KCA Mutations, their Impact on Signaling and Novel Targeted Therapies

High levels of phosphorylated c-Jun, Fra-1, Fra-2 and ATF-2 proteins correlate with malignant phenotypes in the multistage mouse skin carcinogenesis model

Microarray analysis of the differential transformation mediated by Kirsten and Harvey Ras oncogenes in a human colorectal adenocarcinoma cell line

Sensitization of (colon) cancer cells to death receptor related therapies: A report from the FP6-ONCODEATH research consortium

High Activity of Serum Response Factor in the Mesenchymal Transition of Epithelial Tumor Cells Is Regulated by RhoA Signaling

Net, a new ets transcription factor that is activated by Ras

Erratum: Newly established tumourigenic primary human colon cancer cell lines are sensitive to TRAIL induced apoptosis in vitro and in vivo

Regulation of AP-1/DNA complex formation in vitro

Transformation by the fos or jun oncogene does not increase AP-1 DNA-binding activity

Molecular mechanisms involved in CD43-mediated apoptosis of TF-1 cells. Roles of transcription Daxx expression, and adhesion molecules

Prognostic significance of matrix metalloproteinases 2 and 9 in bladder cancer

TAF7 (TAF sub(II)55) Plays a Role in the Transcription Activation by c-Jun

Expression of ras and myc oncogenes in human hepatocellular carcinoma and non-neoplastic liver tissues

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